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131 related items for PubMed ID: 18059343

  • 1. Differential motility of p190bcr-abl- and p210bcr-abl-expressing cells: respective roles of Vav and Bcr-Abl GEFs.
    Daubon T, Chasseriau J, El Ali A, Rivet J, Kitzis A, Constantin B, Bourmeyster N.
    Oncogene; 2008 Apr 24; 27(19):2673-85. PubMed ID: 18059343
    [Abstract] [Full Text] [Related]

  • 2. Invadopodia and rolling-type motility are specific features of highly invasive p190(bcr-abl) leukemic cells.
    Daubon T, Rochelle T, Bourmeyster N, Génot E.
    Eur J Cell Biol; 2012 Apr 24; 91(11-12):978-87. PubMed ID: 22717125
    [Abstract] [Full Text] [Related]

  • 3. Differential interaction and activation of Rho family GTPases by p210bcr-abl and p190bcr-abl.
    Harnois T, Constantin B, Rioux A, Grenioux E, Kitzis A, Bourmeyster N.
    Oncogene; 2003 Sep 25; 22(41):6445-54. PubMed ID: 14508524
    [Abstract] [Full Text] [Related]

  • 4. p210(Bcr-Abl) desensitizes Cdc42 GTPase signaling for SDF-1alpha-directed migration in chronic myeloid leukemia cells.
    Chang YC, Tien SC, Tien HF, Zhang H, Bokoch GM, Chang ZF.
    Oncogene; 2009 Nov 19; 28(46):4105-15. PubMed ID: 19718053
    [Abstract] [Full Text] [Related]

  • 5. p210bcr-abl induces amoeboid motility by recruiting ADF/destrin through RhoA/ROCK1.
    Rochelle T, Daubon T, Van Troys M, Harnois T, Waterschoot D, Ampe C, Roy L, Bourmeyster N, Constantin B.
    FASEB J; 2013 Jan 19; 27(1):123-34. PubMed ID: 23047898
    [Abstract] [Full Text] [Related]

  • 6. Phosphorylation and activation of the Rac1 and Cdc42 GEF Asef in A431 cells stimulated by EGF.
    Itoh RE, Kiyokawa E, Aoki K, Nishioka T, Akiyama T, Matsuda M.
    J Cell Sci; 2008 Aug 15; 121(Pt 16):2635-42. PubMed ID: 18653540
    [Abstract] [Full Text] [Related]

  • 7. BCR-ABL and constitutively active erythropoietin receptor (cEpoR) activate distinct mechanisms for growth factor-independence and inhibition of apoptosis in Ba/F3 cell line.
    Ahmed M, Dusanter-Fourt I, Bernard M, Mayeux P, Hawley RG, Bennardo T, Novault S, Bonnet ML, Gisselbrecht S, Varet B, Turhan AG.
    Oncogene; 1998 Jan 29; 16(4):489-96. PubMed ID: 9484838
    [Abstract] [Full Text] [Related]

  • 8. p210BCR/ABL, p190BCR/ABL, and TEL/ABL activate similar signal transduction pathways in hematopoietic cell lines.
    Okuda K, Golub TR, Gilliland DG, Griffin JD.
    Oncogene; 1996 Sep 19; 13(6):1147-52. PubMed ID: 8808688
    [Abstract] [Full Text] [Related]

  • 9. p38 MAPK-mediated activation of NF-kappaB by the RhoGEF domain of Bcr.
    Korus M, Mahon GM, Cheng L, Whitehead IP.
    Oncogene; 2002 Jul 11; 21(30):4601-12. PubMed ID: 12096337
    [Abstract] [Full Text] [Related]

  • 10. Bcr-Abl has a greater intrinsic capacity than v-Abl to induce the neoplastic expansion of myeloid cells.
    Gross AW, Ren R.
    Oncogene; 2000 Dec 14; 19(54):6286-96. PubMed ID: 11175343
    [Abstract] [Full Text] [Related]

  • 11. Bcr/Abl P190 interaction with Spa-1, a GTPase activating protein for the small GTPase Rap1.
    Yi SJ, Lee HT, Groffen J, Heisterkamp N.
    Int J Mol Med; 2008 Oct 14; 22(4):453-8. PubMed ID: 18813851
    [Abstract] [Full Text] [Related]

  • 12. Regulation of RhoA GTP hydrolysis by the GTPase-activating proteins p190, p50RhoGAP, Bcr, and 3BP-1.
    Zhang B, Zheng Y.
    Biochemistry; 1998 Apr 14; 37(15):5249-57. PubMed ID: 9548756
    [Abstract] [Full Text] [Related]

  • 13. BCR and its mutants, the reciprocal t(9;22)-associated ABL/BCR fusion proteins, differentially regulate the cytoskeleton and cell motility.
    Zheng X, Güller S, Beissert T, Puccetti E, Ruthardt M.
    BMC Cancer; 2006 Nov 07; 6():262. PubMed ID: 17090304
    [Abstract] [Full Text] [Related]

  • 14. Interleukin-3 and p210 BCR/ABL activate both unique and overlapping pathways of signal transduction in a factor-dependent myeloid cell line.
    Matulonis U, Salgia R, Okuda K, Druker B, Griffin JD.
    Exp Hematol; 1993 Oct 07; 21(11):1460-6. PubMed ID: 8405226
    [Abstract] [Full Text] [Related]

  • 15. Role of SPA-1 in phenotypes of chronic myelogenous leukemia induced by BCR-ABL-expressing hematopoietic progenitors in a mouse model.
    Kometani K, Aoki M, Kawamata S, Shinozuka Y, Era T, Taniwaki M, Hattori M, Minato N.
    Cancer Res; 2006 Oct 15; 66(20):9967-76. PubMed ID: 17047059
    [Abstract] [Full Text] [Related]

  • 16. Ribozyme-mediated inhibition of a Philadelphia chromosome-positive acute lymphoblastic leukemia cell line expressing the p190 bcr-abl oncogene.
    Snyder DS, Wu Y, McMahon R, Yu L, Rossi JJ, Forman SJ.
    Biol Blood Marrow Transplant; 1997 Oct 15; 3(4):179-86. PubMed ID: 9360779
    [Abstract] [Full Text] [Related]

  • 17. Activation of Rac1 and the exchange factor Vav3 are involved in NPM-ALK signaling in anaplastic large cell lymphomas.
    Colomba A, Courilleau D, Ramel D, Billadeau DD, Espinos E, Delsol G, Payrastre B, Gaits-Iacovoni F.
    Oncogene; 2008 Apr 24; 27(19):2728-36. PubMed ID: 17998938
    [Abstract] [Full Text] [Related]

  • 18. Vav cooperates with Ras to transform rodent fibroblasts but is not a Ras GDP/GTP exchange factor.
    Bustelo XR, Suen KL, Leftheris K, Meyers CA, Barbacid M.
    Oncogene; 1994 Aug 24; 9(8):2405-13. PubMed ID: 8036025
    [Abstract] [Full Text] [Related]

  • 19. Vav transformation requires activation of multiple GTPases and regulation of gene expression.
    Palmby TR, Abe K, Karnoub AE, Der CJ.
    Mol Cancer Res; 2004 Dec 24; 2(12):702-11. PubMed ID: 15634759
    [Abstract] [Full Text] [Related]

  • 20. The hematopoietically expressed vav proto-oncogene shares homology with the dbl GDP-GTP exchange factor, the bcr gene and a yeast gene (CDC24) involved in cytoskeletal organization.
    Adams JM, Houston H, Allen J, Lints T, Harvey R.
    Oncogene; 1992 Apr 24; 7(4):611-8. PubMed ID: 1565462
    [Abstract] [Full Text] [Related]

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