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275 related items for PubMed ID: 18545997
1. Cisplatin sensitivity of oral squamous carcinoma cells is regulated by Na+,K+-ATPase activity rather than copper-transporting P-type ATPases, ATP7A and ATP7B. Ahmed Z, Deyama Y, Yoshimura Y, Suzuki K. Cancer Chemother Pharmacol; 2009 Mar; 63(4):643-50. PubMed ID: 18545997 [Abstract] [Full Text] [Related]
2. Copper efflux transporter (ATP7B) contributes to the acquisition of cisplatin-resistance in human oral squamous cell lines. Yoshizawa K, Nozaki S, Kitahara H, Ohara T, Kato K, Kawashiri S, Yamamoto E. Oncol Rep; 2007 Oct; 18(4):987-91. PubMed ID: 17786364 [Abstract] [Full Text] [Related]
3. Expression of copper-transporting P-type adenosine triphosphatase (ATP7B) as a chemoresistance marker in human oral squamous cell carcinoma treated with cisplatin. Miyashita H, Nitta Y, Mori S, Kanzaki A, Nakayama K, Terada K, Sugiyama T, Kawamura H, Sato A, Morikawa H, Motegi K, Takebayashi Y. Oral Oncol; 2003 Feb; 39(2):157-62. PubMed ID: 12509969 [Abstract] [Full Text] [Related]
4. Role of Na+, K+-ATPase alpha1 subunit in the intracellular accumulation of cisplatin. Kishimoto S, Kawazoe Y, Ikeno M, Saitoh M, Nakano Y, Nishi Y, Fukushima S, Takeuchi Y. Cancer Chemother Pharmacol; 2006 Jan; 57(1):84-90. PubMed ID: 16044341 [Abstract] [Full Text] [Related]
5. Altered localisation of the copper efflux transporters ATP7A and ATP7B associated with cisplatin resistance in human ovarian carcinoma cells. Kalayda GV, Wagner CH, Buss I, Reedijk J, Jaehde U. BMC Cancer; 2008 Jun 19; 8():175. PubMed ID: 18565219 [Abstract] [Full Text] [Related]
6. Sensitization of cancer cells towards Cisplatin and Carboplatin by protein kinase D inhibitors through modulation of ATP7A/B (copper transport ATPases). Janardhanan P, Somasundaran AK, Balakrishnan AJ, Pilankatta R. Cancer Treat Res Commun; 2022 Jun 19; 32():100613. PubMed ID: 35908410 [Abstract] [Full Text] [Related]
7. Increased expression of the copper efflux transporter ATP7A mediates resistance to cisplatin, carboplatin, and oxaliplatin in ovarian cancer cells. Samimi G, Safaei R, Katano K, Holzer AK, Rochdi M, Tomioka M, Goodman M, Howell SB. Clin Cancer Res; 2004 Jul 15; 10(14):4661-9. PubMed ID: 15269138 [Abstract] [Full Text] [Related]
8. Effect of copper and role of the copper transporters ATP7A and CTR1 in intracellular accumulation of cisplatin. Matsumoto S, Tanaka T, Kurokawa H, Matsuno K, Hayashida Y, Takahashi T. Anticancer Res; 2007 Jul 15; 27(4B):2209-16. PubMed ID: 17695505 [Abstract] [Full Text] [Related]
13. Functional interactions of Cu-ATPase ATP7B with cisplatin and the role of ATP7B in the resistance of cells to the drug. Leonhardt K, Gebhardt R, Mössner J, Lutsenko S, Huster D. J Biol Chem; 2009 Mar 20; 284(12):7793-802. PubMed ID: 19141620 [Abstract] [Full Text] [Related]
14. Copper-transporting P-type ATPase, ATP7A, confers multidrug resistance and its expression is related to resistance to SN-38 in clinical colon cancer. Owatari S, Akune S, Komatsu M, Ikeda R, Firth SD, Che XF, Yamamoto M, Tsujikawa K, Kitazono M, Ishizawa T, Takeuchi T, Aikou T, Mercer JF, Akiyama S, Furukawa T. Cancer Res; 2007 May 15; 67(10):4860-8. PubMed ID: 17510416 [Abstract] [Full Text] [Related]
15. Confocal microscopic analysis of the interaction between cisplatin and the copper transporter ATP7B in human ovarian carcinoma cells. Katano K, Safaei R, Samimi G, Holzer A, Tomioka M, Goodman M, Howell SB. Clin Cancer Res; 2004 Jul 01; 10(13):4578-88. PubMed ID: 15240550 [Abstract] [Full Text] [Related]
17. The CXXC motifs in the metal binding domains are required for ATP7B to mediate resistance to cisplatin. Safaei R, Adams PL, Maktabi MH, Mathews RA, Howell SB. J Inorg Biochem; 2012 May 01; 110():8-17. PubMed ID: 22459168 [Abstract] [Full Text] [Related]
18. Cellular pharmacology of cisplatin in relation to the expression of human copper transporter CTR1 in different pairs of cisplatin-sensitive and -resistant cells. Beretta GL, Gatti L, Tinelli S, Corna E, Colangelo D, Zunino F, Perego P. Biochem Pharmacol; 2004 Jul 15; 68(2):283-91. PubMed ID: 15194000 [Abstract] [Full Text] [Related]