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Journal Abstract Search

516 related items for PubMed ID: 18767145

  • 1. Cryptic and partial deletions of PRDM16 and RUNX1 without t(1;21)(p36;q22) and/or RUNX1-PRDM16 fusion in a case of progressive chronic myeloid leukemia: a complex chromosomal rearrangement of underestimated frequency in disease progression?
    Deluche L, Joha S, Corm S, Daudignon A, Geffroy S, Quief S, Villenet C, Kerckaert JP, Laï JL, Preudhomme C, Roche-Lestienne C.
    Genes Chromosomes Cancer; 2008 Dec; 47(12):1110-7. PubMed ID: 18767145
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  • 2. Novel RUNX1-PRDM16 fusion transcripts in a patient with acute myeloid leukemia showing t(1;21)(p36;q22).
    Sakai I, Tamura T, Narumi H, Uchida N, Yakushijin Y, Hato T, Fujita S, Yasukawa M.
    Genes Chromosomes Cancer; 2005 Nov; 44(3):265-70. PubMed ID: 16015645
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  • 3. Amplification of BCR-ABL and t(3;21) in a patient with blast crisis of chronic myelogenous leukemia.
    Phan CL, Megat Baharuddin PJ, Chin LP, Zakaria Z, Yegappan S, Sathar J, Tan SM, Purushothaman V, Chang KM.
    Cancer Genet Cytogenet; 2008 Jan 01; 180(1):60-4. PubMed ID: 18068536
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  • 7. Development of AML with t(8;21)(q22;q22) and RUNX1-RUNX1T1 fusion following Philadelphia-negative clonal evolution during treatment of CML with Imatinib.
    Schafhausen P, Dierlamm J, Bokemeyer C, Bruemmendorf TH, Bacher U, Zander AR, Schnittger S, Hochhaus A.
    Cancer Genet Cytogenet; 2009 Feb 01; 189(1):63-7. PubMed ID: 19167615
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  • 9. Acute myeloid leukemia with del(X)(p21) and cryptic RUNX1/RUNX1T1 from ins(8;21)(q22;q22q22) revealed by atypical FISH signals.
    Jang JH, Yoo EH, Kim HJ, Kim DH, Jung CW, Kim SH.
    Ann Clin Lab Sci; 2010 Feb 01; 40(1):80-4. PubMed ID: 20124335
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  • 10. Identification of truncated RUNX1 and RUNX1-PRDM16 fusion transcripts in a case of t(1;21)(p36;q22)-positive therapy-related AML.
    Stevens-Kroef MJ, Schoenmakers EF, van Kraaij M, Huys E, Vermeulen S, van der Reijden B, van Kessel AG.
    Leukemia; 2006 Jun 01; 20(6):1187-9. PubMed ID: 16598304
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  • 11. Multicolor COBRA-FISH analysis of chronic myeloid leukemia reveals novel cryptic balanced translocations during disease progression.
    Barbouti A, Johansson B, Höglund M, Mauritzson N, Strömbeck B, Nilsson PG, Tanke HJ, Hagemeijer A, Mitelman F, Fioretos T.
    Genes Chromosomes Cancer; 2002 Oct 01; 35(2):127-37. PubMed ID: 12203776
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  • 12. Molecular cytogenetic and clinical findings in ETV6/ABL1-positive leukemia.
    Van Limbergen H, Beverloo HB, van Drunen E, Janssens A, Hählen K, Poppe B, Van Roy N, Marynen P, De Paepe A, Slater R, Speleman F.
    Genes Chromosomes Cancer; 2001 Mar 01; 30(3):274-82. PubMed ID: 11170285
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  • 13. AML1/RUNX1 fusion gene and t(5;21)(q13;q22) in a case of chronic myelomonocytic leukemia with progressive thrombocytopenia and monocytosis.
    Liu S, Li C, Bo L, Dai Y, Xiao Z, Wang J.
    Cancer Genet Cytogenet; 2004 Jul 15; 152(2):172-4. PubMed ID: 15262442
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  • 15. RUNX1-MTG16 fusion gene in acute myeloblastic leukemia with t(16;21)(q24;q22): case report and review of the literature.
    De Braekeleer E, Douet-Guilbert N, Le Bris MJ, Morel F, Férec C, De Braekeleer M.
    Cancer Genet Cytogenet; 2008 Aug 15; 185(1):47-50. PubMed ID: 18656694
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  • 16. Involvement of der(12)t(12;21)(p13;q22) and as well as additional rearrangements of chromosome 12 homolog in ETV6/RUNX1-positive acute lymphoblastic leukemia.
    Stanchescu R, Betts DR, Rechavi G, Amariglio N, Trakhtenbrot L.
    Cancer Genet Cytogenet; 2009 Apr 01; 190(1):26-32. PubMed ID: 19264230
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  • 17. Unique secondary chromosomal abnormalities are frequently found in the chronic phase of chronic myeloid leukemia in southern Vietnam.
    Phan TX, Hoang AV, Huynh VM, Nguyen KT, Nguyen TB, Huynh N, Pham QT, Tran VB, Tran VB, Tokunaga K, Sato Y.
    Cancer Genet Cytogenet; 2006 Jul 01; 168(1):59-68. PubMed ID: 16772122
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  • 19. PRDX4, a member of the peroxiredoxin family, is fused to AML1 (RUNX1) in an acute myeloid leukemia patient with a t(X;21)(p22;q22).
    Zhang Y, Emmanuel N, Kamboj G, Chen J, Shurafa M, Van Dyke DL, Wiktor A, Rowley JD.
    Genes Chromosomes Cancer; 2004 Aug 01; 40(4):365-70. PubMed ID: 15188461
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  • 20. RUNX1 DNA-binding mutations and RUNX1-PRDM16 cryptic fusions in BCR-ABL+ leukemias are frequently associated with secondary trisomy 21 and may contribute to clonal evolution and imatinib resistance.
    Roche-Lestienne C, Deluche L, Corm S, Tigaud I, Joha S, Philippe N, Geffroy S, Laï JL, Nicolini FE, Preudhomme C, Fi-LMC group.
    Blood; 2008 Apr 01; 111(7):3735-41. PubMed ID: 18202228
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