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Journal Abstract Search

253 related items for PubMed ID: 18978814

  • 1. A positive role for Myc in TGFbeta-induced Snail transcription and epithelial-to-mesenchymal transition.
    Smith AP, Verrecchia A, Fagà G, Doni M, Perna D, Martinato F, Guccione E, Amati B.
    Oncogene; 2009 Jan 22; 28(3):422-30. PubMed ID: 18978814
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  • 2. Smad2 and Smad3 cooperate and antagonize simultaneously in vertebrate neurogenesis.
    Míguez DG, Gil-Guiñón E, Pons S, Martí E.
    J Cell Sci; 2013 Dec 01; 126(Pt 23):5335-43. PubMed ID: 24105267
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  • 4. Hematopoiesis controlled by distinct TIF1gamma and Smad4 branches of the TGFbeta pathway.
    He W, Dorn DC, Erdjument-Bromage H, Tempst P, Moore MA, Massagué J.
    Cell; 2006 Jun 02; 125(5):929-41. PubMed ID: 16751102
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  • 5. The tumor suppressor KLF11 mediates a novel mechanism in transforming growth factor beta-induced growth inhibition that is inactivated in pancreatic cancer.
    Buck A, Buchholz M, Wagner M, Adler G, Gress T, Ellenrieder V.
    Mol Cancer Res; 2006 Nov 02; 4(11):861-72. PubMed ID: 17114344
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  • 6. Snail is required for transforming growth factor-beta-induced epithelial-mesenchymal transition by activating PI3 kinase/Akt signal pathway.
    Cho HJ, Baek KE, Saika S, Jeong MJ, Yoo J.
    Biochem Biophys Res Commun; 2007 Feb 09; 353(2):337-43. PubMed ID: 17187756
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  • 9. A novel approach for the generation of genetically modified mammary epithelial cell cultures yields new insights into TGFβ signaling in the mammary gland.
    Kohn EA, Du Z, Sato M, Van Schyndle CM, Welsh MA, Yang YA, Stuelten CH, Tang B, Ju W, Bottinger EP, Wakefield LM.
    Breast Cancer Res; 2010 Feb 09; 12(5):R83. PubMed ID: 20942910
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  • 11. Integration of TGF-beta/Smad and Jagged1/Notch signalling in epithelial-to-mesenchymal transition.
    Zavadil J, Cermak L, Soto-Nieves N, Böttinger EP.
    EMBO J; 2004 Mar 10; 23(5):1155-65. PubMed ID: 14976548
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  • 12. The transcription factor GLI1 interacts with SMAD proteins to modulate transforming growth factor β-induced gene expression in a p300/CREB-binding protein-associated factor (PCAF)-dependent manner.
    Nye MD, Almada LL, Fernandez-Barrena MG, Marks DL, Elsawa SF, Vrabel A, Tolosa EJ, Ellenrieder V, Fernandez-Zapico ME.
    J Biol Chem; 2014 May 30; 289(22):15495-506. PubMed ID: 24739390
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  • 14. Specific interactions between Smad proteins and AP-1 components determine TGFβ-induced breast cancer cell invasion.
    Sundqvist A, Zieba A, Vasilaki E, Herrera Hidalgo C, Söderberg O, Koinuma D, Miyazono K, Heldin CH, Landegren U, Ten Dijke P, van Dam H.
    Oncogene; 2013 Aug 01; 32(31):3606-15. PubMed ID: 22926518
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  • 16. LMO4 can interact with Smad proteins and modulate transforming growth factor-beta signaling in epithelial cells.
    Lu Z, Lam KS, Wang N, Xu X, Cortes M, Andersen B.
    Oncogene; 2006 May 11; 25(20):2920-30. PubMed ID: 16331278
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  • 17. SMAD3 and SMAD4 have a more dominant role than SMAD2 in TGFβ-induced chondrogenic differentiation of bone marrow-derived mesenchymal stem cells.
    de Kroon LM, Narcisi R, van den Akker GG, Vitters EL, Blaney Davidson EN, van Osch GJ, van der Kraan PM.
    Sci Rep; 2017 Feb 27; 7():43164. PubMed ID: 28240243
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  • 18. Long Noncoding RNA ELIT-1 Acts as a Smad3 Cofactor to Facilitate TGFβ/Smad Signaling and Promote Epithelial-Mesenchymal Transition.
    Sakai S, Ohhata T, Kitagawa K, Uchida C, Aoshima T, Niida H, Suzuki T, Inoue Y, Miyazawa K, Kitagawa M.
    Cancer Res; 2019 Jun 01; 79(11):2821-2838. PubMed ID: 30952633
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