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Journal Abstract Search

106 related items for PubMed ID: 1851549

  • 1.
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  • 2. Most of the substrates of oncogenic viral tyrosine protein kinases can be phosphorylated by cellular tyrosine protein kinases in normal cells.
    Kamps MP, Sefton BM.
    Oncogene Res; 1988 Sep; 3(2):105-15. PubMed ID: 2465525
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  • 3. Activation of the colony-stimulating factor 1 receptor leads to the rapid tyrosine phosphorylation of GTPase-activating protein and activation of cellular p21ras.
    Heidaran MA, Molloy CJ, Pangelinan M, Choudhury GG, Wang LM, Fleming TP, Sakaguchi AY, Pierce JH.
    Oncogene; 1992 Jan; 7(1):147-52. PubMed ID: 1311060
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  • 5. Role of c-Src in cellular events associated with colony-stimulating factor-1-induced spreading in osteoclasts.
    Insogna K, Tanaka S, Neff L, Horne W, Levy J, Baron R.
    Mol Reprod Dev; 1997 Jan; 46(1):104-8. PubMed ID: 8981371
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  • 7. Induced direct binding of the adapter protein Nck to the GTPase-activating protein-associated protein p62 by epidermal growth factor.
    Tang J, Feng GS, Li W.
    Oncogene; 1997 Oct 09; 15(15):1823-32. PubMed ID: 9362449
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  • 8. A novel 110 kDa form of myosin XVIIIA (MysPDZ) is tyrosine-phosphorylated after colony-stimulating factor-1 receptor signalling.
    Cross M, Csar XF, Wilson NJ, Manes G, Addona TA, Marks DC, Whitty GA, Ashman K, Hamilton JA.
    Biochem J; 2004 May 15; 380(Pt 1):243-53. PubMed ID: 14969583
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  • 9. Increased levels of p21ras-GTP and enhanced DNA synthesis accompany elevated tyrosyl phosphorylation of GAP-associated proteins, p190 and p62, in c-src overexpressors.
    Chang JH, Wilson LK, Moyers JS, Zhang K, Parsons SJ.
    Oncogene; 1993 Apr 15; 8(4):959-67. PubMed ID: 7681161
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  • 10. Phosphorylation of growth factor receptor binding protein-2 by pp60c-src tyrosine kinase.
    Jones DA, Benjamin CW.
    Arch Biochem Biophys; 1997 Jan 15; 337(2):143-8. PubMed ID: 9016807
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  • 11. Tyrosine phosphorylation of EGF-R and PDGF-R proteins during acute cutaneous wound healing process in mice.
    Gope ML, Gope R.
    Wound Repair Regen; 2009 Jan 15; 17(1):71-9. PubMed ID: 19152653
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  • 12. Mutational activation of pp60(c-src) leads to a tumorigenic phenotype in a preneoplastic Syrian hamster embryo cell line.
    Lansing TJ, Turk BF, Kanner SB, Gilmer TM.
    Cancer Res; 1997 May 15; 57(10):1962-9. PubMed ID: 9157992
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  • 13. STAT activation by the PDGF receptor requires juxtamembrane phosphorylation sites but not Src tyrosine kinase activation.
    Sachsenmaier C, Sadowski HB, Cooper JA.
    Oncogene; 1999 Jun 17; 18(24):3583-92. PubMed ID: 10380880
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  • 16. Transformation by pp60src or stimulation of cells with epidermal growth factor induces the stable association of tyrosine-phosphorylated cellular proteins with GTPase-activating protein.
    Bouton AH, Kanner SB, Vines RR, Wang HC, Gibbs JB, Parsons JT.
    Mol Cell Biol; 1991 Feb 17; 11(2):945-53. PubMed ID: 1703633
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  • 17. The catalytic subunit of phosphatidylinositol 3-kinase is a substrate for the activated platelet-derived growth factor receptor, but not for middle-T antigen-pp60c-src complexes.
    Roche S, Dhand R, Waterfield MD, Courtneidge SA.
    Biochem J; 1994 Aug 01; 301 ( Pt 3)(Pt 3):703-11. PubMed ID: 7519847
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  • 20. Polypeptide growth factors: roles in normal and abnormal cell growth.
    Deuel TF.
    Annu Rev Cell Biol; 1987 Aug 01; 3():443-92. PubMed ID: 3318882
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