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484 related items for PubMed ID: 18276596
1. Discovery of TBC1D1 as an insulin-, AICAR-, and contraction-stimulated signaling nexus in mouse skeletal muscle. Taylor EB, An D, Kramer HF, Yu H, Fujii NL, Roeckl KS, Bowles N, Hirshman MF, Xie J, Feener EP, Goodyear LJ. J Biol Chem; 2008 Apr 11; 283(15):9787-96. PubMed ID: 18276596 [Abstract] [Full Text] [Related]
2. Inhibition of GLUT4 translocation by Tbc1d1, a Rab GTPase-activating protein abundant in skeletal muscle, is partially relieved by AMP-activated protein kinase activation. Chavez JA, Roach WG, Keller SR, Lane WS, Lienhard GE. J Biol Chem; 2008 Apr 04; 283(14):9187-95. PubMed ID: 18258599 [Abstract] [Full Text] [Related]
3. Thr649Ala-AS160 knock-in mutation does not impair contraction/AICAR-induced glucose transport in mouse muscle. Ducommun S, Wang HY, Sakamoto K, MacKintosh C, Chen S. Am J Physiol Endocrinol Metab; 2012 May 15; 302(9):E1036-43. PubMed ID: 22318952 [Abstract] [Full Text] [Related]
4. Inhibition of contraction-stimulated AMP-activated protein kinase inhibits contraction-stimulated increases in PAS-TBC1D1 and glucose transport without altering PAS-AS160 in rat skeletal muscle. Funai K, Cartee GD. Diabetes; 2009 May 15; 58(5):1096-104. PubMed ID: 19208911 [Abstract] [Full Text] [Related]
5. The Rab-GTPase-activating protein TBC1D1 regulates skeletal muscle glucose metabolism. Szekeres F, Chadt A, Tom RZ, Deshmukh AS, Chibalin AV, Björnholm M, Al-Hasani H, Zierath JR. Am J Physiol Endocrinol Metab; 2012 Aug 15; 303(4):E524-33. PubMed ID: 22693207 [Abstract] [Full Text] [Related]
6. Distinct signals regulate AS160 phosphorylation in response to insulin, AICAR, and contraction in mouse skeletal muscle. Kramer HF, Witczak CA, Fujii N, Jessen N, Taylor EB, Arnolds DE, Sakamoto K, Hirshman MF, Goodyear LJ. Diabetes; 2006 Jul 15; 55(7):2067-76. PubMed ID: 16804077 [Abstract] [Full Text] [Related]
7. Contraction-stimulated glucose transport in rat skeletal muscle is sustained despite reversal of increased PAS-phosphorylation of AS160 and TBC1D1. Funai K, Cartee GD. J Appl Physiol (1985); 2008 Dec 15; 105(6):1788-95. PubMed ID: 18818383 [Abstract] [Full Text] [Related]
8. Contraction regulates site-specific phosphorylation of TBC1D1 in skeletal muscle. Vichaiwong K, Purohit S, An D, Toyoda T, Jessen N, Hirshman MF, Goodyear LJ. Biochem J; 2010 Oct 15; 431(2):311-20. PubMed ID: 20701589 [Abstract] [Full Text] [Related]
9. Identification of a novel phosphorylation site on TBC1D4 regulated by AMP-activated protein kinase in skeletal muscle. Treebak JT, Taylor EB, Witczak CA, An D, Toyoda T, Koh HJ, Xie J, Feener EP, Wojtaszewski JF, Hirshman MF, Goodyear LJ. Am J Physiol Cell Physiol; 2010 Feb 15; 298(2):C377-85. PubMed ID: 19923418 [Abstract] [Full Text] [Related]
10. Disruption of AMPKalpha1 signaling prevents AICAR-induced inhibition of AS160/TBC1D4 phosphorylation and glucose uptake in primary rat adipocytes. Gaidhu MP, Perry RL, Noor F, Ceddia RB. Mol Endocrinol; 2010 Jul 15; 24(7):1434-40. PubMed ID: 20501641 [Abstract] [Full Text] [Related]
11. Prior treatment with the AMPK activator AICAR induces subsequently enhanced glucose uptake in isolated skeletal muscles from 24-month-old rats. Oki K, Arias EB, Kanzaki M, Cartee GD. Appl Physiol Nutr Metab; 2018 Aug 15; 43(8):795-805. PubMed ID: 29518344 [Abstract] [Full Text] [Related]
12. Increased phosphorylation of Akt substrate of 160 kDa (AS160) in rat skeletal muscle in response to insulin or contractile activity. Bruss MD, Arias EB, Lienhard GE, Cartee GD. Diabetes; 2005 Jan 15; 54(1):41-50. PubMed ID: 15616009 [Abstract] [Full Text] [Related]
13. Oligomeric resistin impairs insulin and AICAR-stimulated glucose uptake in mouse skeletal muscle by inhibiting GLUT4 translocation. Jørgensen SB, Honeyman J, Oakhill JS, Fazakerley D, Stöckli J, Kemp BE, Steinberg GR. Am J Physiol Endocrinol Metab; 2009 Jul 15; 297(1):E57-66. PubMed ID: 19435854 [Abstract] [Full Text] [Related]
14. Exercise increases TBC1D1 phosphorylation in human skeletal muscle. Jessen N, An D, Lihn AS, Nygren J, Hirshman MF, Thorell A, Goodyear LJ. Am J Physiol Endocrinol Metab; 2011 Jul 15; 301(1):E164-71. PubMed ID: 21505148 [Abstract] [Full Text] [Related]
15. Whole body glucoregulation and tissue-specific glucose uptake in a novel Akt substrate of 160 kDa knockout rat model. Arias EB, Zheng X, Agrawal S, Cartee GD. PLoS One; 2019 Jul 15; 14(4):e0216236. PubMed ID: 31034517 [Abstract] [Full Text] [Related]
16. A Tbc1d1 Ser231Ala-knockin mutation partially impairs AICAR- but not exercise-induced muscle glucose uptake in mice. Chen Q, Xie B, Zhu S, Rong P, Sheng Y, Ducommun S, Chen L, Quan C, Li M, Sakamoto K, MacKintosh C, Chen S, Wang HY. Diabetologia; 2017 Feb 15; 60(2):336-345. PubMed ID: 27826658 [Abstract] [Full Text] [Related]
17. “Deletion of both Rab-GTPase–activating proteins TBC1D1 and TBC1D4 in mice eliminates insulin- and AICAR-stimulated glucose transport [corrected]. Chadt A, Immisch A, de Wendt C, Springer C, Zhou Z, Stermann T, Holman GD, Loffing-Cueni D, Loffing J, Joost HG, Al-Hasani H. Diabetes; 2015 Mar 15; 64(3):746-59. PubMed ID: 25249576 [Abstract] [Full Text] [Related]
18. Genetic disruption of AMPK signaling abolishes both contraction- and insulin-stimulated TBC1D1 phosphorylation and 14-3-3 binding in mouse skeletal muscle. Pehmøller C, Treebak JT, Birk JB, Chen S, Mackintosh C, Hardie DG, Richter EA, Wojtaszewski JF. Am J Physiol Endocrinol Metab; 2009 Sep 15; 297(3):E665-75. PubMed ID: 19531644 [Abstract] [Full Text] [Related]
19. Insulin-stimulated phosphorylation of the Rab GTPase-activating protein TBC1D1 regulates GLUT4 translocation. Peck GR, Chavez JA, Roach WG, Budnik BA, Lane WS, Karlsson HK, Zierath JR, Lienhard GE. J Biol Chem; 2009 Oct 30; 284(44):30016-23. PubMed ID: 19740738 [Abstract] [Full Text] [Related]
20. Restoring AS160 phosphorylation rescues skeletal muscle insulin resistance and fatty acid oxidation while not reducing intramuscular lipids. Alkhateeb H, Chabowski A, Glatz JF, Gurd B, Luiken JJ, Bonen A. Am J Physiol Endocrinol Metab; 2009 Nov 30; 297(5):E1056-66. PubMed ID: 19724017 [Abstract] [Full Text] [Related] Page: [Next] [New Search]