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Pubmed for Handhelds

PUBMED FOR HANDHELDS

Journal Abstract Search

1162 related items for PubMed ID: 18059005

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  • 4. Antisense oligonucleotide-induced exon skipping restores dystrophin expression in vitro in a canine model of DMD.
    McClorey G, Moulton HM, Iversen PL, Fletcher S, Wilton SD.
    Gene Ther; 2006 Oct; 13(19):1373-81. PubMed ID: 16724091
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  • 6. Antisense-induced exon skipping for duplications in Duchenne muscular dystrophy.
    Aartsma-Rus A, Janson AA, van Ommen GJ, van Deutekom JC.
    BMC Med Genet; 2007 Jul 05; 8():43. PubMed ID: 17612397
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  • 7. Becker muscular dystrophy caused by an intronic mutation reducing the efficiency of the splice donor site of intron 26 of the dystrophin gene.
    Baskin B, Banwell B, Khater RA, Hawkins C, Ray PN.
    Neuromuscul Disord; 2009 Mar 05; 19(3):189-92. PubMed ID: 19230662
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  • 11. Characterization of a complex Duchenne muscular dystrophy-causing dystrophin gene inversion and restoration of the reading frame by induced exon skipping.
    Madden HR, Fletcher S, Davis MR, Wilton SD.
    Hum Mutat; 2009 Jan 05; 30(1):22-8. PubMed ID: 18570328
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  • 12. In vivo comparison of 2'-O-methyl phosphorothioate and morpholino antisense oligonucleotides for Duchenne muscular dystrophy exon skipping.
    Heemskerk HA, de Winter CL, de Kimpe SJ, van Kuik-Romeijn P, Heuvelmans N, Platenburg GJ, van Ommen GJ, van Deutekom JC, Aartsma-Rus A.
    J Gene Med; 2009 Mar 05; 11(3):257-66. PubMed ID: 19140108
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  • 13. Comparative analysis of antisense oligonucleotide analogs for targeted DMD exon 46 skipping in muscle cells.
    Aartsma-Rus A, Kaman WE, Bremmer-Bout M, Janson AA, den Dunnen JT, van Ommen GJ, van Deutekom JC.
    Gene Ther; 2004 Sep 05; 11(18):1391-8. PubMed ID: 15229633
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  • 14. Bioinformatic and functional optimization of antisense phosphorodiamidate morpholino oligomers (PMOs) for therapeutic modulation of RNA splicing in muscle.
    Popplewell LJ, Graham IR, Malerba A, Dickson G.
    Methods Mol Biol; 2011 Sep 05; 709():153-78. PubMed ID: 21194027
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  • 15. Theoretic applicability of antisense-mediated exon skipping for Duchenne muscular dystrophy mutations.
    Aartsma-Rus A, Fokkema I, Verschuuren J, Ginjaar I, van Deutekom J, van Ommen GJ, den Dunnen JT.
    Hum Mutat; 2009 Mar 05; 30(3):293-9. PubMed ID: 19156838
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  • 16. Point mutations in the dystrophin gene: evidence for frequent use of cryptic splice sites as a result of splicing defects.
    Tuffery-Giraud S, Chambert S, Demaille J, Claustres M.
    Hum Mutat; 1999 Mar 05; 14(5):359-68. PubMed ID: 10533061
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  • 18. Exploring the frontiers of therapeutic exon skipping for Duchenne muscular dystrophy by double targeting within one or multiple exons.
    Aartsma-Rus A, Kaman WE, Weij R, den Dunnen JT, van Ommen GJ, van Deutekom JC.
    Mol Ther; 2006 Sep 05; 14(3):401-7. PubMed ID: 16753346
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  • 19. A novel splice site mutation (3157+1G>T) in the dystrophin gene causing total exon skipping and DMD phenotype.
    Sironi M, Corti S, Locatelli F, Cagliani R, Comi GP.
    Hum Mutat; 2001 Mar 05; 17(3):239. PubMed ID: 11241855
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