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Journal Abstract Search
180 related items for PubMed ID: 1872835
1. NG-monomethyl-l-arginine (NMA) restores arterial blood pressure but reduces cardiac output in a canine model of endotoxic shock. Klabunde RE, Ritger RC. Biochem Biophys Res Commun; 1991 Aug 15; 178(3):1135-40. PubMed ID: 1872835 [Abstract] [Full Text] [Related]
2. Cardiovascular actions of NG-methyl-L-arginine are abolished in a canine shock model using high-dose endotoxin. Klabunde RE, Helgren MC. Res Commun Chem Pathol Pharmacol; 1992 Oct 15; 78(1):57-68. PubMed ID: 1462049 [Abstract] [Full Text] [Related]
3. Reversal of endotoxin-mediated shock by NG-methyl-L-arginine, an inhibitor of nitric oxide synthesis. Kilbourn RG, Jubran A, Gross SS, Griffith OW, Levi R, Adams J, Lodato RF. Biochem Biophys Res Commun; 1990 Nov 15; 172(3):1132-8. PubMed ID: 2244897 [Abstract] [Full Text] [Related]
4. Cardiovascular actions of inhibitors of endothelium-derived relaxing factor (nitric oxide) formation/release in anesthetized dogs. Klabunde RE, Ritger RC, Helgren MC. Eur J Pharmacol; 1991 Jun 18; 199(1):51-9. PubMed ID: 1893927 [Abstract] [Full Text] [Related]
5. Effects of N-methyl-L-arginine on cardiac and regional blood flow in a dog endotoxin shock model. Doursout MF, Kilbourn RG, Hartley CJ, Chelly JE. J Crit Care; 2000 Mar 18; 15(1):22-9. PubMed ID: 10757195 [Abstract] [Full Text] [Related]
6. The role of nitric oxide in endotoxin-elicited hypodynamic circulatory failure. Brackett DJ, Lerner MR, Wilson MF. Adv Exp Med Biol; 1994 Mar 18; 366():412-3. PubMed ID: 7771273 [No Abstract] [Full Text] [Related]
7. NG-methyl-L-arginine restores arterial pressure in hemorrhaged rats. Klabunde RE, Slayton KJ, Ritger RC. Circ Shock; 1993 May 18; 40(1):47-52. PubMed ID: 8324889 [Abstract] [Full Text] [Related]
8. Effects of a nitric oxide synthase inhibitor in humans with septic shock. Petros A, Lamb G, Leone A, Moncada S, Bennett D, Vallance P. Cardiovasc Res; 1994 Jan 18; 28(1):34-9. PubMed ID: 7509259 [Abstract] [Full Text] [Related]
10. Effects of nitric oxide on blood flow distribution and O2 extraction capabilities during endotoxic shock. Zhang H, Rogiers P, Smail N, Cabral A, Preiser JC, Peny MO, Vincent JL. J Appl Physiol (1985); 1997 Oct 18; 83(4):1164-73. PubMed ID: 9338425 [Abstract] [Full Text] [Related]
11. Nitric oxide inhibition in the treatment of the sepsis syndrome is detrimental to tissue oxygenation. Statman R, Cheng W, Cunningham JN, Henderson JL, Damiani P, Siconolfi A, Rogers D, Horovitz JH. J Surg Res; 1994 Jul 18; 57(1):93-8. PubMed ID: 8041156 [Abstract] [Full Text] [Related]