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416 related items for PubMed ID: 19375359

  • 1. Preservation of mitochondrial integrity and energy metabolism during experimental status epilepticus leads to neuronal apoptotic cell death in the hippocampus of the rat.
    Chuang YC, Lin JW, Chen SD, Lin TK, Liou CW, Lu CH, Chang WN.
    Seizure; 2009 Jul; 18(6):420-8. PubMed ID: 19375359
    [Abstract] [Full Text] [Related]

  • 2. Mitochondrial dysfunction and ultrastructural damage in the hippocampus during kainic acid-induced status epilepticus in the rat.
    Chuang YC, Chang AY, Lin JW, Hsu SP, Chan SH.
    Epilepsia; 2004 Oct; 45(10):1202-9. PubMed ID: 15461674
    [Abstract] [Full Text] [Related]

  • 3. Contribution of nitric oxide, superoxide anion, and peroxynitrite to activation of mitochondrial apoptotic signaling in hippocampal CA3 subfield following experimental temporal lobe status epilepticus.
    Chuang YC, Chen SD, Liou CW, Lin TK, Chang WN, Chan SH, Chang AY.
    Epilepsia; 2009 Apr; 50(4):731-46. PubMed ID: 19178557
    [Abstract] [Full Text] [Related]

  • 4. Upregulation of nitric oxide synthase II contributes to apoptotic cell death in the hippocampal CA3 subfield via a cytochrome c/caspase-3 signaling cascade following induction of experimental temporal lobe status epilepticus in the rat.
    Chuang YC, Chen SD, Lin TK, Liou CW, Chang WN, Chan SH, Chang AY.
    Neuropharmacology; 2007 Apr; 52(5):1263-73. PubMed ID: 17336342
    [Abstract] [Full Text] [Related]

  • 5. Transcriptional upregulation of nitric oxide synthase II by nuclear factor-kappaB promotes apoptotic neuronal cell death in the hippocampus following experimental status epilepticus.
    Chuang YC, Chen SD, Lin TK, Chang WN, Lu CH, Liou CW, Chan SH, Chang AY.
    J Neurosci Res; 2010 Jul; 88(9):1898-907. PubMed ID: 20155797
    [Abstract] [Full Text] [Related]

  • 6. Kainic acid dose affects delayed cell death mechanism after status epilepticus.
    Tokuhara D, Sakuma S, Hattori H, Matsuoka O, Yamano T.
    Brain Dev; 2007 Jan; 29(1):2-8. PubMed ID: 16790331
    [Abstract] [Full Text] [Related]

  • 7. Heat shock protein 70 protects against seizure-induced neuronal cell death in the hippocampus following experimental status epilepticus via inhibition of nuclear factor-κB activation-induced nitric oxide synthase II expression.
    Chang CC, Chen SD, Lin TK, Chang WN, Liou CW, Chang AY, Chan SH, Chuang YC.
    Neurobiol Dis; 2014 Feb; 62():241-9. PubMed ID: 24141017
    [Abstract] [Full Text] [Related]

  • 8. Kainic acid-induced seizures produce necrotic, not apoptotic, neurons with internucleosomal DNA cleavage: implications for programmed cell death mechanisms.
    Fujikawa DG, Shinmei SS, Cai B.
    Neuroscience; 2000 Feb; 98(1):41-53. PubMed ID: 10858610
    [Abstract] [Full Text] [Related]

  • 9. Dynamin-Related Protein 1 Promotes Mitochondrial Fission and Contributes to The Hippocampal Neuronal Cell Death Following Experimental Status Epilepticus.
    Chen SD, Zhen YY, Lin JW, Lin TK, Huang CW, Liou CW, Chan SH, Chuang YC.
    CNS Neurosci Ther; 2016 Dec; 22(12):988-999. PubMed ID: 27577016
    [Abstract] [Full Text] [Related]

  • 10. Peroxisome proliferator-activated receptors γ/mitochondrial uncoupling protein 2 signaling protects against seizure-induced neuronal cell death in the hippocampus following experimental status epilepticus.
    Chuang YC, Lin TK, Huang HY, Chang WN, Liou CW, Chen SD, Chang AY, Chan SH.
    J Neuroinflammation; 2012 Jul 31; 9():184. PubMed ID: 22849356
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  • 12. Decreased interaction between FoxO3a and Akt correlates with seizure-induced neuronal death.
    Kim YS, Choi MY, Lee DH, Jeon BT, Roh GS, Kim HJ, Kang SS, Cho GJ, Choi WS.
    Epilepsy Res; 2014 Mar 31; 108(3):367-78. PubMed ID: 24518891
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  • 15. MicroRNA expression profile of the hippocampus in a rat model of temporal lobe epilepsy and miR-34a-targeted neuroprotection against hippocampal neurone cell apoptosis post-status epilepticus.
    Hu K, Xie YY, Zhang C, Ouyang DS, Long HY, Sun DN, Long LL, Feng L, Li Y, Xiao B.
    BMC Neurosci; 2012 Sep 22; 13():115. PubMed ID: 22998082
    [Abstract] [Full Text] [Related]

  • 16. High-frequency stimulation of the hippocampus protects against seizure activity and hippocampal neuronal apoptosis induced by kainic acid administration in macaques.
    Chen N, Gao Y, Yan N, Liu C, Zhang JG, Xing WM, Kong DM, Meng FG.
    Neuroscience; 2014 Jan 03; 256():370-8. PubMed ID: 24200923
    [Abstract] [Full Text] [Related]

  • 17. Hypoxic preconditioning attenuated in kainic acid-induced neurotoxicity in rat hippocampus.
    Chang AY, Wang CH, Chiu TH, Chi JW, Chen CF, Ho LT, Lin AM.
    Exp Neurol; 2005 Sep 03; 195(1):40-8. PubMed ID: 15950222
    [Abstract] [Full Text] [Related]

  • 18. Proteomic identification of hippocampal proteins vulnerable to oxidative stress in excitotoxin-induced acute neuronal injury.
    Furukawa A, Kawamoto Y, Chiba Y, Takei S, Hasegawa-Ishii S, Kawamura N, Yoshikawa K, Hosokawa M, Oikawa S, Kato M, Shimada A.
    Neurobiol Dis; 2011 Sep 03; 43(3):706-14. PubMed ID: 21669285
    [Abstract] [Full Text] [Related]

  • 19. Caffeic acid phenethyl ester prevents apoptotic cell death in the developing rat brain after pentylenetetrazole-induced status epilepticus.
    Yiş U, Topçu Y, Özbal S, Tuğyan K, Bayram E, Karakaya P, Yilmaz O, Kurul SH.
    Epilepsy Behav; 2013 Nov 03; 29(2):275-80. PubMed ID: 24012504
    [Abstract] [Full Text] [Related]

  • 20. Perinatal seizures preferentially protect CA1 neurons from seizure-induced damage in prepubescent rats.
    Liu H, Friedman LK, Kaur J.
    Seizure; 2006 Jan 03; 15(1):1-16. PubMed ID: 16309925
    [Abstract] [Full Text] [Related]


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