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PUBMED FOR HANDHELDS

Journal Abstract Search


603 related items for PubMed ID: 19131648

  • 1. miR-1 overexpression enhances Ca(2+) release and promotes cardiac arrhythmogenesis by targeting PP2A regulatory subunit B56alpha and causing CaMKII-dependent hyperphosphorylation of RyR2.
    Terentyev D, Belevych AE, Terentyeva R, Martin MM, Malana GE, Kuhn DE, Abdellatif M, Feldman DS, Elton TS, Györke S.
    Circ Res; 2009 Feb 27; 104(4):514-21. PubMed ID: 19131648
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  • 2. Calcium/calmodulin-dependent protein kinase II contributes to cardiac arrhythmogenesis in heart failure.
    Sag CM, Wadsack DP, Khabbazzadeh S, Abesser M, Grefe C, Neumann K, Opiela MK, Backs J, Olson EN, Brown JH, Neef S, Maier SK, Maier LS.
    Circ Heart Fail; 2009 Nov 27; 2(6):664-75. PubMed ID: 19919992
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  • 3. Calcium-calmodulin kinase II mediates digitalis-induced arrhythmias.
    Gonano LA, Sepúlveda M, Rico Y, Kaetzel M, Valverde CA, Dedman J, Mattiazzi A, Vila Petroff M.
    Circ Arrhythm Electrophysiol; 2011 Dec 27; 4(6):947-57. PubMed ID: 22009705
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  • 12. Increased intracellular Ca2+ and SR Ca2+ load contribute to arrhythmias after acidosis in rat heart. Role of Ca2+/calmodulin-dependent protein kinase II.
    Said M, Becerra R, Palomeque J, Rinaldi G, Kaetzel MA, Diaz-Sylvester PL, Copello JA, Dedman JR, Mundiña-Weilenmann C, Vittone L, Mattiazzi A.
    Am J Physiol Heart Circ Physiol; 2008 Oct 27; 295(4):H1669-83. PubMed ID: 18723772
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  • 13. A Single Protein Kinase A or Calmodulin Kinase II Site Does Not Control the Cardiac Pacemaker Ca2+ Clock.
    Wu Y, Valdivia HH, Wehrens XH, Anderson ME.
    Circ Arrhythm Electrophysiol; 2016 Feb 27; 9(2):e003180. PubMed ID: 26857906
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  • 14. Ca2+/calmodulin-dependent protein kinase modulates cardiac ryanodine receptor phosphorylation and sarcoplasmic reticulum Ca2+ leak in heart failure.
    Ai X, Curran JW, Shannon TR, Bers DM, Pogwizd SM.
    Circ Res; 2005 Dec 09; 97(12):1314-22. PubMed ID: 16269653
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  • 17. Dual ablation of the RyR2-Ser2808 and RyR2-Ser2814 sites increases propensity for pro-arrhythmic spontaneous Ca2+ releases.
    Janicek R, Camors EM, Potenza DM, Fernandez-Tenorio M, Zhao Y, Dooge HC, Loaiza R, Alvarado FJ, Egger M, Valdivia HH, Niggli E.
    J Physiol; 2024 Oct 09; 602(20):5179-5201. PubMed ID: 39316734
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  • 19. Ryanodine receptor phosphorylation by CaMKII promotes spontaneous Ca(2+) release events in a rodent model of early stage diabetes: The arrhythmogenic substrate.
    Sommese L, Valverde CA, Blanco P, Castro MC, Rueda OV, Kaetzel M, Dedman J, Anderson ME, Mattiazzi A, Palomeque J.
    Int J Cardiol; 2016 Jan 01; 202():394-406. PubMed ID: 26432489
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  • 20. The role of spatial organization of Ca2+ release sites in the generation of arrhythmogenic diastolic Ca2+ release in myocytes from failing hearts.
    Belevych AE, Ho HT, Bonilla IM, Terentyeva R, Schober KE, Terentyev D, Carnes CA, Györke S.
    Basic Res Cardiol; 2017 Jul 01; 112(4):44. PubMed ID: 28612155
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