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5. Adenosine A(2A) receptor antagonists are broad facilitators of antinicotinic neuromuscular blockade monitored either with 2 Hz train-of-four or 50 Hz tetanic stimuli. Pereira MW, Correia-de-Sá P, Alves-Do-Prado W. Clin Exp Pharmacol Physiol; 2012 Oct; 39(10):869-77. PubMed ID: 23013133 [Abstract] [Full Text] [Related]
6. Train-of-four and tetanic fade are not always a prejunctional phenomenon as evaluated by toxins having highly specific pre- and postjunctional actions. Nagashima M, Yasuhara S, Martyn JAJ. Anesth Analg; 2013 May; 116(5):994-1000. PubMed ID: 23477960 [Abstract] [Full Text] [Related]
7. Reversals of the neostigmine-induced tetanic fade and endplate potential run-down with respect to the autoregulation of transmitter release. Chang CC, Chen SM, Hong SJ. Br J Pharmacol; 1988 Dec; 95(4):1255-61. PubMed ID: 2905913 [Abstract] [Full Text] [Related]
8. The role of pre-and post-junctional cholinoceptors in the action of neostigmine at the neuromuscular junction. Webb SN, Bowman WC. Clin Exp Pharmacol Physiol; 1974 Dec; 1(2):123-34. PubMed ID: 4458990 [No Abstract] [Full Text] [Related]
9. Train-of-four fade during onset of neuromuscular block with nondepolarising neuromuscular blocking agents. Gibson FM, Mirakhur RK. Acta Anaesthesiol Scand; 1989 Apr; 33(3):204-6. PubMed ID: 2567104 [Abstract] [Full Text] [Related]
19. Nicotinic antagonists produce differing amounts of tetanic fade in the isolated diaphragm of the rat. Gibb AJ, Marshall IG. Br J Pharmacol; 1986 Nov; 89(3):619-24. PubMed ID: 2879597 [Abstract] [Full Text] [Related]