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Journal Abstract Search

208 related items for PubMed ID: 19071115

  • 1. Increased persistent sodium current determines cortical hyperexcitability in a genetic model of amyotrophic lateral sclerosis.
    Pieri M, Carunchio I, Curcio L, Mercuri NB, Zona C.
    Exp Neurol; 2009 Feb; 215(2):368-79. PubMed ID: 19071115
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  • 2. Over-expression of N-type calcium channels in cortical neurons from a mouse model of Amyotrophic Lateral Sclerosis.
    Pieri M, Caioli S, Canu N, Mercuri NB, Guatteo E, Zona C.
    Exp Neurol; 2013 Sep; 247():349-58. PubMed ID: 23142186
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  • 4. Voltage-dependent sodium channels in spinal cord motor neurons display rapid recovery from fast inactivation in a mouse model of amyotrophic lateral sclerosis.
    Zona C, Pieri M, Carunchio I.
    J Neurophysiol; 2006 Dec; 96(6):3314-22. PubMed ID: 16899637
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  • 5. Increased levels of p70S6 phosphorylation in the G93A mouse model of Amyotrophic Lateral Sclerosis and in valine-exposed cortical neurons in culture.
    Carunchio I, Curcio L, Pieri M, Pica F, Caioli S, Viscomi MT, Molinari M, Canu N, Bernardi G, Zona C.
    Exp Neurol; 2010 Nov; 226(1):218-30. PubMed ID: 20832409
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  • 6. Zinc pre-treatment enhances NMDAR-mediated excitotoxicity in cultured cortical neurons from SOD1(G93A) mouse, a model of amyotrophic lateral sclerosis.
    Nutini M, Frazzini V, Marini C, Spalloni A, Sensi SL, Longone P.
    Neuropharmacology; 2011 Jun; 60(7-8):1200-8. PubMed ID: 21056589
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  • 7. Persistent sodium currents in mesencephalic v neurons participate in burst generation and control of membrane excitability.
    Wu N, Enomoto A, Tanaka S, Hsiao CF, Nykamp DQ, Izhikevich E, Chandler SH.
    J Neurophysiol; 2005 May; 93(5):2710-22. PubMed ID: 15625100
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  • 15. Overexpression of human mutated G93A SOD1 changes dynamics of the ER mitochondria calcium cycle specifically in mouse embryonic motor neurons.
    Lautenschläger J, Prell T, Ruhmer J, Weidemann L, Witte OW, Grosskreutz J.
    Exp Neurol; 2013 Sep; 247():91-100. PubMed ID: 23578819
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  • 16. Neonatal neuronal circuitry shows hyperexcitable disturbance in a mouse model of the adult-onset neurodegenerative disease amyotrophic lateral sclerosis.
    van Zundert B, Peuscher MH, Hynynen M, Chen A, Neve RL, Brown RH, Constantine-Paton M, Bellingham MC.
    J Neurosci; 2008 Oct 22; 28(43):10864-74. PubMed ID: 18945894
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  • 17. GAB(A) receptors present higher affinity and modified subunit composition in spinal motor neurons from a genetic model of amyotrophic lateral sclerosis.
    Carunchio I, Mollinari C, Pieri M, Merlo D, Zona C.
    Eur J Neurosci; 2008 Oct 22; 28(7):1275-85. PubMed ID: 18973555
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  • 18. Altered long-term corticostriatal synaptic plasticity in transgenic mice overexpressing human CU/ZN superoxide dismutase (GLY(93)-->ALA) mutation.
    Geracitano R, Paolucci E, Prisco S, Guatteo E, Zona C, Longone P, Ammassari-Teule M, Bernardi G, Berretta N, Mercuri NB.
    Neuroscience; 2003 Oct 22; 118(2):399-408. PubMed ID: 12699776
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  • 19. Sodium currents in medullary neurons isolated from the pre-Bötzinger complex region.
    Ptak K, Zummo GG, Alheid GF, Tkatch T, Surmeier DJ, McCrimmon DR.
    J Neurosci; 2005 May 25; 25(21):5159-70. PubMed ID: 15917456
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  • 20. Differential effect of D623N variant and wild-type Na(v)1.7 sodium channels on resting potential and interspike membrane potential of dorsal root ganglion neurons.
    Ahn HS, Vasylyev DV, Estacion M, Macala LJ, Shah P, Faber CG, Merkies IS, Dib-Hajj SD, Waxman SG.
    Brain Res; 2013 Sep 05; 1529():165-77. PubMed ID: 23850641
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