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Journal Abstract Search

232 related items for PubMed ID: 19386911

  • 1. Cross-seeding fibrillation of Q/N-rich proteins offers new pathomechanism of polyglutamine diseases.
    Furukawa Y, Kaneko K, Matsumoto G, Kurosawa M, Nukina N.
    J Neurosci; 2009 Apr 22; 29(16):5153-62. PubMed ID: 19386911
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  • 2. RNA-binding protein TLS is a major nuclear aggregate-interacting protein in huntingtin exon 1 with expanded polyglutamine-expressing cells.
    Doi H, Okamura K, Bauer PO, Furukawa Y, Shimizu H, Kurosawa M, Machida Y, Miyazaki H, Mitsui K, Kuroiwa Y, Nukina N.
    J Biol Chem; 2008 Mar 07; 283(10):6489-500. PubMed ID: 18167354
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  • 3. Calmodulin regulates transglutaminase 2 cross-linking of huntingtin.
    Zainelli GM, Ross CA, Troncoso JC, Fitzgerald JK, Muma NA.
    J Neurosci; 2004 Feb 25; 24(8):1954-61. PubMed ID: 14985437
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  • 4. Adenovirus vector-based in vitro neuronal cell model for Huntington's disease with human disease-like differential aggregation and degeneration.
    Dong X, Zong S, Witting A, Lindenberg KS, Kochanek S, Huang B.
    J Gene Med; 2012 Jul 25; 14(7):468-81. PubMed ID: 22700462
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  • 5. Amyloid formation by mutant huntingtin: threshold, progressivity and recruitment of normal polyglutamine proteins.
    Huang CC, Faber PW, Persichetti F, Mittal V, Vonsattel JP, MacDonald ME, Gusella JF.
    Somat Cell Mol Genet; 1998 Jul 25; 24(4):217-33. PubMed ID: 10410676
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  • 6. A structure-based analysis of huntingtin mutant polyglutamine aggregation and toxicity: evidence for a compact beta-sheet structure.
    Poirier MA, Jiang H, Ross CA.
    Hum Mol Genet; 2005 Mar 15; 14(6):765-74. PubMed ID: 15689354
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  • 7. Selection of behaviors and segmental coordination during larval locomotion is disrupted by nuclear polyglutamine inclusions in a new Drosophila Huntington's disease-like model.
    Nishimura Y, Yalgin C, Akimoto S, Doumanis J, Sasajima R, Nukina N, Miyakawa H, Moore AW, Morimoto T.
    J Neurogenet; 2010 Dec 15; 24(4):194-206. PubMed ID: 21087194
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  • 9. Huntingtin affinity for partners is not changed by polyglutamine length: aggregation itself triggers aberrant interactions.
    Davranche A, Aviolat H, Zeder-Lutz G, Busso D, Altschuh D, Trottier Y, Klein FA.
    Hum Mol Genet; 2011 Jul 15; 20(14):2795-806. PubMed ID: 21518730
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  • 14. Deletion of the huntingtin polyglutamine stretch enhances neuronal autophagy and longevity in mice.
    Zheng S, Clabough EB, Sarkar S, Futter M, Rubinsztein DC, Zeitlin SO.
    PLoS Genet; 2010 Feb 05; 6(2):e1000838. PubMed ID: 20140187
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  • 15. Full length mutant huntingtin is required for altered Ca2+ signaling and apoptosis of striatal neurons in the YAC mouse model of Huntington's disease.
    Zhang H, Li Q, Graham RK, Slow E, Hayden MR, Bezprozvanny I.
    Neurobiol Dis; 2008 Jul 05; 31(1):80-8. PubMed ID: 18502655
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  • 16. Effects of overexpression of huntingtin proteins on mitochondrial integrity.
    Wang H, Lim PJ, Karbowski M, Monteiro MJ.
    Hum Mol Genet; 2009 Feb 15; 18(4):737-52. PubMed ID: 19039036
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  • 17. 14-3-3zeta is indispensable for aggregate formation of polyglutamine-expanded huntingtin protein.
    Omi K, Hachiya NS, Tanaka M, Tokunaga K, Kaneko K.
    Neurosci Lett; 2008 Jan 24; 431(1):45-50. PubMed ID: 18078716
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  • 18. Huntingtin inclusions do not deplete polyglutamine-containing transcription factors in HD mice.
    Yu ZX, Li SH, Nguyen HP, Li XJ.
    Hum Mol Genet; 2002 Apr 15; 11(8):905-14. PubMed ID: 11971872
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  • 19. Clinico-pathological rescue of a model mouse of Huntington's disease by siRNA.
    Wang YL, Liu W, Wada E, Murata M, Wada K, Kanazawa I.
    Neurosci Res; 2005 Nov 15; 53(3):241-9. PubMed ID: 16095740
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  • 20. The Gln-Ala repeat transcriptional activator CA150 interacts with huntingtin: neuropathologic and genetic evidence for a role in Huntington's disease pathogenesis.
    Holbert S, Denghien I, Kiechle T, Rosenblatt A, Wellington C, Hayden MR, Margolis RL, Ross CA, Dausset J, Ferrante RJ, Néri C.
    Proc Natl Acad Sci U S A; 2001 Feb 13; 98(4):1811-6. PubMed ID: 11172033
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