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Journal Abstract Search

219 related items for PubMed ID: 18619523

  • 1. Tempol diminishes cocaine-induced oxidative damage and attenuates the development and expression of behavioral sensitization.
    Numa R, Kohen R, Poltyrev T, Yaka R.
    Neuroscience; 2008 Aug 26; 155(3):649-58. PubMed ID: 18619523
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  • 2. Cocaine-induced status epilepticus and death generate oxidative stress in prefrontal cortex and striatum of mice.
    Macêdo DS, Vasconcelos SM, Andrade-Neto M, Belchior LD, Honório Júnior JE, Gonçalves DO, Fonteles MM, Silva MI, Aguiar LM, Viana GS, de Sousa FC.
    Neurochem Int; 2010 Jan 26; 56(1):183-7. PubMed ID: 19822180
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  • 6. Chronic treatment with Tempol during acquisition or withdrawal from CPP abolishes the expression of cocaine reward and diminishes oxidative damage.
    Beiser T, Numa R, Kohen R, Yaka R.
    Sci Rep; 2017 Sep 11; 7(1):11162. PubMed ID: 28894248
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  • 7. Cocaine treatment increases expression of a 40 kDa catecholamine-regulated protein in discrete brain regions.
    Sharan N, Chong VZ, Nair VD, Mishra RK, Hayes RJ, Gardner EL.
    Synapse; 2003 Jan 11; 47(1):33-44. PubMed ID: 12422371
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  • 12. Involvement of adenosine A2A and dopamine receptors in the locomotor and sensitizing effects of cocaine.
    Filip M, Frankowska M, Zaniewska M, Przegaliński E, Muller CE, Agnati L, Franco R, Roberts DC, Fuxe K.
    Brain Res; 2006 Mar 10; 1077(1):67-80. PubMed ID: 16516871
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  • 13. Renal and metabolic effects of tempol in obese ZSF1 rats--distinct role for superoxide and hydrogen peroxide in diabetic renal injury.
    Rafikova O, Salah EM, Tofovic SP.
    Metabolism; 2008 Oct 10; 57(10):1434-44. PubMed ID: 18803950
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