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103 related items for PubMed ID: 19287315
1. Mitochondrial alterations in transgenic mice with an H46R mutant Cu/Zn superoxide dismutase gene. Sasaki S, Aoki M, Nagai M, Kobayashi M, Itoyama Y. J Neuropathol Exp Neurol; 2009 Apr; 68(4):365-73. PubMed ID: 19287315 [Abstract] [Full Text] [Related]
6. ALS-associated mutant SOD1G93A causes mitochondrial vacuolation by expansion of the intermembrane space and by involvement of SOD1 aggregation and peroxisomes. Higgins CM, Jung C, Xu Z. BMC Neurosci; 2003 Jul 15; 4():16. PubMed ID: 12864925 [Abstract] [Full Text] [Related]
9. Loss of prion protein in a transgenic model of amyotrophic lateral sclerosis. Dupuis L, Mbebi C, Gonzalez de Aguilar JL, Rene F, Muller A, de Tapia M, Loeffler JP. Mol Cell Neurosci; 2002 Feb 15; 19(2):216-24. PubMed ID: 11860274 [Abstract] [Full Text] [Related]
10. Ultrastructural study of aggregates in the spinal cord of transgenic mice with a G93A mutant SOD1 gene. Sasaki S, Warita H, Murakami T, Shibata N, Komori T, Abe K, Kobayashi M, Iwata M. Acta Neuropathol; 2005 Mar 15; 109(3):247-55. PubMed ID: 15614580 [Abstract] [Full Text] [Related]
11. Familial amyotrophic lateral sclerosis with Gly93Ser mutation in Cu/Zn superoxide dismutase: a clinical and neuropathological study. Suzuki M, Irie T, Watanabe T, Mikami H, Yamazaki T, Oyanagi K, Ono S. J Neurol Sci; 2008 May 15; 268(1-2):140-4. PubMed ID: 18191946 [Abstract] [Full Text] [Related]
15. The Golgi apparatus of spinal cord motor neurons in transgenic mice expressing mutant Cu,Zn superoxide dismutase becomes fragmented in early, preclinical stages of the disease. Mourelatos Z, Gonatas NK, Stieber A, Gurney ME, Dal Canto MC. Proc Natl Acad Sci U S A; 1996 May 28; 93(11):5472-7. PubMed ID: 8643599 [Abstract] [Full Text] [Related]
17. [Molecular mechanism of ALS and a possible gene therapy]. Abe K, Warita H. Rinsho Shinkeigaku; 1999 Jan 28; 39(1):68-9. PubMed ID: 10377808 [Abstract] [Full Text] [Related]