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239 related items for PubMed ID: 18323749

  • 1. Increased aerobic glycolysis through beta2 stimulation is a common mechanism involved in lactate formation during shock states.
    Levy B, Desebbe O, Montemont C, Gibot S.
    Shock; 2008 Oct; 30(4):417-21. PubMed ID: 18323749
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  • 2. Adrenergic blockade reduces skeletal muscle glycolysis and Na(+), K(+)-ATPase activity during hemorrhage.
    McCarter FD, James JH, Luchette FA, Wang L, Friend LA, King JK, Evans JM, George MA, Fischer JE.
    J Surg Res; 2001 Aug; 99(2):235-44. PubMed ID: 11469892
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  • 3. Relation between muscle Na+K+ ATPase activity and raised lactate concentrations in septic shock: a prospective study.
    Levy B, Gibot S, Franck P, Cravoisy A, Bollaert PE.
    Lancet; 2001 Aug; 365(9462):871-5. PubMed ID: 15752531
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  • 4. Increased muscle-to-serum lactate gradient predicts progression towards septic shock in septic patients.
    Levy B, Perez P, Gibot S, Gerard A.
    Intensive Care Med; 2010 Oct; 36(10):1703-1709. PubMed ID: 20577713
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  • 5. Hypoxia is not the sole cause of lactate production during shock.
    Luchette FA, Jenkins WA, Friend LA, Su C, Fischer JE, James JH.
    J Trauma; 2002 Mar; 52(3):415-9. PubMed ID: 11901313
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  • 6. Linkage of aerobic glycolysis to sodium-potassium transport in rat skeletal muscle. Implications for increased muscle lactate production in sepsis.
    James JH, Fang CH, Schrantz SJ, Hasselgren PO, Paul RJ, Fischer JE.
    J Clin Invest; 1996 Nov 15; 98(10):2388-97. PubMed ID: 8941658
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  • 7. Myocardial lactate deprivation is associated with decreased cardiovascular performance, decreased myocardial energetics, and early death in endotoxic shock.
    Levy B, Mansart A, Montemont C, Gibot S, Mallie JP, Regnault V, Lecompte T, Lacolley P.
    Intensive Care Med; 2007 Mar 15; 33(3):495-502. PubMed ID: 17242933
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  • 8. Role of skeletal muscle Na+-K+ ATPase activity in increased lactate production in sub-acute sepsis.
    McCarter FD, Nierman SR, James JH, Wang L, King JK, Friend LA, Fischer JE.
    Life Sci; 2002 Mar 08; 70(16):1875-88. PubMed ID: 12005173
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  • 13. Increased skeletal muscle Na+, K+-ATPase activity as a cause of increased lactate production after hemorrhagic shock.
    Luchette FA, Friend LA, Brown CC, Upputuri RK, James JH.
    J Trauma; 1998 May 08; 44(5):796-801; discussion 801-3. PubMed ID: 9603080
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  • 16. Catecholamine regulation of local lactate production in vivo in skeletal muscle and adipose tissue: role of -adrenoreceptor subtypes.
    Qvisth V, Hagström-Toft E, Enoksson S, Bolinder J.
    J Clin Endocrinol Metab; 2008 Jan 08; 93(1):240-6. PubMed ID: 17986640
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  • 17. [Impact of intracellular Na+ concentration alteration on intracellular aerobic glycolysis in skeletal muscles in sepsis].
    Chai J, Diao L, Sheng Z.
    Zhonghua Yi Xue Za Zhi; 1999 Jul 08; 79(7):546-8. PubMed ID: 11715429
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  • 18. Effects of epinephrine and norepinephrine on hemodynamics, oxidative metabolism, and organ energetics in endotoxemic rats.
    Levy B, Mansart A, Bollaert PE, Franck P, Mallie JP.
    Intensive Care Med; 2003 Feb 08; 29(2):292-300. PubMed ID: 12594589
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