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Journal Abstract Search

219 related items for PubMed ID: 18167354

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  • 4. Polyglutamine length-dependent interaction of Hsp40 and Hsp70 family chaperones with truncated N-terminal huntingtin: their role in suppression of aggregation and cellular toxicity.
    Jana NR, Tanaka M, Wang Gh, Nukina N.
    Hum Mol Genet; 2000 Aug 12; 9(13):2009-18. PubMed ID: 10942430
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  • 5. Altered proteasomal function due to the expression of polyglutamine-expanded truncated N-terminal huntingtin induces apoptosis by caspase activation through mitochondrial cytochrome c release.
    Jana NR, Zemskov EA, Wang Gh, Nukina N.
    Hum Mol Genet; 2001 May 01; 10(10):1049-59. PubMed ID: 11331615
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  • 6. R6/2 neurons with intranuclear inclusions survive for prolonged periods in the brains of chimeric mice.
    Reiner A, Del Mar N, Deng YP, Meade CA, Sun Z, Goldowitz D.
    J Comp Neurol; 2007 Dec 20; 505(6):603-29. PubMed ID: 17948889
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  • 7. BAG-1 associates with the polyglutamine-expanded huntingtin aggregates.
    Jana NR, Nukina N.
    Neurosci Lett; 2005 Apr 22; 378(3):171-5. PubMed ID: 15781153
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  • 8. Decreased expression of hypothalamic neuropeptides in Huntington disease transgenic mice with expanded polyglutamine-EGFP fluorescent aggregates.
    Kotliarova S, Jana NR, Sakamoto N, Kurosawa M, Miyazaki H, Nekooki M, Doi H, Machida Y, Wong HK, Suzuki T, Uchikawa C, Kotliarov Y, Uchida K, Nagao Y, Nagaoka U, Tamaoka A, Oyanagi K, Oyama F, Nukina N.
    J Neurochem; 2005 May 22; 93(3):641-53. PubMed ID: 15836623
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  • 9. Cross-seeding fibrillation of Q/N-rich proteins offers new pathomechanism of polyglutamine diseases.
    Furukawa Y, Kaneko K, Matsumoto G, Kurosawa M, Nukina N.
    J Neurosci; 2009 Apr 22; 29(16):5153-62. PubMed ID: 19386911
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  • 10. Differential recruitment of UBQLN2 to nuclear inclusions in the polyglutamine diseases HD and SCA3.
    Zeng L, Wang B, Merillat SA, Minakawa EN, Perkins MD, Ramani B, Tallaksen-Greene SJ, Costa MDC, Albin RL, Paulson HL.
    Neurobiol Dis; 2015 Oct 22; 82():281-288. PubMed ID: 26141599
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  • 11. Ubiquitin-conjugating enzyme E2-25K increases aggregate formation and cell death in polyglutamine diseases.
    de Pril R, Fischer DF, Roos RA, van Leeuwen FW.
    Mol Cell Neurosci; 2007 Jan 22; 34(1):10-9. PubMed ID: 17092742
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  • 13. Huntingtin inclusions do not deplete polyglutamine-containing transcription factors in HD mice.
    Yu ZX, Li SH, Nguyen HP, Li XJ.
    Hum Mol Genet; 2002 Apr 15; 11(8):905-14. PubMed ID: 11971872
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  • 14. Differential morphology and composition of inclusions in the R6/2 mouse and PC12 cell models of Huntington's disease.
    Wanderer J, Morton AJ.
    Histochem Cell Biol; 2007 May 15; 127(5):473-84. PubMed ID: 17285342
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  • 15. Selection of behaviors and segmental coordination during larval locomotion is disrupted by nuclear polyglutamine inclusions in a new Drosophila Huntington's disease-like model.
    Nishimura Y, Yalgin C, Akimoto S, Doumanis J, Sasajima R, Nukina N, Miyakawa H, Moore AW, Morimoto T.
    J Neurogenet; 2010 Dec 15; 24(4):194-206. PubMed ID: 21087194
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  • 16. Proteomics of polyglutamine aggregates.
    Mitsui K, Doi H, Nukina N.
    Methods Enzymol; 2006 Dec 15; 412():63-76. PubMed ID: 17046652
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  • 18. Calmodulin regulates transglutaminase 2 cross-linking of huntingtin.
    Zainelli GM, Ross CA, Troncoso JC, Fitzgerald JK, Muma NA.
    J Neurosci; 2004 Feb 25; 24(8):1954-61. PubMed ID: 14985437
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  • 20. Inactivation of Drosophila Apaf-1 related killer suppresses formation of polyglutamine aggregates and blocks polyglutamine pathogenesis.
    Sang TK, Li C, Liu W, Rodriguez A, Abrams JM, Zipursky SL, Jackson GR.
    Hum Mol Genet; 2005 Feb 01; 14(3):357-72. PubMed ID: 15590702
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