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Journal Abstract Search

339 related items for PubMed ID: 19309449

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  • 23. beta-cell genes and diabetes: quantitative and qualitative differences in the pathophysiology of hepatic nuclear factor-1alpha and glucokinase mutations.
    Pearson ER, Velho G, Clark P, Stride A, Shepherd M, Frayling TM, Bulman MP, Ellard S, Froguel P, Hattersley AT.
    Diabetes; 2001 Feb; 50 Suppl 1():S101-7. PubMed ID: 11272165
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  • 25. Diagnosis of MODY in the offspring of parents with insulin-dependent and non-insulin-dependent diabetes mellitus.
    Guazzarotti L, Fumelli P, Testa I, Pecora R, Panicari F, Bellanné-Chantelot C, Bartolotta E.
    J Pediatr Endocrinol Metab; 2001 Feb; 14 Suppl 1():611-7. PubMed ID: 11393552
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  • 26. Prevalence of vascular complications among patients with glucokinase mutations and prolonged, mild hyperglycemia.
    Steele AM, Shields BM, Wensley KJ, Colclough K, Ellard S, Hattersley AT.
    JAMA; 2014 Jan 15; 311(3):279-86. PubMed ID: 24430320
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  • 29. Genetic testing for glucokinase mutations in clinically selected patients with MODY: a worthwhile investment.
    Schnyder S, Mullis PE, Ellard S, Hattersley AT, Flück CE.
    Swiss Med Wkly; 2005 Jun 11; 135(23-24):352-6. PubMed ID: 16059790
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  • 32. Identification of a novel beta-cell glucokinase (GCK) promoter mutation (-71G>C) that modulates GCK gene expression through loss of allele-specific Sp1 binding causing mild fasting hyperglycemia in humans.
    Gasperíková D, Tribble ND, Staník J, Hucková M, Misovicová N, van de Bunt M, Valentínová L, Barrow BA, Barák L, Dobránsky R, Bereczková E, Michálek J, Wicks K, Colclough K, Knight JC, Ellard S, Klimes I, Gloyn AL.
    Diabetes; 2009 Aug 11; 58(8):1929-35. PubMed ID: 19411616
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  • 33. Intima-media thickness and endothelial dysfunction in GCK and HNF1A-MODY patients.
    Szopa M, Osmenda G, Wilk G, Matejko B, Skupien J, Zapala B, Młynarski W, Guzik T, Malecki MT.
    Eur J Endocrinol; 2015 Mar 11; 172(3):277-83. PubMed ID: 25501962
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  • 34. Prevalence, characteristics and clinical diagnosis of maturity onset diabetes of the young due to mutations in HNF1A, HNF4A, and glucokinase: results from the SEARCH for Diabetes in Youth.
    Pihoker C, Gilliam LK, Ellard S, Dabelea D, Davis C, Dolan LM, Greenbaum CJ, Imperatore G, Lawrence JM, Marcovina SM, Mayer-Davis E, Rodriguez BL, Steck AK, Williams DE, Hattersley AT, SEARCH for Diabetes in Youth Study Group.
    J Clin Endocrinol Metab; 2013 Oct 11; 98(10):4055-62. PubMed ID: 23771925
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  • 39. Nine novel mutations in maturity-onset diabetes of the young (MODY) candidate genes in 22 Spanish families.
    Barrio R, Bellanné-Chantelot C, Moreno JC, Morel V, Calle H, Alonso M, Mustieles C.
    J Clin Endocrinol Metab; 2002 Jun 11; 87(6):2532-9. PubMed ID: 12050210
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  • 40. The glucokinase mutation p.T206P is common among MODY patients of Jewish Ashkenazi descent.
    Gozlan Y, Tenenbaum A, Shalitin S, Lebenthal Y, Oron T, Cohen O, Phillip M, Gat-Yablonski G.
    Pediatr Diabetes; 2012 Sep 11; 13(6):e14-21. PubMed ID: 21978167
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