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Journal Abstract Search

154 related items for PubMed ID: 18558095

  • 1. YY1 restrained cell senescence through repressing the transcription of p16.
    Wang X, Feng Y, Xu L, Chen Y, Zhang Y, Su D, Ren G, Lu J, Huang B.
    Biochim Biophys Acta; 2008 Oct; 1783(10):1876-83. PubMed ID: 18558095
    [Abstract] [Full Text] [Related]

  • 2. The transcription factor ZBP-89 suppresses p16 expression through a histone modification mechanism to affect cell senescence.
    Feng Y, Wang X, Xu L, Pan H, Zhu S, Liang Q, Huang B, Lu J.
    FEBS J; 2009 Aug; 276(15):4197-206. PubMed ID: 19583777
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  • 3. Recruitment of HDAC4 by transcription factor YY1 represses HOXB13 to affect cell growth in AR-negative prostate cancers.
    Ren G, Zhang G, Dong Z, Liu Z, Li L, Feng Y, Su D, Zhang Y, Huang B, Lu J.
    Int J Biochem Cell Biol; 2009 May; 41(5):1094-101. PubMed ID: 19013255
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  • 4. Histone deacetylase 3 (HDAC3) participates in the transcriptional repression of the p16 (INK4a) gene in mammary gland of the female rat offspring exposed to an early-life high-fat diet.
    Zheng S, Li Q, Zhang Y, Balluff Z, Pan YX.
    Epigenetics; 2012 Feb; 7(2):183-90. PubMed ID: 22395468
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  • 11. JDP2 (Jun Dimerization Protein 2)-deficient mouse embryonic fibroblasts are resistant to replicative senescence.
    Nakade K, Pan J, Yamasaki T, Murata T, Wasylyk B, Yokoyama KK.
    J Biol Chem; 2009 Apr 17; 284(16):10808-17. PubMed ID: 19233846
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  • 12. Cellular senescence and tumor suppressor gene p16.
    Rayess H, Wang MB, Srivatsan ES.
    Int J Cancer; 2012 Apr 15; 130(8):1715-25. PubMed ID: 22025288
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  • 13. Adenovirus transforming protein E1A induces c-Myc in quiescent cells by a novel mechanism.
    Kadeppagari RK, Sankar N, Thimmapaya B.
    J Virol; 2009 May 15; 83(10):4810-22. PubMed ID: 19279113
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  • 14. p16(INK4a) suppression by glucose restriction contributes to human cellular lifespan extension through SIRT1-mediated epigenetic and genetic mechanisms.
    Li Y, Tollefsbol TO.
    PLoS One; 2011 Feb 24; 6(2):e17421. PubMed ID: 21390332
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  • 15. Hepatitis B virus X protein overcomes stress-induced premature senescence by repressing p16(INK4a) expression via DNA methylation.
    Kim YJ, Jung JK, Lee SY, Jang KL.
    Cancer Lett; 2010 Feb 28; 288(2):226-35. PubMed ID: 19656618
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  • 16. Inhibition of p16INK4A to Rejuvenate Aging Human Cardiac Progenitor Cells via the Upregulation of Anti-oxidant and NFκB Signal Pathways.
    Khatiwala RV, Zhang S, Li X, Devejian N, Bennett E, Cai C.
    Stem Cell Rev Rep; 2018 Aug 28; 14(4):612-625. PubMed ID: 29675777
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  • 17. Sp1 is essential for p16 expression in human diploid fibroblasts during senescence.
    Wu J, Xue L, Weng M, Sun Y, Zhang Z, Wang W, Tong T.
    PLoS One; 2007 Jan 17; 2(1):e164. PubMed ID: 17225865
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  • 18. Twist-1 induces Ezh2 recruitment regulating histone methylation along the Ink4A/Arf locus in mesenchymal stem cells.
    Cakouros D, Isenmann S, Cooper L, Zannettino A, Anderson P, Glackin C, Gronthos S.
    Mol Cell Biol; 2012 Apr 17; 32(8):1433-41. PubMed ID: 22290439
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  • 19. Id1 regulation of cellular senescence through transcriptional repression of p16/Ink4a.
    Alani RM, Young AZ, Shifflett CB.
    Proc Natl Acad Sci U S A; 2001 Jul 03; 98(14):7812-6. PubMed ID: 11427735
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  • 20. P16INK4a is required for hSNF5 chromatin remodeler-induced cellular senescence in malignant rhabdoid tumor cells.
    Oruetxebarria I, Venturini F, Kekarainen T, Houweling A, Zuijderduijn LM, Mohd-Sarip A, Vries RG, Hoeben RC, Verrijzer CP.
    J Biol Chem; 2004 Jan 30; 279(5):3807-16. PubMed ID: 14604992
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