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Journal Abstract Search


881 related items for PubMed ID: 19339605

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  • 2. Spinal injection of TNF-α-activated astrocytes produces persistent pain symptom mechanical allodynia by releasing monocyte chemoattractant protein-1.
    Gao YJ, Zhang L, Ji RR.
    Glia; 2010 Nov 15; 58(15):1871-80. PubMed ID: 20737477
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  • 3. TRAF6 upregulation in spinal astrocytes maintains neuropathic pain by integrating TNF-α and IL-1β signaling.
    Lu Y, Jiang BC, Cao DL, Zhang ZJ, Zhang X, Ji RR, Gao YJ.
    Pain; 2014 Dec 15; 155(12):2618-2629. PubMed ID: 25267210
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  • 7. A peptide c-Jun N-terminal kinase (JNK) inhibitor blocks mechanical allodynia after spinal nerve ligation: respective roles of JNK activation in primary sensory neurons and spinal astrocytes for neuropathic pain development and maintenance.
    Zhuang ZY, Wen YR, Zhang DR, Borsello T, Bonny C, Strichartz GR, Decosterd I, Ji RR.
    J Neurosci; 2006 Mar 29; 26(13):3551-60. PubMed ID: 16571763
    [Abstract] [Full Text] [Related]

  • 8. Contribution of the spinal cord BDNF to the development of neuropathic pain by activation of the NR2B-containing NMDA receptors in rats with spinal nerve ligation.
    Geng SJ, Liao FF, Dang WH, Ding X, Liu XD, Cai J, Han JS, Wan Y, Xing GG.
    Exp Neurol; 2010 Apr 29; 222(2):256-66. PubMed ID: 20079352
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  • 9. Connexin-43 induces chemokine release from spinal cord astrocytes to maintain late-phase neuropathic pain in mice.
    Chen G, Park CK, Xie RG, Berta T, Nedergaard M, Ji RR.
    Brain; 2014 Aug 29; 137(Pt 8):2193-209. PubMed ID: 24919967
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  • 10. Cytokine mechanisms of central sensitization: distinct and overlapping role of interleukin-1beta, interleukin-6, and tumor necrosis factor-alpha in regulating synaptic and neuronal activity in the superficial spinal cord.
    Kawasaki Y, Zhang L, Cheng JK, Ji RR.
    J Neurosci; 2008 May 14; 28(20):5189-94. PubMed ID: 18480275
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  • 11. Epigenetic transcriptional activation of monocyte chemotactic protein 3 contributes to long-lasting neuropathic pain.
    Imai S, Ikegami D, Yamashita A, Shimizu T, Narita M, Niikura K, Furuya M, Kobayashi Y, Miyashita K, Okutsu D, Kato A, Nakamura A, Araki A, Omi K, Nakamura M, James Okano H, Okano H, Ando T, Takeshima H, Ushijima T, Kuzumaki N, Suzuki T, Narita M.
    Brain; 2013 Mar 14; 136(Pt 3):828-43. PubMed ID: 23364351
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  • 12. Minocycline, a microglial inhibitor, blocks spinal CCL2-induced heat hyperalgesia and augmentation of glutamatergic transmission in substantia gelatinosa neurons.
    Huang CY, Chen YL, Li AH, Lu JC, Wang HL.
    J Neuroinflammation; 2014 Jan 10; 11():7. PubMed ID: 24405660
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  • 13. Neuronal NR2B-containing NMDA receptor mediates spinal astrocytic c-Jun N-terminal kinase activation in a rat model of neuropathic pain.
    Wang W, Mei XP, Wei YY, Zhang MM, Zhang T, Wang W, Xu LX, Wu SX, Li YQ.
    Brain Behav Immun; 2011 Oct 10; 25(7):1355-66. PubMed ID: 21496481
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  • 14. Promoted Interaction of C/EBPα with Demethylated Cxcr3 Gene Promoter Contributes to Neuropathic Pain in Mice.
    Jiang BC, He LN, Wu XB, Shi H, Zhang WW, Zhang ZJ, Cao DL, Li CH, Gu J, Gao YJ.
    J Neurosci; 2017 Jan 18; 37(3):685-700. PubMed ID: 28100749
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  • 15. The c-Jun N-terminal kinase 1 (JNK1) in spinal astrocytes is required for the maintenance of bilateral mechanical allodynia under a persistent inflammatory pain condition.
    Gao YJ, Xu ZZ, Liu YC, Wen YR, Decosterd I, Ji RR.
    Pain; 2010 Feb 18; 148(2):309-319. PubMed ID: 20022176
    [Abstract] [Full Text] [Related]

  • 16. Spinal CCL2 Promotes Central Sensitization, Long-Term Potentiation, and Inflammatory Pain via CCR2: Further Insights into Molecular, Synaptic, and Cellular Mechanisms.
    Xie RG, Gao YJ, Park CK, Lu N, Luo C, Wang WT, Wu SX, Ji RR.
    Neurosci Bull; 2018 Feb 18; 34(1):13-21. PubMed ID: 28265898
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  • 17. Role of the spinal cord NR2B-containing NMDA receptors in the development of neuropathic pain.
    Qu XX, Cai J, Li MJ, Chi YN, Liao FF, Liu FY, Wan Y, Han JS, Xing GG.
    Exp Neurol; 2009 Feb 18; 215(2):298-307. PubMed ID: 19046970
    [Abstract] [Full Text] [Related]

  • 18. The analgesic effect of rolipram is associated with the inhibition of the activation of the spinal astrocytic JNK/CCL2 pathway in bone cancer pain.
    Guo CH, Bai L, Wu HH, Yang J, Cai GH, Wang X, Wu SX, Ma W.
    Int J Mol Med; 2016 Nov 18; 38(5):1433-1442. PubMed ID: 28025994
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  • 19. Intrathecal administration of low-dose nociceptin/orphanin FQ induces allodynia via c-Jun N-terminal kinase and monocyte chemoattractant protein-1.
    Kawabata K, Nishimura I, Fujiwara T, Terauchi S, Minami T, Ito S, Okuda-Ashitaka E.
    Eur J Neurosci; 2016 Jun 18; 43(11):1499-508. PubMed ID: 27038234
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  • 20. Spinal astrocytic c-Jun N-terminal kinase (JNK) activation as counteracting mechanism to the amitriptyline analgesic efficacy in painful peripheral neuropathies.
    Sanna MD, Ghelardini C, Galeotti N.
    Eur J Pharmacol; 2017 Mar 05; 798():85-93. PubMed ID: 28119076
    [Abstract] [Full Text] [Related]


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