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Journal Abstract Search

388 related items for PubMed ID: 18182777

  • 1. Role of aberrant glycosylation of IgA1 molecules in the pathogenesis of IgA nephropathy.
    Mestecky J, Tomana M, Moldoveanu Z, Julian BA, Suzuki H, Matousovic K, Renfrow MB, Novak L, Wyatt RJ, Novak J.
    Kidney Blood Press Res; 2008; 31(1):29-37. PubMed ID: 18182777
    [Abstract] [Full Text] [Related]

  • 2. Circulating immune complexes in IgA nephropathy consist of IgA1 with galactose-deficient hinge region and antiglycan antibodies.
    Tomana M, Novak J, Julian BA, Matousovic K, Konecny K, Mestecky J.
    J Clin Invest; 1999 Jul; 104(1):73-81. PubMed ID: 10393701
    [Abstract] [Full Text] [Related]

  • 3. IgA nephropathy and Henoch-Schoenlein purpura nephritis: aberrant glycosylation of IgA1, formation of IgA1-containing immune complexes, and activation of mesangial cells.
    Novak J, Moldoveanu Z, Renfrow MB, Yanagihara T, Suzuki H, Raska M, Hall S, Brown R, Huang WQ, Goepfert A, Kilian M, Poulsen K, Tomana M, Wyatt RJ, Julian BA, Mestecky J.
    Contrib Nephrol; 2007 Jul; 157():134-8. PubMed ID: 17495451
    [Abstract] [Full Text] [Related]

  • 4. Recognition of galactose-deficient O-glycans in the hinge region of IgA1 by N-acetylgalactosamine-specific snail lectins: a comparative binding study.
    Gomes MM, Suzuki H, Brooks MT, Tomana M, Moldoveanu Z, Mestecky J, Julian BA, Novak J, Herr AB.
    Biochemistry; 2010 Jul 13; 49(27):5671-82. PubMed ID: 20507092
    [Abstract] [Full Text] [Related]

  • 5. IgA nephropathy: current views of immune complex formation.
    Mestecky J, Suzuki H, Yanagihara T, Moldoveanu Z, Tomana M, Matousovic K, Julian BA, Novak J.
    Contrib Nephrol; 2007 Jul 13; 157():56-63. PubMed ID: 17495438
    [Abstract] [Full Text] [Related]

  • 6. IgA nephropathy: characterization of IgG antibodies specific for galactose-deficient IgA1.
    Suzuki H, Moldoveanu Z, Hall S, Brown R, Julian BA, Wyatt RJ, Tomana M, Tomino Y, Novak J, Mestecky J.
    Contrib Nephrol; 2007 Jul 13; 157():129-33. PubMed ID: 17495450
    [Abstract] [Full Text] [Related]

  • 7. The glycans deficiencies of macromolecular IgA1 is a contributory factor of variable pathological phenotypes of IgA nephropathy.
    Xu LX, Yan Y, Zhang JJ, Zhang Y, Zhao MH.
    Clin Exp Immunol; 2005 Dec 13; 142(3):569-75. PubMed ID: 16297170
    [Abstract] [Full Text] [Related]

  • 8. Aberrantly glycosylated serum IgA1 are closely associated with pathologic phenotypes of IgA nephropathy.
    Xu LX, Zhao MH.
    Kidney Int; 2005 Jul 13; 68(1):167-72. PubMed ID: 15954905
    [Abstract] [Full Text] [Related]

  • 9. Pathogenic role of glycan-specific IgG antibodies in IgA nephropathy.
    Zhao YF, Zhu L, Liu LJ, Shi SF, Lv JC, Zhang H.
    BMC Nephrol; 2017 Sep 29; 18(1):301. PubMed ID: 28969604
    [Abstract] [Full Text] [Related]

  • 10. Enzymatic sialylation of IgA1 O-glycans: implications for studies of IgA nephropathy.
    Takahashi K, Raska M, Stuchlova Horynova M, Hall SD, Poulsen K, Kilian M, Hiki Y, Yuzawa Y, Moldoveanu Z, Julian BA, Renfrow MB, Novak J.
    PLoS One; 2014 Sep 29; 9(2):e99026. PubMed ID: 24918438
    [Abstract] [Full Text] [Related]

  • 11. N-acetylgalactosaminide α2,6-sialyltransferase II is a candidate enzyme for sialylation of galactose-deficient IgA1, the key autoantigen in IgA nephropathy.
    Stuchlova Horynova M, Vrablikova A, Stewart TJ, Takahashi K, Czernekova L, Yamada K, Suzuki H, Julian BA, Renfrow MB, Novak J, Raska M.
    Nephrol Dial Transplant; 2015 Feb 29; 30(2):234-8. PubMed ID: 25281698
    [Abstract] [Full Text] [Related]

  • 12. Sialylation of IgG inhibits the formation of galactose-deficient IgA1-containing immune complexes and protects mesangial cells from injury in IgA nephropathy.
    Liu Y, Li H, Yu H, Wang F, Jia J, Yan T.
    BMC Nephrol; 2022 Jan 11; 23(1):25. PubMed ID: 35016642
    [Abstract] [Full Text] [Related]

  • 13. Aberrant glycosylation of IgA1 and anti-glycan antibodies in IgA nephropathy: role of mucosal immune system.
    Novak J, Moldoveanu Z, Julian BA, Raska M, Wyatt RJ, Suzuki Y, Tomino Y, Gharavi AG, Mestecky J, Suzuki H.
    Adv Otorhinolaryngol; 2011 Jan 11; 72():60-3. PubMed ID: 21865691
    [Abstract] [Full Text] [Related]

  • 14. IgA glycosylation and IgA immune complexes in the pathogenesis of IgA nephropathy.
    Novak J, Julian BA, Tomana M, Mestecky J.
    Semin Nephrol; 2008 Jan 11; 28(1):78-87. PubMed ID: 18222349
    [Abstract] [Full Text] [Related]

  • 15. Elucidating heterogeneity of IgA1 hinge-region O-glycosylation by use of MALDI-TOF/TOF mass spectrometry: role of cysteine alkylation during sample processing.
    Franc V, Řehulka P, Raus M, Stulík J, Novak J, Renfrow MB, Šebela M.
    J Proteomics; 2013 Oct 30; 92():299-312. PubMed ID: 23891555
    [Abstract] [Full Text] [Related]

  • 16. O-glycosylation of IgA1 and the pathogenesis of an autoimmune disease IgA nephropathy.
    Novak J, King RG, Yother J, Renfrow MB, Green TJ.
    Glycobiology; 2024 Sep 30; 34(11):. PubMed ID: 39095059
    [Abstract] [Full Text] [Related]

  • 17. Glycosylation profile of differently charged IgA1 and their binding characteristics to cultured mesangial cells in IgA nephropathy.
    Leung JC, Chan LY, Tang SC, Tam PC, Fenn J, Lai KN.
    Nephron Exp Nephrol; 2007 Sep 30; 107(3):e107-18. PubMed ID: 17957128
    [Abstract] [Full Text] [Related]

  • 18. Increased sialylation of polymeric lambda-IgA1 in patients with IgA nephropathy.
    Leung JC, Tang SC, Chan DT, Lui SL, Lai KN.
    J Clin Lab Anal; 2002 Sep 30; 16(1):11-9. PubMed ID: 11835525
    [Abstract] [Full Text] [Related]

  • 19. Difference in IgA1 O-glycosylation between IgA deposition donors and IgA nephropathy recipients.
    Nakazawa S, Imamura R, Kawamura M, Kato T, Abe T, Namba T, Iwatani H, Yamanaka K, Uemura M, Kishikawa H, Nishimura K, Oka K, Tajiri M, Wada Y, Nonomura N.
    Biochem Biophys Res Commun; 2019 Jan 22; 508(4):1106-1112. PubMed ID: 30553446
    [Abstract] [Full Text] [Related]

  • 20. Aberrant glycosylation in IgA nephropathy (IgAN).
    Coppo R, Amore A.
    Kidney Int; 2004 May 22; 65(5):1544-7. PubMed ID: 15086888
    [Abstract] [Full Text] [Related]

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