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Journal Abstract Search

185 related items for PubMed ID: 18477768

  • 21.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 22. Increased lung resistance after diesel particulate and ozone co-exposure not associated with enhanced lung inflammation in allergic mice.
    Farraj AK, Boykin E, Ledbetter A, Andrews D, Gavett SH.
    Inhal Toxicol; 2010 Jan; 22(1):33-41. PubMed ID: 20017592
    [Abstract] [Full Text] [Related]

  • 23. Acute lung injury from intratracheal exposure to fugitive residual oil fly ash and its constituent metals in normo- and spontaneously hypertensive rats.
    Kodavanti UP, Schladweiler MC, Richards JR, Costa DL.
    Inhal Toxicol; 2001 Jan; 13(1):37-54. PubMed ID: 11153059
    [Abstract] [Full Text] [Related]

  • 24. Ultrafine but not fine particulate matter causes airway inflammation and allergic airway sensitization to co-administered antigen in mice.
    de Haar C, Hassing I, Bol M, Bleumink R, Pieters R.
    Clin Exp Allergy; 2006 Nov; 36(11):1469-79. PubMed ID: 17083358
    [Abstract] [Full Text] [Related]

  • 25. ST depression, arrhythmia, vagal dominance, and reduced cardiac micro-RNA in particulate-exposed rats.
    Farraj AK, Hazari MS, Haykal-Coates N, Lamb C, Winsett DW, Ge Y, Ledbetter AD, Carll AP, Bruno M, Ghio A, Costa DL.
    Am J Respir Cell Mol Biol; 2011 Feb; 44(2):185-96. PubMed ID: 20378750
    [Abstract] [Full Text] [Related]

  • 26. Pulmonary responses to oil fly ash particles in the rat differ by virtue of their specific soluble metals.
    Kodavanti UP, Hauser R, Christiani DC, Meng ZH, McGee J, Ledbetter A, Richards J, Costa DL.
    Toxicol Sci; 1998 Jun; 43(2):204-12. PubMed ID: 9710962
    [Abstract] [Full Text] [Related]

  • 27. Ultrafine carbon black particles cause early airway inflammation and have adjuvant activity in a mouse allergic airway disease model.
    de Haar C, Hassing I, Bol M, Bleumink R, Pieters R.
    Toxicol Sci; 2005 Oct; 87(2):409-18. PubMed ID: 16014737
    [Abstract] [Full Text] [Related]

  • 28. Cardiac arrhythmia induction after exposure to residual oil fly ash particles in a rodent model of pulmonary hypertension.
    Watkinson WP, Campen MJ, Costa DL.
    Toxicol Sci; 1998 Feb; 41(2):209-16. PubMed ID: 9520357
    [Abstract] [Full Text] [Related]

  • 29. Adjuvant activity of various diesel exhaust and ambient particles in two allergic models.
    Steerenberg PA, Withagen CE, Dormans JA, van Dalen WJ, van Loveren H, Casee FR.
    J Toxicol Environ Health A; 2003 Aug 08; 66(15):1421-39. PubMed ID: 12857633
    [Abstract] [Full Text] [Related]

  • 30. Role of Toll-like receptor-4 in genetic susceptibility to lung injury induced by residual oil fly ash.
    Cho HY, Jedlicka AE, Clarke R, Kleeberger SR.
    Physiol Genomics; 2005 Jun 16; 22(1):108-17. PubMed ID: 15784698
    [Abstract] [Full Text] [Related]

  • 31. Time course of systemic oxidative stress and inflammatory response induced by an acute exposure to Residual Oil Fly Ash.
    Marchini T, Magnani ND, Paz ML, Vanasco V, Tasat D, González Maglio DH, Alvarez S, Evelson PA.
    Toxicol Appl Pharmacol; 2014 Jan 15; 274(2):274-82. PubMed ID: 24321338
    [Abstract] [Full Text] [Related]

  • 32. Application of laser capture microdissection and protein microarray technologies in the molecular analysis of airway injury following pollution particle exposure.
    Roberts E, Charboneau L, Espina V, Liotta L, Petricoin E, Dreher K.
    J Toxicol Environ Health A; 2004 Jun 11; 67(11):851-61. PubMed ID: 15205040
    [Abstract] [Full Text] [Related]

  • 33. Increased non-conducted P-wave arrhythmias after a single oil fly ash inhalation exposure in hypertensive rats.
    Farraj AK, Haykal-Coates N, Winsett DW, Hazari MS, Carll AP, Rowan WH, Ledbetter AD, Cascio WE, Costa DL.
    Environ Health Perspect; 2009 May 11; 117(5):709-15. PubMed ID: 19479011
    [Abstract] [Full Text] [Related]

  • 34. Prior SO2 exposure promotes airway inflammation and subepithelial fibrosis following repeated ovalbumin challenge.
    Cai C, Xu J, Zhang M, Chen XD, Li L, Wu J, Lai HW, Zhong NS.
    Clin Exp Allergy; 2008 Oct 11; 38(10):1680-7. PubMed ID: 18631350
    [Abstract] [Full Text] [Related]

  • 35. Lung injury from intratracheal and inhalation exposures to residual oil fly ash in a rat model of monocrotaline-induced pulmonary hypertension.
    Kodavanti UP, Jackson MC, Ledbetter AD, Richards JR, Gardner SY, Watkinson WP, Campen MJ, Costa DL.
    J Toxicol Environ Health A; 1999 Aug 27; 57(8):543-63. PubMed ID: 10515573
    [Abstract] [Full Text] [Related]

  • 36.
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  • 37.
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  • 38. Metal composition and solubility determine lung toxicity induced by residual oil fly ash collected from different sites within a power plant.
    Antonini JM, Taylor MD, Leonard SS, Lawryk NJ, Shi X, Clarke RW, Roberts JR.
    Mol Cell Biochem; 2004 Jan 27; 255(1-2):257-65. PubMed ID: 14971666
    [Abstract] [Full Text] [Related]

  • 39. Is cardiac tissue more susceptible than lung to oxidative effects induced by chronic nasotropic instillation of residual oil fly ash (ROFA)?
    Damiani RM, Piva MO, Petry MR, Saldiva PH, Tavares Duarte de Oliveira A, Rhoden CR.
    Toxicol Mech Methods; 2012 Sep 27; 22(7):533-9. PubMed ID: 22563929
    [Abstract] [Full Text] [Related]

  • 40. Soluble transition metals mediate residual oil fly ash induced acute lung injury.
    Dreher KL, Jaskot RH, Lehmann JR, Richards JH, McGee JK, Ghio AJ, Costa DL.
    J Toxicol Environ Health; 1997 Feb 21; 50(3):285-305. PubMed ID: 9055877
    [Abstract] [Full Text] [Related]

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