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588 related items for PubMed ID: 18410539

  • 21. Interaction of CagA with Crk plays an important role in Helicobacter pylori-induced loss of gastric epithelial cell adhesion.
    Suzuki M, Mimuro H, Suzuki T, Park M, Yamamoto T, Sasakawa C.
    J Exp Med; 2005 Nov 07; 202(9):1235-47. PubMed ID: 16275761
    [Abstract] [Full Text] [Related]

  • 22. CagA protein secreted by the intact type IV secretion system leads to gastric epithelial inflammation in the Mongolian gerbil model.
    Shibata W, Hirata Y, Maeda S, Ogura K, Ohmae T, Yanai A, Mitsuno Y, Yamaji Y, Okamoto M, Yoshida H, Kawabe T, Omata M.
    J Pathol; 2006 Nov 07; 210(3):306-14. PubMed ID: 16933206
    [Abstract] [Full Text] [Related]

  • 23. Assembly and molecular mode of action of the Helicobacter pylori Cag type IV secretion apparatus.
    Fischer W.
    FEBS J; 2011 Apr 07; 278(8):1203-12. PubMed ID: 21352490
    [Abstract] [Full Text] [Related]

  • 24. Transgenic expression of Helicobacter pylori CagA induces gastrointestinal and hematopoietic neoplasms in mouse.
    Ohnishi N, Yuasa H, Tanaka S, Sawa H, Miura M, Matsui A, Higashi H, Musashi M, Iwabuchi K, Suzuki M, Yamada G, Azuma T, Hatakeyama M.
    Proc Natl Acad Sci U S A; 2008 Jan 22; 105(3):1003-8. PubMed ID: 18192401
    [Abstract] [Full Text] [Related]

  • 25. Helicobacter exploits integrin for type IV secretion and kinase activation.
    Kwok T, Zabler D, Urman S, Rohde M, Hartig R, Wessler S, Misselwitz R, Berger J, Sewald N, König W, Backert S.
    Nature; 2007 Oct 18; 449(7164):862-6. PubMed ID: 17943123
    [Abstract] [Full Text] [Related]

  • 26. CagA-independent disruption of adherence junction complexes involves E-cadherin shedding and implies multiple steps in Helicobacter pylori pathogenicity.
    Weydig C, Starzinski-Powitz A, Carra G, Löwer J, Wessler S.
    Exp Cell Res; 2007 Oct 01; 313(16):3459-71. PubMed ID: 17692843
    [Abstract] [Full Text] [Related]

  • 27. Pathogenicity island-dependent effects of Helicobacter pylori on intracellular signal transduction in epithelial cells.
    Naumann M.
    Int J Med Microbiol; 2005 Sep 01; 295(5):335-41. PubMed ID: 16173500
    [Abstract] [Full Text] [Related]

  • 28. Dual infection system identifies a crucial role for PKA-mediated serine phosphorylation of the EPEC-Tir-injected effector protein in regulating Rac1 function.
    Brandt S, Kenny B, Rohde M, Martinez-Quiles N, Backert S.
    Cell Microbiol; 2009 Aug 01; 11(8):1254-71. PubMed ID: 19438518
    [Abstract] [Full Text] [Related]

  • 29. Characterization of the pilin ortholog of the Helicobacter pylori type IV cag pathogenicity apparatus, a surface-associated protein expressed during infection.
    Andrzejewska J, Lee SK, Olbermann P, Lotzing N, Katzowitsch E, Linz B, Achtman M, Kado CI, Suerbaum S, Josenhans C.
    J Bacteriol; 2006 Aug 01; 188(16):5865-77. PubMed ID: 16885455
    [Abstract] [Full Text] [Related]

  • 30. Expression of CEACAM1 or CEACAM5 in AZ-521 cells restores the type IV secretion deficiency for translocation of CagA by Helicobacter pylori.
    Tegtmeyer N, Harrer A, Schmitt V, Singer BB, Backert S.
    Cell Microbiol; 2019 Jan 01; 21(1):e12965. PubMed ID: 30321907
    [Abstract] [Full Text] [Related]

  • 31. CagA-mediated pathogenesis of Helicobacter pylori.
    Tohidpour A.
    Microb Pathog; 2016 Apr 01; 93():44-55. PubMed ID: 26796299
    [Abstract] [Full Text] [Related]

  • 32. Pathogenesis of Helicobacter pylori infection.
    Maeda S, Mentis AF.
    Helicobacter; 2007 Oct 01; 12 Suppl 1():10-4. PubMed ID: 17727454
    [Abstract] [Full Text] [Related]

  • 33. The Helicobacter pylori Type IV Secretion System Encoded by the cag Pathogenicity Island: Architecture, Function, and Signaling.
    Backert S, Haas R, Gerhard M, Naumann M.
    Curr Top Microbiol Immunol; 2017 Oct 01; 413():187-220. PubMed ID: 29536360
    [Abstract] [Full Text] [Related]

  • 34. [Advances in CagA protein and CagAmediated pathogenesis of Helicobacter pylori-A review].
    Wan X, Liu C.
    Wei Sheng Wu Xue Bao; 2016 Dec 04; 56(12):1821-30. PubMed ID: 29741846
    [Abstract] [Full Text] [Related]

  • 35. Helicobacter pylori infection and CagA protein translocation in human primary gastric epithelial cell culture.
    Lai YP, Yang JC, Lin TZ, Lin JT, Wang JT.
    Helicobacter; 2006 Oct 04; 11(5):451-9. PubMed ID: 16961808
    [Abstract] [Full Text] [Related]

  • 36. Helicobacter pylori exploits a unique repertoire of type IV secretion system components for pilus assembly at the bacteria-host cell interface.
    Shaffer CL, Gaddy JA, Loh JT, Johnson EM, Hill S, Hennig EE, McClain MS, McDonald WH, Cover TL.
    PLoS Pathog; 2011 Sep 04; 7(9):e1002237. PubMed ID: 21909278
    [Abstract] [Full Text] [Related]

  • 37. Interleukin-1 receptor phosphorylation activates Rho kinase to disrupt human gastric tight junctional claudin-4 during Helicobacter pylori infection.
    Lapointe TK, O'Connor PM, Jones NL, Menard D, Buret AG.
    Cell Microbiol; 2010 May 01; 12(5):692-703. PubMed ID: 20070312
    [Abstract] [Full Text] [Related]

  • 38. Functional antagonism between Helicobacter pylori CagA and vacuolating toxin VacA in control of the NFAT signaling pathway in gastric epithelial cells.
    Yokoyama K, Higashi H, Ishikawa S, Fujii Y, Kondo S, Kato H, Azuma T, Wada A, Hirayama T, Aburatani H, Hatakeyama M.
    Proc Natl Acad Sci U S A; 2005 Jul 05; 102(27):9661-6. PubMed ID: 15980153
    [Abstract] [Full Text] [Related]

  • 39. Pathogenesis of Helicobacter pylori infection.
    Backert S, Clyne M.
    Helicobacter; 2011 Sep 05; 16 Suppl 1():19-25. PubMed ID: 21896081
    [Abstract] [Full Text] [Related]

  • 40. Pathogenicity of Helicobacter pylori infection.
    Radosz-Komoniewska H, Bek T, Jóźwiak J, Martirosian G.
    Clin Microbiol Infect; 2005 Aug 05; 11(8):602-10. PubMed ID: 16008611
    [Abstract] [Full Text] [Related]

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