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Journal Abstract Search

514 related items for PubMed ID: 18974993

  • 1. Transient intraneuronal A beta rather than extracellular plaque pathology correlates with neuron loss in the frontal cortex of APP/PS1KI mice.
    Christensen DZ, Kraus SL, Flohr A, Cotel MC, Wirths O, Bayer TA.
    Acta Neuropathol; 2008 Dec; 116(6):647-55. PubMed ID: 18974993
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  • 2. Review on the APP/PS1KI mouse model: intraneuronal Abeta accumulation triggers axonopathy, neuron loss and working memory impairment.
    Bayer TA, Wirths O.
    Genes Brain Behav; 2008 Feb; 7 Suppl 1():6-11. PubMed ID: 18184366
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  • 3. Age-dependent loss of dentate gyrus granule cells in APP/PS1KI mice.
    Cotel MC, Bayer TA, Wirths O.
    Brain Res; 2008 Jul 30; 1222():207-13. PubMed ID: 18585693
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  • 4. Accelerated amyloid deposition, neurofibrillary degeneration and neuronal loss in double mutant APP/tau transgenic mice.
    Ribé EM, Pérez M, Puig B, Gich I, Lim F, Cuadrado M, Sesma T, Catena S, Sánchez B, Nieto M, Gómez-Ramos P, Morán MA, Cabodevilla F, Samaranch L, Ortiz L, Pérez A, Ferrer I, Avila J, Gómez-Isla T.
    Neurobiol Dis; 2005 Dec 30; 20(3):814-22. PubMed ID: 16125396
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  • 5. Intracellular Aß triggers neuron loss in the cholinergic system of the APP/PS1KI mouse model of Alzheimer's disease.
    Christensen DZ, Bayer TA, Wirths O.
    Neurobiol Aging; 2010 Jul 30; 31(7):1153-63. PubMed ID: 18771817
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  • 7. Intraneuronal beta-amyloid is a major risk factor--novel evidence from the APP/PS1KI mouse model.
    Bayer TA, Breyhan H, Duan K, Rettig J, Wirths O.
    Neurodegener Dis; 2008 Jul 30; 5(3-4):140-2. PubMed ID: 18322372
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  • 10. Axonopathy in an APP/PS1 transgenic mouse model of Alzheimer's disease.
    Wirths O, Weis J, Szczygielski J, Multhaup G, Bayer TA.
    Acta Neuropathol; 2006 Apr 30; 111(4):312-9. PubMed ID: 16520967
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  • 11. Deposition of mouse amyloid beta in human APP/PS1 double and single AD model transgenic mice.
    van Groen T, Kiliaan AJ, Kadish I.
    Neurobiol Dis; 2006 Sep 30; 23(3):653-62. PubMed ID: 16829076
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  • 12. Age-related changes of neuron numbers in the frontal cortex of a transgenic mouse model of Alzheimer's disease.
    Lemmens MA, Sierksma AS, Rutten BP, Dennissen F, Steinbusch HW, Lucassen PJ, Schmitz C.
    Brain Struct Funct; 2011 Sep 30; 216(3):227-37. PubMed ID: 21409417
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  • 13. Non-tau based neuronal degeneration in Alzheimer's disease -- an immunocytochemical and quantitative study in the supragranular layers of the middle temporal neocortex.
    van de Nes JA, Nafe R, Schlote W.
    Brain Res; 2008 Jun 05; 1213():152-65. PubMed ID: 18455153
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  • 14. Age-related accumulation of Reelin in amyloid-like deposits.
    Knuesel I, Nyffeler M, Mormède C, Muhia M, Meyer U, Pietropaolo S, Yee BK, Pryce CR, LaFerla FM, Marighetto A, Feldon J.
    Neurobiol Aging; 2009 May 05; 30(5):697-716. PubMed ID: 17904250
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  • 18. Amyloid precursor protein cytoplasmic domain with phospho-Thr668 accumulates in Alzheimer's disease and its transgenic models: a role to mediate interaction of Abeta and tau.
    Shin RW, Ogino K, Shimabuku A, Taki T, Nakashima H, Ishihara T, Kitamoto T.
    Acta Neuropathol; 2007 Jun 05; 113(6):627-36. PubMed ID: 17431643
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  • 19. Phosphorylated tau in neuritic plaques of APP(sw)/Tau (vlw) transgenic mice and Alzheimer disease.
    Pérez M, Morán MA, Ferrer I, Avila J, Gómez-Ramos P.
    Acta Neuropathol; 2008 Oct 05; 116(4):409-18. PubMed ID: 18679696
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