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PUBMED FOR HANDHELDS

Journal Abstract Search


1269 related items for PubMed ID: 18193822

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  • 5. Mitochondria induce oxidative stress, generation of reactive oxygen species and redox state unbalance of the eye lens leading to human cataract formation: disruption of redox lens organization by phospholipid hydroperoxides as a common basis for cataract disease.
    Babizhayev MA.
    Cell Biochem Funct; 2011 Apr; 29(3):183-206. PubMed ID: 21381059
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  • 7. Protective role of arjunolic acid in response to streptozotocin-induced type-I diabetes via the mitochondrial dependent and independent pathways.
    Manna P, Sinha M, Sil PC.
    Toxicology; 2009 Mar 04; 257(1-2):53-63. PubMed ID: 19133311
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  • 8. NecroX as a novel class of mitochondrial reactive oxygen species and ONOO⁻ scavenger.
    Kim HJ, Koo SY, Ahn BH, Park O, Park DH, Seo DO, Won JH, Yim HJ, Kwak HS, Park HS, Chung CW, Oh YL, Kim SH.
    Arch Pharm Res; 2010 Nov 04; 33(11):1813-23. PubMed ID: 21116785
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  • 9. Hemigramicidin-TEMPO conjugates: novel mitochondria-targeted anti-oxidants.
    Fink MP, Macias CA, Xiao J, Tyurina YY, Jiang J, Belikova N, Delude RL, Greenberger JS, Kagan VE, Wipf P.
    Biochem Pharmacol; 2007 Sep 15; 74(6):801-9. PubMed ID: 17601494
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  • 10. Involvement of mitochondrial alteration and reactive oxygen species generation in Taiwan cobra cardiotoxin-induced apoptotic death of human neuroblastoma SK-N-SH cells.
    Chen KC, Lin SR, Chang LS.
    Toxicon; 2008 Aug 01; 52(2):361-8. PubMed ID: 18619991
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  • 14. Novel 2-substituted nitronyl nitroxides as free radical scavengers: synthesis, biological evaluation and structure-activity relationship.
    Wu Y, Bi L, Bi W, Li Z, Zhao M, Wang C, Ju J, Peng S.
    Bioorg Med Chem; 2006 Aug 15; 14(16):5711-20. PubMed ID: 16650996
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  • 15. Mitochondrial reactive oxygen species regulate the temporal activation of nuclear factor kappaB to modulate tumour necrosis factor-induced apoptosis: evidence from mitochondria-targeted antioxidants.
    Hughes G, Murphy MP, Ledgerwood EC.
    Biochem J; 2005 Jul 01; 389(Pt 1):83-9. PubMed ID: 15727562
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  • 16. An epigrammatic (abridged) recounting of the myriad tales of astonishing deeds and dire consequences pertaining to nitric oxide and reactive oxygen species in mitochondria with an ancillary missive concerning the origins of apoptosis.
    Heck DE, Kagan VE, Shvedova AA, Laskin JD.
    Toxicology; 2005 Mar 15; 208(2):259-71. PubMed ID: 15691590
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  • 17. Isofraxidin, a potent reactive oxygen species (ROS) scavenger, protects human leukemia cells from radiation-induced apoptosis via ROS/mitochondria pathway in p53-independent manner.
    Li P, Zhao QL, Wu LH, Jawaid P, Jiao YF, Kadowaki M, Kondo T.
    Apoptosis; 2014 Jun 15; 19(6):1043-53. PubMed ID: 24692054
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  • 18. Mitochondrial alterations related to programmed cell death in tobacco cells under aluminium stress.
    Panda SK, Yamamoto Y, Kondo H, Matsumoto H.
    C R Biol; 2008 Aug 15; 331(8):597-610. PubMed ID: 18606389
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  • 19. The causes of cancer revisited: "mitochondrial malignancy" and ROS-induced oncogenic transformation - why mitochondria are targets for cancer therapy.
    Ralph SJ, Rodríguez-Enríquez S, Neuzil J, Saavedra E, Moreno-Sánchez R.
    Mol Aspects Med; 2010 Apr 15; 31(2):145-70. PubMed ID: 20206201
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  • 20. Mitochondria-dependent reactive oxygen species-mediated programmed cell death induced by 3,3'-diindolylmethane through inhibition of F0F1-ATP synthase in unicellular protozoan parasite Leishmania donovani.
    Roy A, Ganguly A, BoseDasgupta S, Das BB, Pal C, Jaisankar P, Majumder HK.
    Mol Pharmacol; 2008 Nov 15; 74(5):1292-307. PubMed ID: 18703668
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