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167 related items for PubMed ID: 19196551

  • 1. Accessory cell dependent NK cell mediated PBMC IFN-gamma production is defective in HIV infection.
    Yonkers NL, Milkovich KA, Rodriguez B, Post AB, Asaad R, Heinzel FP, Valdez H, Tary-Lehmann M, Anthony DD.
    Clin Immunol; 2009 May; 131(2):288-97. PubMed ID: 19196551
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  • 3. IFN-γ production by human natural killer cells in response to HCV-infected hepatoma cells is dependent on accessory cells.
    Zhang S, Saha B, Kodys K, Szabo G.
    J Hepatol; 2013 Sep; 59(3):442-9. PubMed ID: 23665181
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  • 4. Restoration of TLR3-activated myeloid dendritic cell activity leads to improved natural killer cell function in chronic hepatitis B virus infection.
    Tjwa ET, van Oord GW, Biesta PJ, Boonstra A, Janssen HL, Woltman AM.
    J Virol; 2012 Apr; 86(8):4102-9. PubMed ID: 22318141
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  • 5. HCV viraemia associates with NK cell activation and dysfunction in antiretroviral therapy-treated HIV/HCV-co-infected subjects.
    Papasavvas E, Azzoni L, Yin X, Liu Q, Joseph J, Mackiewicz A, Ross B, Lynn KM, Jacobson JM, Mounzer K, Kostman JR, Montaner LJ.
    J Viral Hepat; 2017 Oct; 24(10):865-876. PubMed ID: 28419653
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  • 6. Selective impairments in dendritic cell-associated function distinguish hepatitis C virus and HIV infection.
    Anthony DD, Yonkers NL, Post AB, Asaad R, Heinzel FP, Lederman MM, Lehmann PV, Valdez H.
    J Immunol; 2004 Apr 15; 172(8):4907-16. PubMed ID: 15067070
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  • 7. CD3(+)CD56(+) Natural Killer-Like T Cells Display Anti-HCV Activity but Are Functionally Impaired in HIV(+) Patients With Acute Hepatitis C.
    Kokordelis P, Krämer B, Boesecke C, Voigt E, Ingiliz P, Glässner A, Wolter F, Srassburg CP, Spengler U, Rockstroh JK, Nattermann J.
    J Acquir Immune Defic Syndr; 2015 Dec 01; 70(4):338-46. PubMed ID: 26509931
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  • 10. Loss of NK stimulatory capacity by plasmacytoid and monocyte-derived DC but not myeloid DC in HIV-1 infected patients.
    Benlahrech A, Gotch F, Kelleher P, Patterson S.
    PLoS One; 2011 Mar 08; 6(3):e17525. PubMed ID: 21408163
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  • 11. An effective interferon-gamma-mediated inhibition of hepatitis C virus replication by natural killer cells is associated with spontaneous clearance of acute hepatitis C in human immunodeficiency virus-positive patients.
    Kokordelis P, Krämer B, Körner C, Boesecke C, Voigt E, Ingiliz P, Glässner A, Eisenhardt M, Wolter F, Kaczmarek D, Nischalke HD, Rockstroh JK, Spengler U, Nattermann J.
    Hepatology; 2014 Mar 08; 59(3):814-27. PubMed ID: 24382664
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  • 12. Hepatitis C Virus-Induced Myeloid-Derived Suppressor Cells Suppress NK Cell IFN-γ Production by Altering Cellular Metabolism via Arginase-1.
    Goh CC, Roggerson KM, Lee HC, Golden-Mason L, Rosen HR, Hahn YS.
    J Immunol; 2016 Mar 01; 196(5):2283-92. PubMed ID: 26826241
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  • 13. Interferon α-stimulated natural killer cells from patients with acute hepatitis C virus (HCV) infection recognize HCV-infected and uninfected hepatoma cells via DNAX accessory molecule-1.
    Stegmann KA, Björkström NK, Ciesek S, Lunemann S, Jaroszewicz J, Wiegand J, Malinski P, Dustin LB, Rice CM, Manns MP, Pietschmann T, Cornberg M, Ljunggren HG, Wedemeyer H.
    J Infect Dis; 2012 May 01; 205(9):1351-62. PubMed ID: 22457290
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  • 14. Hepatitis C virus impairs natural killer cell activity via viral serine protease NS3.
    Yang CM, Yoon JC, Park JH, Lee JM.
    PLoS One; 2017 May 01; 12(4):e0175793. PubMed ID: 28410411
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  • 16. IL-10+ NK and TGF-β+ NK cells play negative regulatory roles in HIV infection.
    Jiang Y, Yang M, Sun X, Chen X, Ma M, Yin X, Qian S, Zhang Z, Fu Y, Liu J, Han X, Xu J, Shang H.
    BMC Infect Dis; 2018 Feb 13; 18(1):80. PubMed ID: 29439673
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  • 18. Impaired CD4⁺ T cell stimulation of NK cell anti-fibrotic activity may contribute to accelerated liver fibrosis progression in HIV/HCV patients.
    Glässner A, Eisenhardt M, Kokordelis P, Krämer B, Wolter F, Nischalke HD, Boesecke C, Sauerbruch T, Rockstroh JK, Spengler U, Nattermann J.
    J Hepatol; 2013 Sep 13; 59(3):427-33. PubMed ID: 23665286
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  • 20. Characterization of the defective interaction between a subset of natural killer cells and dendritic cells in HIV-1 infection.
    Mavilio D, Lombardo G, Kinter A, Fogli M, La Sala A, Ortolano S, Farschi A, Follmann D, Gregg R, Kovacs C, Marcenaro E, Pende D, Moretta A, Fauci AS.
    J Exp Med; 2006 Oct 02; 203(10):2339-50. PubMed ID: 17000867
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