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396 related items for PubMed ID: 1793460

  • 1. Amyloid plaques, neurofibrillary tangles and neuronal loss in brains of transgenic mice overexpressing a C-terminal fragment of human amyloid precursor protein.
    Kawabata S, Higgins GA, Gordon JW.
    Nature; 1991 Dec 12; 354(6353):476-8. PubMed ID: 1793460
    [Abstract] [Full Text] [Related]

  • 2. Central cholinergic functions in human amyloid precursor protein knock-in/presenilin-1 transgenic mice.
    Hartmann J, Erb C, Ebert U, Baumann KH, Popp A, König G, Klein J.
    Neuroscience; 2004 Dec 12; 125(4):1009-17. PubMed ID: 15120860
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  • 3. Targeted introduction of V642I mutation in amyloid precursor protein gene causes functional abnormality resembling early stage of Alzheimer's disease in aged mice.
    Kawasumi M, Chiba T, Yamada M, Miyamae-Kaneko M, Matsuoka M, Nakahara J, Tomita T, Iwatsubo T, Kato S, Aiso S, Nishimoto I, Kouyama K.
    Eur J Neurosci; 2004 May 12; 19(10):2826-38. PubMed ID: 15147316
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  • 4. Alzheimer-type neuropathology in transgenic mice overexpressing V717F beta-amyloid precursor protein.
    Games D, Adams D, Alessandrini R, Barbour R, Berthelette P, Blackwell C, Carr T, Clemens J, Donaldson T, Gillespie F.
    Nature; 1995 Feb 09; 373(6514):523-7. PubMed ID: 7845465
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  • 6. Accelerated amyloid deposition, neurofibrillary degeneration and neuronal loss in double mutant APP/tau transgenic mice.
    Ribé EM, Pérez M, Puig B, Gich I, Lim F, Cuadrado M, Sesma T, Catena S, Sánchez B, Nieto M, Gómez-Ramos P, Morán MA, Cabodevilla F, Samaranch L, Ortiz L, Pérez A, Ferrer I, Avila J, Gómez-Isla T.
    Neurobiol Dis; 2005 Dec 09; 20(3):814-22. PubMed ID: 16125396
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  • 7. Enhanced neurofibrillary degeneration in transgenic mice expressing mutant tau and APP.
    Lewis J, Dickson DW, Lin WL, Chisholm L, Corral A, Jones G, Yen SH, Sahara N, Skipper L, Yager D, Eckman C, Hardy J, Hutton M, McGowan E.
    Science; 2001 Aug 24; 293(5534):1487-91. PubMed ID: 11520987
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  • 8. Beta-amyloid precursor protein transgenic mice that harbor diffuse A beta deposits but do not form plaques show increased ischemic vulnerability: role of inflammation.
    Koistinaho M, Kettunen MI, Goldsteins G, Keinänen R, Salminen A, Ort M, Bures J, Liu D, Kauppinen RA, Higgins LS, Koistinaho J.
    Proc Natl Acad Sci U S A; 2002 Feb 05; 99(3):1610-5. PubMed ID: 11818564
    [Abstract] [Full Text] [Related]

  • 9. Amyloid cored plaques in Tg2576 transgenic mice are characterized by giant plaques, slightly activated microglia, and the lack of paired helical filament-typed, dystrophic neurites.
    Sasaki A, Shoji M, Harigaya Y, Kawarabayashi T, Ikeda M, Naito M, Matsubara E, Abe K, Nakazato Y.
    Virchows Arch; 2002 Oct 05; 441(4):358-67. PubMed ID: 12404061
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  • 11. Neuropathology of mice carrying mutant APP(swe) and/or PS1(M146L) transgenes: alterations in the p75(NTR) cholinergic basal forebrain septohippocampal pathway.
    Jaffar S, Counts SE, Ma SY, Dadko E, Gordon MN, Morgan D, Mufson EJ.
    Exp Neurol; 2001 Aug 05; 170(2):227-43. PubMed ID: 11476589
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  • 12. Intraneuronal amyloid beta and reduced brain volume in a novel APP T714I mouse model for Alzheimer's disease.
    Van Broeck B, Vanhoutte G, Pirici D, Van Dam D, Wils H, Cuijt I, Vennekens K, Zabielski M, Michalik A, Theuns J, De Deyn PP, Van der Linden A, Van Broeckhoven C, Kumar-Singh S.
    Neurobiol Aging; 2008 Feb 05; 29(2):241-52. PubMed ID: 17112635
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  • 13. Neocortical synaptic bouton number is maintained despite robust amyloid deposition in APP23 transgenic mice.
    Boncristiano S, Calhoun ME, Howard V, Bondolfi L, Kaeser SA, Wiederhold KH, Staufenbiel M, Jucker M.
    Neurobiol Aging; 2005 May 05; 26(5):607-13. PubMed ID: 15708435
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  • 14. Progressive cognitive impairment and anxiety induction in the absence of plaque deposition in C57BL/6 inbred mice expressing transgenic amyloid precursor protein.
    Lee KW, Lee SH, Kim H, Song JS, Yang SD, Paik SG, Han PL.
    J Neurosci Res; 2004 May 15; 76(4):572-80. PubMed ID: 15114629
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  • 15. Non-tau based neuronal degeneration in Alzheimer's disease -- an immunocytochemical and quantitative study in the supragranular layers of the middle temporal neocortex.
    van de Nes JA, Nafe R, Schlote W.
    Brain Res; 2008 Jun 05; 1213():152-65. PubMed ID: 18455153
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  • 16. Time course of the development of Alzheimer-like pathology in the doubly transgenic PS1+APP mouse.
    Gordon MN, Holcomb LA, Jantzen PT, DiCarlo G, Wilcock D, Boyett KW, Connor K, Melachrino J, O'Callaghan JP, Morgan D.
    Exp Neurol; 2002 Feb 05; 173(2):183-95. PubMed ID: 11822882
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  • 17. Genetic background regulates beta-amyloid precursor protein processing and beta-amyloid deposition in the mouse.
    Lehman EJ, Kulnane LS, Gao Y, Petriello MC, Pimpis KM, Younkin L, Dolios G, Wang R, Younkin SG, Lamb BT.
    Hum Mol Genet; 2003 Nov 15; 12(22):2949-56. PubMed ID: 14506131
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  • 18. Transient intraneuronal A beta rather than extracellular plaque pathology correlates with neuron loss in the frontal cortex of APP/PS1KI mice.
    Christensen DZ, Kraus SL, Flohr A, Cotel MC, Wirths O, Bayer TA.
    Acta Neuropathol; 2008 Dec 15; 116(6):647-55. PubMed ID: 18974993
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  • 19. The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics.
    Hardy J, Selkoe DJ.
    Science; 2002 Jul 19; 297(5580):353-6. PubMed ID: 12130773
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  • 20. Amyloid precursor protein cytoplasmic domain with phospho-Thr668 accumulates in Alzheimer's disease and its transgenic models: a role to mediate interaction of Abeta and tau.
    Shin RW, Ogino K, Shimabuku A, Taki T, Nakashima H, Ishihara T, Kitamoto T.
    Acta Neuropathol; 2007 Jun 19; 113(6):627-36. PubMed ID: 17431643
    [Abstract] [Full Text] [Related]

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