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171 related items for PubMed ID: 19147786

  • 1. Wound healing defect of Vav3-/- mice due to impaired {beta}2-integrin-dependent macrophage phagocytosis of apoptotic neutrophils.
    Sindrilaru A, Peters T, Schymeinsky J, Oreshkova T, Wang H, Gompf A, Mannella F, Wlaschek M, Sunderkötter C, Rudolph KL, Walzog B, Bustelo XR, Fischer KD, Scharffetter-Kochanek K.
    Blood; 2009 May 21; 113(21):5266-76. PubMed ID: 19147786
    [Abstract] [Full Text] [Related]

  • 2. Wound-healing defect of CD18(-/-) mice due to a decrease in TGF-beta1 and myofibroblast differentiation.
    Peters T, Sindrilaru A, Hinz B, Hinrichs R, Menke A, Al-Azzeh EA, Holzwarth K, Oreshkova T, Wang H, Kess D, Walzog B, Sulyok S, Sunderkötter C, Friedrich W, Wlaschek M, Krieg T, Scharffetter-Kochanek K.
    EMBO J; 2005 Oct 05; 24(19):3400-10. PubMed ID: 16148944
    [Abstract] [Full Text] [Related]

  • 3. Vav GEFs are required for beta2 integrin-dependent functions of neutrophils.
    Gakidis MA, Cullere X, Olson T, Wilsbacher JL, Zhang B, Moores SL, Ley K, Swat W, Mayadas T, Brugge JS.
    J Cell Biol; 2004 Jul 19; 166(2):273-82. PubMed ID: 15249579
    [Abstract] [Full Text] [Related]

  • 4. Requirements for Vav guanine nucleotide exchange factors and Rho GTPases in FcgammaR- and complement-mediated phagocytosis.
    Hall AB, Gakidis MA, Glogauer M, Wilsbacher JL, Gao S, Swat W, Brugge JS.
    Immunity; 2006 Mar 19; 24(3):305-16. PubMed ID: 16546099
    [Abstract] [Full Text] [Related]

  • 5. Nonphlogistic clearance of late apoptotic neutrophils by macrophages: efficient phagocytosis independent of beta 2 integrins.
    Ren Y, Stuart L, Lindberg FP, Rosenkranz AR, Chen Y, Mayadas TN, Savill J.
    J Immunol; 2001 Apr 01; 166(7):4743-50. PubMed ID: 11254736
    [Abstract] [Full Text] [Related]

  • 6. Vav proteins in neutrophils are required for FcgammaR-mediated signaling to Rac GTPases and nicotinamide adenine dinucleotide phosphate oxidase component p40(phox).
    Utomo A, Cullere X, Glogauer M, Swat W, Mayadas TN.
    J Immunol; 2006 Nov 01; 177(9):6388-97. PubMed ID: 17056570
    [Abstract] [Full Text] [Related]

  • 7. Deficiency of Src family kinases p59/61hck and p58c-fgr results in defective adhesion-dependent neutrophil functions.
    Lowell CA, Fumagalli L, Berton G.
    J Cell Biol; 1996 May 01; 133(4):895-910. PubMed ID: 8666673
    [Abstract] [Full Text] [Related]

  • 8. CD18 in monogenic and polygenic inflammatory processes of the skin.
    Peters T, Sindrilaru A, Wang H, Oreshkova T, Renkl AC, Kess D, Scharffetter-Kochanek K.
    J Investig Dermatol Symp Proc; 2006 Sep 01; 11(1):7-15. PubMed ID: 17069006
    [Abstract] [Full Text] [Related]

  • 9. Macrophage peroxisome proliferator-activated receptor γ deficiency delays skin wound healing through impairing apoptotic cell clearance in mice.
    Chen H, Shi R, Luo B, Yang X, Qiu L, Xiong J, Jiang M, Liu Y, Zhang Z, Wu Y.
    Cell Death Dis; 2015 Jan 15; 6(1):e1597. PubMed ID: 25590807
    [Abstract] [Full Text] [Related]

  • 10. Major transcript variants of VAV3, a new member of the VAV family of guanine nucleotide exchange factors.
    Trenkle T, McClelland M, Adlkofer K, Welsh J.
    Gene; 2000 Mar 07; 245(1):139-49. PubMed ID: 10713454
    [Abstract] [Full Text] [Related]

  • 11. Chemokine receptor CX3CR1 mediates skin wound healing by promoting macrophage and fibroblast accumulation and function.
    Ishida Y, Gao JL, Murphy PM.
    J Immunol; 2008 Jan 01; 180(1):569-79. PubMed ID: 18097059
    [Abstract] [Full Text] [Related]

  • 12. The Vav binding site of the non-receptor tyrosine kinase Syk at Tyr 348 is critical for beta2 integrin (CD11/CD18)-mediated neutrophil migration.
    Schymeinsky J, Sindrilaru A, Frommhold D, Sperandio M, Gerstl R, Then C, Mócsai A, Scharffetter-Kochanek K, Walzog B.
    Blood; 2006 Dec 01; 108(12):3919-27. PubMed ID: 16882714
    [Abstract] [Full Text] [Related]

  • 13. Impaired TGF-β signaling and a defect in resolution of inflammation contribute to delayed wound healing in a female rat model of type 2 diabetes.
    Al-Mulla F, Leibovich SJ, Francis IM, Bitar MS.
    Mol Biosyst; 2011 Nov 01; 7(11):3006-20. PubMed ID: 21850315
    [Abstract] [Full Text] [Related]

  • 14. The β-glucan receptor Dectin-1 activates the integrin Mac-1 in neutrophils via Vav protein signaling to promote Candida albicans clearance.
    Li X, Utomo A, Cullere X, Choi MM, Milner DA, Venkatesh D, Yun SH, Mayadas TN.
    Cell Host Microbe; 2011 Dec 15; 10(6):603-15. PubMed ID: 22177564
    [Abstract] [Full Text] [Related]

  • 15. A Frog-Derived Immunomodulatory Peptide Promotes Cutaneous Wound Healing by Regulating Cellular Response.
    He X, Yang Y, Mu L, Zhou Y, Chen Y, Wu J, Wang Y, Yang H, Li M, Xu W, Wei L.
    Front Immunol; 2019 Dec 15; 10():2421. PubMed ID: 31681309
    [Abstract] [Full Text] [Related]

  • 16. IL-36 receptor antagonist deficiency resulted in delayed wound healing due to excessive recruitment of immune cells.
    Saito K, Iwata Y, Fukushima H, Watanabe S, Tanaka Y, Hasegawa Y, Akiyama M, Sugiura K.
    Sci Rep; 2020 Sep 08; 10(1):14772. PubMed ID: 32901055
    [Abstract] [Full Text] [Related]

  • 17. A transgenic mouse model of inducible macrophage depletion: effects of diphtheria toxin-driven lysozyme M-specific cell lineage ablation on wound inflammatory, angiogenic, and contractive processes.
    Goren I, Allmann N, Yogev N, Schürmann C, Linke A, Holdener M, Waisman A, Pfeilschifter J, Frank S.
    Am J Pathol; 2009 Jul 08; 175(1):132-47. PubMed ID: 19528348
    [Abstract] [Full Text] [Related]

  • 18. The guanine nucleotide exchange factor Vav3 regulates differentiation of progenitor cells in the developing mouse retina.
    Luft V, Reinhard J, Shibuya M, Fischer KD, Faissner A.
    Cell Tissue Res; 2015 Feb 08; 359(2):423-440. PubMed ID: 25501893
    [Abstract] [Full Text] [Related]

  • 19. The phosphatidylserine receptor TIM4 utilizes integrins as coreceptors to effect phagocytosis.
    Flannagan RS, Canton J, Furuya W, Glogauer M, Grinstein S.
    Mol Biol Cell; 2014 May 08; 25(9):1511-22. PubMed ID: 24623723
    [Abstract] [Full Text] [Related]

  • 20. Distinct role of phosphatidylinositol 3-kinase and Rho family GTPases in Vav3-induced cell transformation, cell motility, and morphological changes.
    Sachdev P, Zeng L, Wang LH.
    J Biol Chem; 2002 May 17; 277(20):17638-48. PubMed ID: 11884391
    [Abstract] [Full Text] [Related]

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