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377 related items for PubMed ID: 18467595
1. M protein-mediated plasminogen binding is essential for the virulence of an invasive Streptococcus pyogenes isolate. Sanderson-Smith ML, Dinkla K, Cole JN, Cork AJ, Maamary PG, McArthur JD, Chhatwal GS, Walker MJ. FASEB J; 2008 Aug; 22(8):2715-22. PubMed ID: 18467595 [Abstract] [Full Text] [Related]
2. Analysis of plasminogen-binding M proteins of Streptococcus pyogenes. Ringdahl U, Sjöbring U. Methods; 2000 Jun; 21(2):143-50. PubMed ID: 10816375 [Abstract] [Full Text] [Related]
3. Trigger for group A streptococcal M1T1 invasive disease. Cole JN, McArthur JD, McKay FC, Sanderson-Smith ML, Cork AJ, Ranson M, Rohde M, Itzek A, Sun H, Ginsburg D, Kotb M, Nizet V, Chhatwal GS, Walker MJ. FASEB J; 2006 Aug; 20(10):1745-7. PubMed ID: 16790522 [Abstract] [Full Text] [Related]
4. Contribution of protein G-related alpha2-macroglobulin-binding protein to bacterial virulence in a mouse skin model of group A streptococcal infection. Toppel AW, Rasmussen M, Rohde M, Medina E, Chhatwal GS. J Infect Dis; 2003 Jun 01; 187(11):1694-703. PubMed ID: 12751026 [Abstract] [Full Text] [Related]
5. Direct Host Plasminogen Binding to Bacterial Surface M-protein in Pattern D Strains of Streptococcus pyogenes Is Required for Activation by Its Natural Coinherited SK2b Protein. Chandrahas V, Glinton K, Liang Z, Donahue DL, Ploplis VA, Castellino FJ. J Biol Chem; 2015 Jul 24; 290(30):18833-42. PubMed ID: 26070561 [Abstract] [Full Text] [Related]
6. Increased expression of the ska gene in emm49-genotyped Streptococcus pyogenes strains isolated from patients with severe invasive streptococcal infections. Ikebe T, Endoh M, Watanabe H. Jpn J Infect Dis; 2005 Oct 24; 58(5):272-5. PubMed ID: 16249619 [Abstract] [Full Text] [Related]
7. The plasminogen-binding group A streptococcal M protein-related protein Prp binds plasminogen via arginine and histidine residues. Sanderson-Smith ML, Dowton M, Ranson M, Walker MJ. J Bacteriol; 2007 Feb 24; 189(4):1435-40. PubMed ID: 17012384 [Abstract] [Full Text] [Related]
8. Anti-phagocytic mechanisms of Streptococcus pyogenes: binding of fibrinogen to M-related protein. Courtney HS, Hasty DL, Dale JB. Mol Microbiol; 2006 Feb 24; 59(3):936-47. PubMed ID: 16420362 [Abstract] [Full Text] [Related]
9. Is plasminogen deployed as a Streptococcus pyogenes virulence factor? Walker MJ, McArthur JD, McKay F, Ranson M. Trends Microbiol; 2005 Jul 24; 13(7):308-13. PubMed ID: 15936195 [Abstract] [Full Text] [Related]
10. Inactivation of single genes within the virulence regulon of an M2 group A streptococcal isolate result in differences in virulence for chicken embryos and for mice. Schmidt KH, Podbielski A, Raeder R, Boyle MD. Microb Pathog; 1997 Dec 24; 23(6):347-55. PubMed ID: 9441861 [Abstract] [Full Text] [Related]
11. Identification of active variants of PARF in human pathogenic group C and group G streptococci leads to an amended description of its consensus motif. Barroso V, Rohde M, Davies MR, Gillen CM, Nitsche-Schmitz DP, Dinkla K, Chhatwal GS. Int J Med Microbiol; 2009 Dec 24; 299(8):547-53. PubMed ID: 19520603 [Abstract] [Full Text] [Related]
12. Analysis of the role of CovR and CovS in the dissemination of Streptococcus pyogenes in invasive skin disease. Dalton TL, Hobb RI, Scott JR. Microb Pathog; 2006 May 24; 40(5):221-7. PubMed ID: 16542816 [Abstract] [Full Text] [Related]
13. Human fibrinogen bound to Streptococcus pyogenes M protein inhibits complement deposition via the classical pathway. Carlsson F, Sandin C, Lindahl G. Mol Microbiol; 2005 Apr 24; 56(1):28-39. PubMed ID: 15773976 [Abstract] [Full Text] [Related]
14. Strain prevalence, rather than innate virulence potential, is the major factor responsible for an increase in serious group A streptococcus infections. Rogers S, Commons R, Danchin MH, Selvaraj G, Kelpie L, Curtis N, Robins-Browne R, Carapetis JR. J Infect Dis; 2007 Jun 01; 195(11):1625-33. PubMed ID: 17471432 [Abstract] [Full Text] [Related]
15. Streptokinase variants from Streptococcus pyogenes isolates display altered plasminogen activation characteristics - implications for pathogenesis. Cook SM, Skora A, Gillen CM, Walker MJ, McArthur JD. Mol Microbiol; 2012 Dec 01; 86(5):1052-62. PubMed ID: 23106864 [Abstract] [Full Text] [Related]
16. Interaction between group A streptococci and the plasmin(ogen) system promotes virulence in a mouse skin infection model. Li Z, Ploplis VA, French EL, Boyle MD. J Infect Dis; 1999 Apr 01; 179(4):907-14. PubMed ID: 10068586 [Abstract] [Full Text] [Related]
17. Humanized Plasminogen Mouse Model to Study Group A Streptococcus Invasive Disease. Rivera-Hernandez T, Walker MJ. Methods Mol Biol; 2020 Apr 01; 2136():309-316. PubMed ID: 32430832 [Abstract] [Full Text] [Related]
18. A key role for the urokinase plasminogen activator (uPA) in invasive Group A streptococcal infection. Sanderson-Smith ML, Zhang Y, Ly D, Donahue D, Hollands A, Nizet V, Ranson M, Ploplis VA, Walker MJ, Castellino FJ. PLoS Pathog; 2013 Apr 01; 9(7):e1003469. PubMed ID: 23853591 [Abstract] [Full Text] [Related]
19. Use of DNA arrays to identify a mutation in the negative regulator, csrR, responsible for the high virulence of a naturally occurring type M3 group A streptococcus clinical isolate. Miyoshi-Akiyama T, Ikebe T, Watanabe H, Uchiyama T, Kirikae T, Kawamura Y. J Infect Dis; 2006 Jun 15; 193(12):1677-84. PubMed ID: 16703511 [Abstract] [Full Text] [Related]
20. Similar superantigen gene profiles and superantigen activity in norwegian isolates of invasive and non-invasive group a streptococci. Michaelsen TE, Andreasson IK, Langerud BK, Caugant DA. Scand J Immunol; 2011 Nov 15; 74(5):423-9. PubMed ID: 21707691 [Abstract] [Full Text] [Related] Page: [Next] [New Search]