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Journal Abstract Search

292 related items for PubMed ID: 18403135

  • 1. Identification and characterization of a novel folliculin-interacting protein FNIP2.
    Hasumi H, Baba M, Hong SB, Hasumi Y, Huang Y, Yao M, Valera VA, Linehan WM, Schmidt LS.
    Gene; 2008 May 31; 415(1-2):60-7. PubMed ID: 18403135
    [Abstract] [Full Text] [Related]

  • 2. Interaction of folliculin (Birt-Hogg-Dubé gene product) with a novel Fnip1-like (FnipL/Fnip2) protein.
    Takagi Y, Kobayashi T, Shiono M, Wang L, Piao X, Sun G, Zhang D, Abe M, Hagiwara Y, Takahashi K, Hino O.
    Oncogene; 2008 Sep 11; 27(40):5339-47. PubMed ID: 18663353
    [Abstract] [Full Text] [Related]

  • 3. Folliculin-interacting proteins Fnip1 and Fnip2 play critical roles in kidney tumor suppression in cooperation with Flcn.
    Hasumi H, Baba M, Hasumi Y, Lang M, Huang Y, Oh HF, Matsuo M, Merino MJ, Yao M, Ito Y, Furuya M, Iribe Y, Kodama T, Southon E, Tessarollo L, Nagashima K, Haines DC, Linehan WM, Schmidt LS.
    Proc Natl Acad Sci U S A; 2015 Mar 31; 112(13):E1624-31. PubMed ID: 25775561
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  • 7. FLCN: The causative gene for Birt-Hogg-Dubé syndrome.
    Schmidt LS, Linehan WM.
    Gene; 2018 Jan 15; 640():28-42. PubMed ID: 28970150
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  • 8. Folliculin variants linked to Birt-Hogg-Dubé syndrome are targeted for proteasomal degradation.
    Clausen L, Stein A, Grønbæk-Thygesen M, Nygaard L, Søltoft CL, Nielsen SV, Lisby M, Ravid T, Lindorff-Larsen K, Hartmann-Petersen R.
    PLoS Genet; 2020 Nov 15; 16(11):e1009187. PubMed ID: 33137092
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  • 10. Loss of FLCN-FNIP1/2 induces a non-canonical interferon response in human renal tubular epithelial cells.
    Glykofridis IE, Knol JC, Balk JA, Westland D, Pham TV, Piersma SR, Lougheed SM, Derakhshan S, Veen P, Rooimans MA, van Mil SE, Böttger F, Poddighe PJ, van de Beek I, Drost J, Zwartkruis FJ, de Menezes RX, Meijers-Heijboer HE, Houweling AC, Jimenez CR, Wolthuis RM.
    Elife; 2021 Jan 18; 10():. PubMed ID: 33459596
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  • 11. Mutation of Fnip1 is associated with B-cell deficiency, cardiomyopathy, and elevated AMPK activity.
    Siggs OM, Stockenhuber A, Deobagkar-Lele M, Bull KR, Crockford TL, Kingston BL, Crawford G, Anzilotti C, Steeples V, Ghaffari S, Czibik G, Bellahcene M, Watkins H, Ashrafian H, Davies B, Woods A, Carling D, Yavari A, Beutler B, Cornall RJ.
    Proc Natl Acad Sci U S A; 2016 Jun 28; 113(26):E3706-15. PubMed ID: 27303042
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  • 12. Establishment and characterization of BHD-F59RSVT, an immortalized cell line derived from a renal cell carcinoma in a patient with Birt-Hogg-Dubé syndrome.
    Furuya M, Hasumi H, Baba M, Tanaka R, Iribe Y, Onishi T, Nagashima Y, Nakatani Y, Isono Y, Yao M.
    Lab Invest; 2017 Mar 28; 97(3):343-351. PubMed ID: 27991910
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  • 13. Birt-Hogg-Dubé syndrome: from gene discovery to molecularly targeted therapies.
    Schmidt LS.
    Fam Cancer; 2013 Sep 28; 12(3):357-64. PubMed ID: 23108783
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  • 14. Distinctive expression patterns of glycoprotein non-metastatic B and folliculin in renal tumors in patients with Birt-Hogg-Dubé syndrome.
    Furuya M, Hong SB, Tanaka R, Kuroda N, Nagashima Y, Nagahama K, Suyama T, Yao M, Nakatani Y.
    Cancer Sci; 2015 Mar 28; 106(3):315-23. PubMed ID: 25594584
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  • 15. FLCN, a novel autophagy component, interacts with GABARAP and is regulated by ULK1 phosphorylation.
    Dunlop EA, Seifan S, Claessens T, Behrends C, Kamps MA, Rozycka E, Kemp AJ, Nookala RK, Blenis J, Coull BJ, Murray JT, van Steensel MA, Wilkinson S, Tee AR.
    Autophagy; 2014 Oct 01; 10(10):1749-60. PubMed ID: 25126726
    [Abstract] [Full Text] [Related]

  • 16. Renal tumour suppressor function of the Birt-Hogg-Dubé syndrome gene product folliculin.
    Hudon V, Sabourin S, Dydensborg AB, Kottis V, Ghazi A, Paquet M, Crosby K, Pomerleau V, Uetani N, Pause A.
    J Med Genet; 2010 Mar 01; 47(3):182-9. PubMed ID: 19843504
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  • 17. Characterization of a splice-site mutation in the tumor suppressor gene FLCN associated with renal cancer.
    Bartram MP, Mishra T, Reintjes N, Fabretti F, Gharbi H, Adam AC, Göbel H, Franke M, Schermer B, Haneder S, Benzing T, Beck BB, Müller RU.
    BMC Med Genet; 2017 May 12; 18(1):53. PubMed ID: 28499369
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  • 18. Loss of Fnip1 alters kidney developmental transcriptional program and synergizes with TSC1 loss to promote mTORC1 activation and renal cyst formation.
    Centini R, Tsang M, Iwata T, Park H, Delrow J, Margineantu D, Iritani BM, Gu H, Liggitt HD, Kang J, Kang L, Hockenbery DM, Raftery D, Iritani BM.
    PLoS One; 2018 May 12; 13(6):e0197973. PubMed ID: 29897930
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  • 19. Birt-Hogg-Dubé renal tumors are genetically distinct from other renal neoplasias and are associated with up-regulation of mitochondrial gene expression.
    Klomp JA, Petillo D, Niemi NM, Dykema KJ, Chen J, Yang XJ, Sääf A, Zickert P, Aly M, Bergerheim U, Nordenskjöld M, Gad S, Giraud S, Denoux Y, Yonneau L, Méjean A, Vasiliu V, Richard S, MacKeigan JP, Teh BT, Furge KA.
    BMC Med Genomics; 2010 Dec 16; 3():59. PubMed ID: 21162720
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  • 20. Therapeutic targeting the loss of the birt-hogg-dube suppressor gene.
    Lu X, Wei W, Fenton J, Nahorski MS, Rabai E, Reiman A, Seabra L, Nagy Z, Latif F, Maher ER.
    Mol Cancer Ther; 2011 Jan 16; 10(1):80-9. PubMed ID: 21220493
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