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Journal Abstract Search

246 related items for PubMed ID: 19187267

  • 1. Increase in presynaptic territory of C-terminals on lumbar motoneurons of G93A SOD1 mice during disease progression.
    Pullen AH, Athanasiou D.
    Eur J Neurosci; 2009 Feb; 29(3):551-61. PubMed ID: 19187267
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  • 3. Gender-specific perturbations in modulatory inputs to motoneurons in a mouse model of amyotrophic lateral sclerosis.
    Herron LR, Miles GB.
    Neuroscience; 2012 Dec 13; 226():313-23. PubMed ID: 23000617
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  • 4. Altered development in GABA co-release shapes glycinergic synaptic currents in cultured spinal slices of the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Medelin M, Rancic V, Cellot G, Laishram J, Veeraraghavan P, Rossi C, Muzio L, Sivilotti L, Ballerini L.
    J Physiol; 2016 Jul 01; 594(13):3827-40. PubMed ID: 27098371
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  • 5. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
    Jaarsma D, Haasdijk ED, Grashorn JA, Hawkins R, van Duijn W, Verspaget HW, London J, Holstege JC.
    Neurobiol Dis; 2000 Dec 01; 7(6 Pt B):623-43. PubMed ID: 11114261
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  • 7. Excitatory action of low frequency depolarizing GABA/glycine synaptic inputs is prevalent in prenatal spinal SOD1G93A motoneurons.
    Zhu H, Dalvi U, Cazenave W, Cattaert D, Branchereau P.
    J Physiol; 2024 Mar 01; 602(5):913-932. PubMed ID: 38345477
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  • 8. The effect of peripheral nerve injury on disease progression in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Sharp PS, Dick JR, Greensmith L.
    Neuroscience; 2005 Mar 01; 130(4):897-910. PubMed ID: 15652988
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  • 9. Early excitability changes in lumbar motoneurons of transgenic SOD1G85R and SOD1G(93A-Low) mice.
    Pambo-Pambo A, Durand J, Gueritaud JP.
    J Neurophysiol; 2009 Dec 01; 102(6):3627-42. PubMed ID: 19828728
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  • 10. Early electrophysiological abnormalities in lumbar motoneurons in a transgenic mouse model of amyotrophic lateral sclerosis.
    Bories C, Amendola J, Lamotte d'Incamps B, Durand J.
    Eur J Neurosci; 2007 Jan 01; 25(2):451-9. PubMed ID: 17284186
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  • 11. Neuregulin 1 confers neuroprotection in SOD1-linked amyotrophic lateral sclerosis mice via restoration of C-boutons of spinal motor neurons.
    Lasiene J, Komine O, Fujimori-Tonou N, Powers B, Endo F, Watanabe S, Shijie J, Ravits J, Horner P, Misawa H, Yamanaka K.
    Acta Neuropathol Commun; 2016 Feb 18; 4():15. PubMed ID: 26891847
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  • 12. Hyperexcitability of cultured spinal motoneurons from presymptomatic ALS mice.
    Kuo JJ, Schonewille M, Siddique T, Schults AN, Fu R, Bär PR, Anelli R, Heckman CJ, Kroese AB.
    J Neurophysiol; 2004 Jan 18; 91(1):571-5. PubMed ID: 14523070
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  • 14. Compensatory changes in degenerating spinal motoneurons sustain functional sparing in the SOD1-G93A mouse model of amyotrophic lateral sclerosis.
    Giusto E, Codrich M, de Leo G, Francardo V, Coradazzi M, Parenti R, Gulisano M, Vicario N, Gulino R, Leanza G.
    J Comp Neurol; 2020 Feb 01; 528(2):231-243. PubMed ID: 31364764
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  • 15. Ultrastructural changes in lumbar spinal cord in transgenic SOD1G93A rats.
    Fidziańska A, Gadamski R, Rafałowska J, Chrzanowska H, Grieb P.
    Folia Neuropathol; 2006 Feb 01; 44(3):175-82. PubMed ID: 17039412
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  • 16. The vulnerability of spinal motoneurons and soma size plasticity in a mouse model of amyotrophic lateral sclerosis.
    Dukkipati SS, Garrett TL, Elbasiouny SM.
    J Physiol; 2018 May 01; 596(9):1723-1745. PubMed ID: 29502344
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  • 18. Postnatal electrical and morphological abnormalities in lumbar motoneurons from transgenic mouse models of amyotrophic lateral sclerosis.
    Amendola J, Gueritaud JP, Lamotte d'Incamps B, Bories C, Liabeuf S, Allene C, Pambo-Pambo A, Durand J.
    Arch Ital Biol; 2007 Nov 01; 145(3-4):311-23. PubMed ID: 18075124
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  • 19. Postnatal dendritic development in lumbar motoneurons in mutant superoxide dismutase 1 mouse model of amyotrophic lateral sclerosis.
    Filipchuk AA, Durand J.
    Neuroscience; 2012 May 03; 209():144-54. PubMed ID: 22387111
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  • 20. Early Hypoexcitability in a Subgroup of Spinal Motoneurons in Superoxide Dismutase 1 Transgenic Mice, a Model of Amyotrophic Lateral Sclerosis.
    Filipchuk A, Pambo-Pambo A, Gaudel F, Liabeuf S, Brocard C, Gueritaud JP, Durand J.
    Neuroscience; 2021 May 21; 463():337-353. PubMed ID: 33556455
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