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Journal Abstract Search

249 related items for PubMed ID: 19233849

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  • 2. Caspase-mediated Bak activation and cytochrome c release during intrinsic apoptotic cell death in Jurkat cells.
    Shawgo ME, Shelton SN, Robertson JD.
    J Biol Chem; 2008 Dec 19; 283(51):35532-8. PubMed ID: 18927073
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  • 3. A role for mitochondrial Bak in apoptotic response to anticancer drugs.
    Wang GQ, Gastman BR, Wieckowski E, Goldstein LA, Gambotto A, Kim TH, Fang B, Rabinovitz A, Yin XM, Rabinowich H.
    J Biol Chem; 2001 Sep 07; 276(36):34307-17. PubMed ID: 11447222
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  • 6. Caspase-9 activation by the apoptosome is not required for fas-mediated apoptosis in type II Jurkat cells.
    Shawgo ME, Shelton SN, Robertson JD.
    J Biol Chem; 2009 Nov 27; 284(48):33447-55. PubMed ID: 19758996
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  • 7. The role of Apaf-1, caspase-9, and bid proteins in etoposide- or paclitaxel-induced mitochondrial events during apoptosis.
    Perkins CL, Fang G, Kim CN, Bhalla KN.
    Cancer Res; 2000 Mar 15; 60(6):1645-53. PubMed ID: 10749135
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  • 8. The caspase-8/Bid/cytochrome c axis links signals from death receptors to mitochondrial reactive oxygen species production.
    Kim WS, Lee KS, Kim JH, Kim CK, Lee G, Choe J, Won MH, Kim TH, Jeoung D, Lee H, Kim JY, Ae Jeong M, Ha KS, Kwon YG, Kim YM.
    Free Radic Biol Med; 2017 Nov 15; 112():567-577. PubMed ID: 28888620
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  • 9. Activation of caspase-9, but not caspase-2 or caspase-8, is essential for heat-induced apoptosis in Jurkat cells.
    Shelton SN, Dillard CD, Robertson JD.
    J Biol Chem; 2010 Dec 24; 285(52):40525-33. PubMed ID: 20978129
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  • 10. Requirement of Apaf-1 for mitochondrial events and the cleavage or activation of all procaspases during genotoxic stress-induced apoptosis.
    Franklin EE, Robertson JD.
    Biochem J; 2007 Jul 01; 405(1):115-22. PubMed ID: 17348858
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  • 12. Galectin-1 induced activation of the mitochondrial apoptotic pathway: evidence for a connection between death-receptor and mitochondrial pathways in human Jurkat T lymphocytes.
    Lange F, Brandt B, Tiedge M, Jonas L, Jeschke U, Pöhland R, Walzel H.
    Histochem Cell Biol; 2009 Aug 01; 132(2):211-23. PubMed ID: 19381674
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  • 13. Bcl-2 family member Bfl-1/A1 sequesters truncated bid to inhibit is collaboration with pro-apoptotic Bak or Bax.
    Werner AB, de Vries E, Tait SW, Bontjer I, Borst J.
    J Biol Chem; 2002 Jun 21; 277(25):22781-8. PubMed ID: 11929871
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  • 14. BID is cleaved by caspase-8 within a native complex on the mitochondrial membrane.
    Schug ZT, Gonzalvez F, Houtkooper RH, Vaz FM, Gottlieb E.
    Cell Death Differ; 2011 Mar 21; 18(3):538-48. PubMed ID: 21072056
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  • 16. Caspase-cleaved arrestin-2 and BID cooperatively facilitate cytochrome C release and cell death.
    Kook S, Zhan X, Cleghorn WM, Benovic JL, Gurevich VV, Gurevich EV.
    Cell Death Differ; 2014 Jan 21; 21(1):172-84. PubMed ID: 24141717
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  • 17. Resistance to granzyme B-mediated cytochrome c release in Bak-deficient cells.
    Wang GQ, Wieckowski E, Goldstein LA, Gastman BR, Rabinovitz A, Gambotto A, Li S, Fang B, Yin XM, Rabinowich H.
    J Exp Med; 2001 Nov 05; 194(9):1325-37. PubMed ID: 11696597
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  • 20. Bid can mediate a pro-apoptotic response to etoposide and ionizing radiation without cleavage in its unstructured loop and in the absence of p53.
    Maas C, de Vries E, Tait SW, Borst J.
    Oncogene; 2011 Aug 18; 30(33):3636-47. PubMed ID: 21423217
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