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Journal Abstract Search


422 related items for PubMed ID: 18680765

  • 21. Contribution of the C-terminal regions of promyelocytic leukemia protein (PML) isoforms II and V to PML nuclear body formation.
    Geng Y, Monajembashi S, Shao A, Cui D, He W, Chen Z, Hemmerich P, Tang J.
    J Biol Chem; 2012 Aug 31; 287(36):30729-42. PubMed ID: 22773875
    [Abstract] [Full Text] [Related]

  • 22. [Promyelocytic leukaemia protein and defect in transforming growth factor-beta signal pathway in acute promyelocytic leukaemia].
    Fuchs O, Provazníková D, Peslová G.
    Cas Lek Cesk; 2005 Aug 31; 144(2):90-4. PubMed ID: 15807293
    [Abstract] [Full Text] [Related]

  • 23.
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  • 24. Regulation of p53 activity in nuclear bodies by a specific PML isoform.
    Fogal V, Gostissa M, Sandy P, Zacchi P, Sternsdorf T, Jensen K, Pandolfi PP, Will H, Schneider C, Del Sal G.
    EMBO J; 2000 Nov 15; 19(22):6185-95. PubMed ID: 11080164
    [Abstract] [Full Text] [Related]

  • 25. Functional reorganization of promyelocytic leukemia nuclear bodies during BK virus infection.
    Jiang M, Entezami P, Gamez M, Stamminger T, Imperiale MJ.
    mBio; 2011 Nov 15; 2(1):e00281-10. PubMed ID: 21304169
    [Abstract] [Full Text] [Related]

  • 26. Regulation of apoptosis by PML and the PML-NBs.
    Bernardi R, Papa A, Pandolfi PP.
    Oncogene; 2008 Oct 20; 27(48):6299-312. PubMed ID: 18931695
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  • 28. Tyrosine kinase inhibitor, methyl 2,5-dihydromethylcinnimate, induces PML nuclear body formation and apoptosis in tumor cells.
    Komura N, Asakawa M, Umezawa K, Segawa K.
    Exp Cell Res; 2007 Aug 01; 313(13):2753-65. PubMed ID: 17585903
    [Abstract] [Full Text] [Related]

  • 29. Pml and TAp73 interacting at nuclear body mediate imatinib-induced p53-independent apoptosis of chronic myeloid leukemia cells.
    Liu JH, Liu CC, Yen CC, Gau JP, Wang WS, Tzeng CH.
    Int J Cancer; 2009 Jul 01; 125(1):71-7. PubMed ID: 19291793
    [Abstract] [Full Text] [Related]

  • 30. The herpesvirus associated ubiquitin specific protease, USP7, is a negative regulator of PML proteins and PML nuclear bodies.
    Sarkari F, Wang X, Nguyen T, Frappier L.
    PLoS One; 2011 Jan 31; 6(1):e16598. PubMed ID: 21305000
    [Abstract] [Full Text] [Related]

  • 31. A nucleolar targeting signal in PML-I addresses PML to nucleolar caps in stressed or senescent cells.
    Condemine W, Takahashi Y, Le Bras M, de Thé H.
    J Cell Sci; 2007 Sep 15; 120(Pt 18):3219-27. PubMed ID: 17878236
    [Abstract] [Full Text] [Related]

  • 32. Modulation of CREB binding protein function by the promyelocytic (PML) oncoprotein suggests a role for nuclear bodies in hormone signaling.
    Doucas V, Tini M, Egan DA, Evans RM.
    Proc Natl Acad Sci U S A; 1999 Mar 16; 96(6):2627-32. PubMed ID: 10077561
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  • 34. Patient mutations alter ATRX targeting to PML nuclear bodies.
    Bérubé NG, Healy J, Medina CF, Wu S, Hodgson T, Jagla M, Picketts DJ.
    Eur J Hum Genet; 2008 Feb 16; 16(2):192-201. PubMed ID: 17957225
    [Abstract] [Full Text] [Related]

  • 35. Regulation of p53 activity by its interaction with homeodomain-interacting protein kinase-2.
    Hofmann TG, Möller A, Sirma H, Zentgraf H, Taya Y, Dröge W, Will H, Schmitz ML.
    Nat Cell Biol; 2002 Jan 16; 4(1):1-10. PubMed ID: 11740489
    [Abstract] [Full Text] [Related]

  • 36. PML bodies: a meeting place for genomic loci?
    Ching RW, Dellaire G, Eskiw CH, Bazett-Jones DP.
    J Cell Sci; 2005 Mar 01; 118(Pt 5):847-54. PubMed ID: 15731002
    [Abstract] [Full Text] [Related]

  • 37. PML IV/ARF interaction enhances p53 SUMO-1 conjugation, activation, and senescence.
    Ivanschitz L, Takahashi Y, Jollivet F, Ayrault O, Le Bras M, de Thé H.
    Proc Natl Acad Sci U S A; 2015 Nov 17; 112(46):14278-83. PubMed ID: 26578773
    [Abstract] [Full Text] [Related]

  • 38. A cytoplasmic PML mutant inhibits p53 function.
    Bellodi C, Kindle K, Bernassola F, Cossarizza A, Dinsdale D, Melino G, Heery D, Salomoni P.
    Cell Cycle; 2006 Nov 17; 5(22):2688-92. PubMed ID: 17172828
    [Abstract] [Full Text] [Related]

  • 39. p53 and p21(Waf1) are recruited to distinct PML-containing nuclear foci in irradiated and Nutlin-3a-treated U2OS cells.
    Shen H, Maki CG.
    J Cell Biochem; 2010 Dec 01; 111(5):1280-90. PubMed ID: 20803550
    [Abstract] [Full Text] [Related]

  • 40. Epstein - Barr virus Latent Membrane Protein 1 suppresses reporter activity through modulation of promyelocytic leukemia protein-nuclear bodies.
    Sides MD, Block GJ, Chadwick RW, Shan B, Flemington EK, Lasky JA.
    Virol J; 2011 Oct 05; 8():461. PubMed ID: 21975125
    [Abstract] [Full Text] [Related]


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