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295 related items for PubMed ID: 19276549

  • 1. Activation of cell cycle proteins in transgenic mice in response to neuronal loss but not amyloid-beta and tau pathology.
    Lopes JP, Blurton-Jones M, Yamasaki TR, Agostinho P, LaFerla FM.
    J Alzheimers Dis; 2009; 16(3):541-9. PubMed ID: 19276549
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  • 2. Genetically augmenting tau levels does not modulate the onset or progression of Abeta pathology in transgenic mice.
    Oddo S, Caccamo A, Cheng D, Jouleh B, Torp R, LaFerla FM.
    J Neurochem; 2007 Aug; 102(4):1053-63. PubMed ID: 17472708
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  • 8. Alpha1-antichymotrypsin, an inflammatory protein overexpressed in Alzheimer's disease brain, induces tau phosphorylation in neurons.
    Padmanabhan J, Levy M, Dickson DW, Potter H.
    Brain; 2006 Nov; 129(Pt 11):3020-34. PubMed ID: 16987932
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  • 9. Truncated tau at D421 is associated with neurodegeneration and tangle formation in the brain of Alzheimer transgenic models.
    Zhang Q, Zhang X, Sun A.
    Acta Neuropathol; 2009 Jun; 117(6):687-97. PubMed ID: 19190923
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  • 10. High tryptophan diet reduces CA1 intraneuronal β-amyloid in the triple transgenic mouse model of Alzheimer's disease.
    Noristani HN, Verkhratsky A, Rodríguez JJ.
    Aging Cell; 2012 Oct; 11(5):810-22. PubMed ID: 22702392
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  • 12. Effects of a tacrine-8-hydroxyquinoline hybrid (IQM-622) on Aβ accumulation and cell death: involvement in hippocampal neuronal loss in Alzheimer's disease.
    Antequera D, Bolos M, Spuch C, Pascual C, Ferrer I, Fernandez-Bachiller MI, Rodríguez-Franco MI, Carro E.
    Neurobiol Dis; 2012 Jun; 46(3):682-91. PubMed ID: 22426395
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  • 13. Altered cell cycle-related gene expression in brain and lymphocytes from a transgenic mouse model of Alzheimer's disease [amyloid precursor protein/presenilin 1 (PS1)].
    Esteras N, Bartolomé F, Alquézar C, Antequera D, Muñoz Ú, Carro E, Martín-Requero Á.
    Eur J Neurosci; 2012 Sep; 36(5):2609-18. PubMed ID: 22702220
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  • 14. High dietary consumption of trans fatty acids decreases brain docosahexaenoic acid but does not alter amyloid-beta and tau pathologies in the 3xTg-AD model of Alzheimer's disease.
    Phivilay A, Julien C, Tremblay C, Berthiaume L, Julien P, Giguère Y, Calon F.
    Neuroscience; 2009 Mar 03; 159(1):296-307. PubMed ID: 19135506
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  • 16. Amyloid-β and tau: the trigger and bullet in Alzheimer disease pathogenesis.
    Bloom GS.
    JAMA Neurol; 2014 Apr 03; 71(4):505-8. PubMed ID: 24493463
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  • 17. Human neural stem cells improve cognition and promote synaptic growth in two complementary transgenic models of Alzheimer's disease and neuronal loss.
    Ager RR, Davis JL, Agazaryan A, Benavente F, Poon WW, LaFerla FM, Blurton-Jones M.
    Hippocampus; 2015 Jul 03; 25(7):813-26. PubMed ID: 25530343
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  • 18. Abeta immunotherapy leads to clearance of early, but not late, hyperphosphorylated tau aggregates via the proteasome.
    Oddo S, Billings L, Kesslak JP, Cribbs DH, LaFerla FM.
    Neuron; 2004 Aug 05; 43(3):321-32. PubMed ID: 15294141
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  • 19. Phosphorylation of Tau at S422 is enhanced by Abeta in TauPS2APP triple transgenic mice.
    Grueninger F, Bohrmann B, Czech C, Ballard TM, Frey JR, Weidensteiner C, von Kienlin M, Ozmen L.
    Neurobiol Dis; 2010 Feb 05; 37(2):294-306. PubMed ID: 19781645
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  • 20. Reduction of aggregated Tau in neuronal processes but not in the cell bodies after Abeta42 immunisation in Alzheimer's disease.
    Boche D, Donald J, Love S, Harris S, Neal JW, Holmes C, Nicoll JA.
    Acta Neuropathol; 2010 Jul 05; 120(1):13-20. PubMed ID: 20532897
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