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150 related items for PubMed ID: 18337461

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  • 3. Superoxide dismutase 1 mutants related to amyotrophic lateral sclerosis induce endoplasmic stress in neuro2a cells.
    Oh YK, Shin KS, Yuan J, Kang SJ.
    J Neurochem; 2008 Feb; 104(4):993-1005. PubMed ID: 18233996
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  • 4. Wild-type human SOD1 overexpression does not accelerate motor neuron disease in mice expressing murine Sod1 G86R.
    Audet JN, Gowing G, Julien JP.
    Neurobiol Dis; 2010 Oct; 40(1):245-50. PubMed ID: 20573565
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  • 5. Rab1-dependent ER-Golgi transport dysfunction is a common pathogenic mechanism in SOD1, TDP-43 and FUS-associated ALS.
    Soo KY, Halloran M, Sundaramoorthy V, Parakh S, Toth RP, Southam KA, McLean CA, Lock P, King A, Farg MA, Atkin JD.
    Acta Neuropathol; 2015 Nov; 130(5):679-97. PubMed ID: 26298469
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  • 6. Extracellular wildtype and mutant SOD1 induces ER-Golgi pathology characteristic of amyotrophic lateral sclerosis in neuronal cells.
    Sundaramoorthy V, Walker AK, Yerbury J, Soo KY, Farg MA, Hoang V, Zeineddine R, Spencer D, Atkin JD.
    Cell Mol Life Sci; 2013 Nov; 70(21):4181-95. PubMed ID: 23765103
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  • 9. Focal loss of the glutamate transporter EAAT2 in a transgenic rat model of SOD1 mutant-mediated amyotrophic lateral sclerosis (ALS).
    Howland DS, Liu J, She Y, Goad B, Maragakis NJ, Kim B, Erickson J, Kulik J, DeVito L, Psaltis G, DeGennaro LJ, Cleveland DW, Rothstein JD.
    Proc Natl Acad Sci U S A; 2002 Feb 05; 99(3):1604-9. PubMed ID: 11818550
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  • 10. Toxicity of familial ALS-linked SOD1 mutants from selective recruitment to spinal mitochondria.
    Liu J, Lillo C, Jonsson PA, Vande Velde C, Ward CM, Miller TM, Subramaniam JR, Rothstein JD, Marklund S, Andersen PM, Brännström T, Gredal O, Wong PC, Williams DS, Cleveland DW.
    Neuron; 2004 Jul 08; 43(1):5-17. PubMed ID: 15233913
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  • 11. Mutant Cu, Zn superoxide dismutase that causes motoneuron degeneration is present in mitochondria in the CNS.
    Higgins CM, Jung C, Ding H, Xu Z.
    J Neurosci; 2002 Mar 15; 22(6):RC215. PubMed ID: 11886899
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  • 12. Mitochondrial localization of mutant superoxide dismutase 1 triggers caspase-dependent cell death in a cellular model of familial amyotrophic lateral sclerosis.
    Takeuchi H, Kobayashi Y, Ishigaki S, Doyu M, Sobue G.
    J Biol Chem; 2002 Dec 27; 277(52):50966-72. PubMed ID: 12393885
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  • 13. Stress signaling from the endoplasmic reticulum: A central player in the pathogenesis of amyotrophic lateral sclerosis.
    Walker AK, Atkin JD.
    IUBMB Life; 2011 Sep 27; 63(9):754-63. PubMed ID: 21834058
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  • 18. Mouse motor neuron disease caused by truncated SOD1 with or without C-terminal modification.
    Watanabe Y, Yasui K, Nakano T, Doi K, Fukada Y, Kitayama M, Ishimoto M, Kurihara S, Kawashima M, Fukuda H, Adachi Y, Inoue T, Nakashima K.
    Brain Res Mol Brain Res; 2005 Apr 27; 135(1-2):12-20. PubMed ID: 15857664
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  • 19. Mutant SOD1 inhibits ER-Golgi transport in amyotrophic lateral sclerosis.
    Atkin JD, Farg MA, Soo KY, Walker AK, Halloran M, Turner BJ, Nagley P, Horne MK.
    J Neurochem; 2014 Apr 27; 129(1):190-204. PubMed ID: 24134191
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  • 20. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
    Jaarsma D, Haasdijk ED, Grashorn JA, Hawkins R, van Duijn W, Verspaget HW, London J, Holstege JC.
    Neurobiol Dis; 2000 Dec 27; 7(6 Pt B):623-43. PubMed ID: 11114261
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