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Journal Abstract Search

230 related items for PubMed ID: 18599530

  • 1. Ser1369Ala variant in sulfonylurea receptor gene ABCC8 is associated with antidiabetic efficacy of gliclazide in Chinese type 2 diabetic patients.
    Feng Y, Mao G, Ren X, Xing H, Tang G, Li Q, Li X, Sun L, Yang J, Ma W, Wang X, Xu X.
    Diabetes Care; 2008 Oct; 31(10):1939-44. PubMed ID: 18599530
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  • 2. ABCC8 polymorphism (Ser1369Ala): influence on severe hypoglycemia due to sulfonylureas.
    Sato R, Watanabe H, Genma R, Takeuchi M, Maekawa M, Nakamura H.
    Pharmacogenomics; 2010 Dec; 11(12):1743-50. PubMed ID: 21142918
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  • 3. Association of sulfonylurea receptor 1 genotype with therapeutic response to gliclazide in type 2 diabetes.
    Zhang H, Liu X, Kuang H, Yi R, Xing H.
    Diabetes Res Clin Pract; 2007 Jul; 77(1):58-61. PubMed ID: 17118480
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  • 4. Association of KCNJ11 and ABCC8 genetic polymorphisms with response to repaglinide in Chinese diabetic patients.
    He YY, Zhang R, Shao XY, Hu C, Wang CR, Lu JX, Bao YQ, Jia WP, Xiang KS.
    Acta Pharmacol Sin; 2008 Aug; 29(8):983-9. PubMed ID: 18664331
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  • 5. Coexpression of the type 2 diabetes susceptibility gene variants KCNJ11 E23K and ABCC8 S1369A alter the ATP and sulfonylurea sensitivities of the ATP-sensitive K(+) channel.
    Hamming KS, Soliman D, Matemisz LC, Niazi O, Lang Y, Gloyn AL, Light PE.
    Diabetes; 2009 Oct; 58(10):2419-24. PubMed ID: 19587354
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  • 6. The Ser1369Ala variant of ABCC8 and the risk for severe sulfonylurea-induced hypoglycemia in German patients with Type 2 diabetes.
    Holstein JD, Kovacs P, Patzer O, Stumvoll M, Holstein A.
    Pharmacogenomics; 2012 Jan; 13(1):5-7; author reply 9-10. PubMed ID: 22176616
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  • 7. Role of common sequence variants in insulin secretion in familial type 2 diabetic kindreds: the sulfonylurea receptor, glucokinase, and hepatocyte nuclear factor 1alpha genes.
    Elbein SC, Sun J, Scroggin E, Teng K, Hasstedt SJ.
    Diabetes Care; 2001 Mar; 24(3):472-8. PubMed ID: 11289470
    [Abstract] [Full Text] [Related]

  • 8. Genetic cause of hyperglycaemia and response to treatment in diabetes.
    Pearson ER, Starkey BJ, Powell RJ, Gribble FM, Clark PM, Hattersley AT.
    Lancet; 2003 Oct 18; 362(9392):1275-81. PubMed ID: 14575972
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  • 11. Effect of genetic variants in KCNJ11, ABCC8, PPARG and HNF4A loci on the susceptibility of type 2 diabetes in Chinese Han population.
    Wang F, Han XY, Ren Q, Zhang XY, Han LC, Luo YY, Zhou XH, Ji LN.
    Chin Med J (Engl); 2009 Oct 20; 122(20):2477-82. PubMed ID: 20079163
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  • 12. Type 2 diabetes-associated missense polymorphisms KCNJ11 E23K and ABCC8 A1369S influence progression to diabetes and response to interventions in the Diabetes Prevention Program.
    Florez JC, Jablonski KA, Kahn SE, Franks PW, Dabelea D, Hamman RF, Knowler WC, Nathan DM, Altshuler D.
    Diabetes; 2007 Feb 20; 56(2):531-6. PubMed ID: 17259403
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  • 14. Heterozygous ABCC8 mutations are a cause of MODY.
    Bowman P, Flanagan SE, Edghill EL, Damhuis A, Shepherd MH, Paisey R, Hattersley AT, Ellard S.
    Diabetologia; 2012 Jan 20; 55(1):123-7. PubMed ID: 21989597
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  • 18. Metabolic control in type 2 diabetes is associated with sulfonylurea receptor-1 (SUR-1) but not with KCNJ11 polymorphisms.
    Nikolac N, Simundic AM, Katalinic D, Topic E, Cipak A, Zjacic Rotkvic V.
    Arch Med Res; 2009 Jul 20; 40(5):387-92. PubMed ID: 19766903
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  • 20. Large-scale association studies of variants in genes encoding the pancreatic beta-cell KATP channel subunits Kir6.2 (KCNJ11) and SUR1 (ABCC8) confirm that the KCNJ11 E23K variant is associated with type 2 diabetes.
    Gloyn AL, Weedon MN, Owen KR, Turner MJ, Knight BA, Hitman G, Walker M, Levy JC, Sampson M, Halford S, McCarthy MI, Hattersley AT, Frayling TM.
    Diabetes; 2003 Feb 20; 52(2):568-72. PubMed ID: 12540637
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