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Journal Abstract Search

129 related items for PubMed ID: 18401423

  • 1. Tie2-R849W mutant in venous malformations chronically activates a functional STAT1 to modulate gene expression.
    Hu HT, Huang YH, Chang YA, Lee CK, Jiang MJ, Wu LW.
    J Invest Dermatol; 2008 Sep; 128(9):2325-33. PubMed ID: 18401423
    [Abstract] [Full Text] [Related]

  • 2. STAT1 activation by venous malformations mutant Tie2-R849W antagonizes VEGF-A-mediated angiogenic response partly via reduced bFGF production.
    Huang YH, Wu MP, Pan SC, Su WC, Chen YW, Wu LW.
    Angiogenesis; 2013 Jan; 16(1):207-22. PubMed ID: 23086340
    [Abstract] [Full Text] [Related]

  • 3. Transgenic Expression of A Venous Malformation Related Mutation, TIE2-R849W, Significantly Induces Multiple Malformations of Zebrafish.
    Du Z, Ma HL, Zhang ZY, Zheng JW, Wang YA.
    Int J Med Sci; 2018 Jan; 15(4):385-394. PubMed ID: 29511374
    [Abstract] [Full Text] [Related]

  • 4. Review of the endothelial pathogenic mechanism of TIE2-related venous malformation.
    Du Z, Zheng J, Zhang Z, Wang Y.
    J Vasc Surg Venous Lymphat Disord; 2017 Sep; 5(5):740-748. PubMed ID: 28818232
    [Abstract] [Full Text] [Related]

  • 5. Functional analysis of a mutant form of the receptor tyrosine kinase Tie2 causing venous malformations.
    Morris PN, Dunmore BJ, Tadros A, Marchuk DA, Darland DC, D'Amore PA, Brindle NP.
    J Mol Med (Berl); 2005 Jan; 83(1):58-63. PubMed ID: 15526080
    [Abstract] [Full Text] [Related]

  • 6. AKT/FOXO1 axis links cross-talking of endothelial cell and pericyte in TIE2-mutated venous malformations.
    Si Y, Huang J, Li X, Fu Y, Xu R, Du Y, Cheng J, Jiang H.
    Cell Commun Signal; 2020 Aug 31; 18(1):139. PubMed ID: 32867785
    [Abstract] [Full Text] [Related]

  • 7. Mutant Tie2 causing venous malformation signals through Shc.
    Morris PN, Dunmore BJ, Brindle NP.
    Biochem Biophys Res Commun; 2006 Jul 21; 346(1):335-8. PubMed ID: 16756945
    [Abstract] [Full Text] [Related]

  • 8. A Tie2 kinase mutation causing venous malformations increases phosphorylation rates and enhances cooperativity.
    Kennedy MA, Xu Z, Wu Y, Sohl CD.
    Biochem Biophys Res Commun; 2019 Feb 19; 509(4):898-902. PubMed ID: 30638931
    [Abstract] [Full Text] [Related]

  • 9. Venous malformation-causative TIE2 mutations mediate an AKT-dependent decrease in PDGFB.
    Uebelhoer M, Nätynki M, Kangas J, Mendola A, Nguyen HL, Soblet J, Godfraind C, Boon LM, Eklund L, Limaye N, Vikkula M.
    Hum Mol Genet; 2013 Sep 01; 22(17):3438-48. PubMed ID: 23633549
    [Abstract] [Full Text] [Related]

  • 10. Rapamycin improves TIE2-mutated venous malformation in murine model and human subjects.
    Boscolo E, Limaye N, Huang L, Kang KT, Soblet J, Uebelhoer M, Mendola A, Natynki M, Seront E, Dupont S, Hammer J, Legrand C, Brugnara C, Eklund L, Vikkula M, Bischoff J, Boon LM.
    J Clin Invest; 2015 Sep 01; 125(9):3491-504. PubMed ID: 26258417
    [Abstract] [Full Text] [Related]

  • 11. Hereditary cutaneomucosal venous malformations are caused by TIE2 mutations with widely variable hyper-phosphorylating effects.
    Wouters V, Limaye N, Uebelhoer M, Irrthum A, Boon LM, Mulliken JB, Enjolras O, Baselga E, Berg J, Dompmartin A, Ivarsson SA, Kangesu L, Lacassie Y, Murphy J, Teebi AS, Penington A, Rieu P, Vikkula M.
    Eur J Hum Genet; 2010 Apr 01; 18(4):414-20. PubMed ID: 19888299
    [Abstract] [Full Text] [Related]

  • 12. Disorganized vascular structures in sporadic venous malformations: a possible correlation with balancing effect between Tie2 and TGF-β.
    Chen G, Ren JG, Zhang W, Sun YF, Wang FQ, Li RF, Zhang J, Zhao YF.
    Sci Rep; 2014 Jun 26; 4():5457. PubMed ID: 24966004
    [Abstract] [Full Text] [Related]

  • 13. Somatic mutations in angiopoietin receptor gene TEK cause solitary and multiple sporadic venous malformations.
    Limaye N, Wouters V, Uebelhoer M, Tuominen M, Wirkkala R, Mulliken JB, Eklund L, Boon LM, Vikkula M.
    Nat Genet; 2009 Jan 26; 41(1):118-24. PubMed ID: 19079259
    [Abstract] [Full Text] [Related]

  • 14. Classification and Tie2 mutations in spinal and soft tissue vascular anomalies.
    Zhou M, Jiang R, Zhao G, Wang L, Wang H, Li W, Li Y, Du X, Bai J.
    Gene; 2015 Oct 15; 571(1):91-6. PubMed ID: 26115772
    [Abstract] [Full Text] [Related]

  • 15. Common and specific effects of TIE2 mutations causing venous malformations.
    Nätynki M, Kangas J, Miinalainen I, Sormunen R, Pietilä R, Soblet J, Boon LM, Vikkula M, Limaye N, Eklund L.
    Hum Mol Genet; 2015 Nov 15; 24(22):6374-89. PubMed ID: 26319232
    [Abstract] [Full Text] [Related]

  • 16. Endothelial receptor tyrosine kinases activate the STAT signaling pathway: mutant Tie-2 causing venous malformations signals a distinct STAT activation response.
    Korpelainen EI, Kärkkäinen M, Gunji Y, Vikkula M, Alitalo K.
    Oncogene; 1999 Jan 07; 18(1):1-8. PubMed ID: 9926914
    [Abstract] [Full Text] [Related]

  • 17. Vascular dysmorphogenesis caused by an activating mutation in the receptor tyrosine kinase TIE2.
    Vikkula M, Boon LM, Carraway KL, Calvert JT, Diamonti AJ, Goumnerov B, Pasyk KA, Marchuk DA, Warman ML, Cantley LC, Mulliken JB, Olsen BR.
    Cell; 1996 Dec 27; 87(7):1181-90. PubMed ID: 8980225
    [Abstract] [Full Text] [Related]

  • 18. Human iPSC and CRISPR targeted gene knock-in strategy for studying the somatic TIE2L914F mutation in endothelial cells.
    Lazovic B, Nguyen HT, Ansarizadeh M, Wigge L, Kohl F, Li S, Carracedo M, Kettunen J, Krimpenfort L, Elgendy R, Richter K, De Silva L, Bilican B, Singh P, Saxena P, Jakobsson L, Hong X, Eklund L, Hicks R.
    Angiogenesis; 2024 Aug 27; 27(3):523-542. PubMed ID: 38771392
    [Abstract] [Full Text] [Related]

  • 19. Vascular endothelial growth factor activates the Tie family of receptor tyrosine kinases.
    Singh H, Milner CS, Aguilar Hernandez MM, Patel N, Brindle NP.
    Cell Signal; 2009 Aug 27; 21(8):1346-50. PubMed ID: 19376222
    [Abstract] [Full Text] [Related]

  • 20. Association study between of Tie2/angiopoietin-2 and VEGF/KDR pathway gene polymorphisms and vascular malformations.
    Zheng Q, Du J, Zhang Z, Xu J, Fu L, Cao Y, Huang X, Guo L.
    Gene; 2013 Jul 10; 523(2):195-8. PubMed ID: 23566851
    [Abstract] [Full Text] [Related]

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