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Journal Abstract Search

329 related items for PubMed ID: 18039111

  • 1. Neurodegenerative diseases: new concepts of pathogenesis and their therapeutic implications.
    Skovronsky DM, Lee VM, Trojanowski JQ.
    Annu Rev Pathol; 2006; 1():151-70. PubMed ID: 18039111
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  • 7. [Amyloidosis: a model of misfolded protein disorder].
    Grateau G, Verine J, Delpech M, Ries M.
    Med Sci (Paris); 2005; 21(6-7):627-33. PubMed ID: 15985206
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  • 8. Autophagy in aging and neurodegenerative diseases: implications for pathogenesis and therapy.
    Tan CC, Yu JT, Tan MS, Jiang T, Zhu XC, Tan L.
    Neurobiol Aging; 2014 May; 35(5):941-57. PubMed ID: 24360503
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  • 10. Drug targets for amyloidosis.
    Kolstoe SE, Wood SP.
    Biochem Soc Trans; 2010 Apr; 38(2):466-70. PubMed ID: 20298204
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  • 14. Fluorescence as a method to reveal structures and membrane-interactions of amyloidogenic proteins.
    Munishkina LA, Fink AL.
    Biochim Biophys Acta; 2007 Aug; 1768(8):1862-85. PubMed ID: 17493579
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  • 15. Heterogeneous amyloid-formed ion channels as a common cytotoxic mechanism: implications for therapeutic strategies against amyloidosis.
    Kourie JI, Culverson AL, Farrelly PV, Henry CL, Laohachai KN.
    Cell Biochem Biophys; 2002 Aug; 36(2-3):191-207. PubMed ID: 12139405
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  • 16. Amino acid sequence determinants and molecular chaperones in amyloid fibril formation.
    Nerelius C, Fitzen M, Johansson J.
    Biochem Biophys Res Commun; 2010 May 21; 396(1):2-6. PubMed ID: 20494101
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  • 18. Common mechanisms of amyloid oligomer pathogenesis in degenerative disease.
    Glabe CG.
    Neurobiol Aging; 2006 Apr 21; 27(4):570-5. PubMed ID: 16481071
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  • 19. Protein-misfolding diseases and chaperone-based therapeutic approaches.
    Chaudhuri TK, Paul S.
    FEBS J; 2006 Apr 21; 273(7):1331-49. PubMed ID: 16689923
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