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Journal Abstract Search

300 related items for PubMed ID: 19387681

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  • 5. Eliminating Nox2 reactive oxygen species production protects dystrophic skeletal muscle from pathological calcium influx assessed in vivo by manganese-enhanced magnetic resonance imaging.
    Loehr JA, Stinnett GR, Hernández-Rivera M, Roten WT, Wilson LJ, Pautler RG, Rodney GG.
    J Physiol; 2016 Nov 01; 594(21):6395-6405. PubMed ID: 27555555
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  • 8. The role of reactive oxygen species in the hearts of dystrophin-deficient mdx mice.
    Williams IA, Allen DG.
    Am J Physiol Heart Circ Physiol; 2007 Sep 01; 293(3):H1969-77. PubMed ID: 17573457
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  • 11. NADPH oxidase-2 inhibition restores contractility and intracellular calcium handling and reduces arrhythmogenicity in dystrophic cardiomyopathy.
    Gonzalez DR, Treuer AV, Lamirault G, Mayo V, Cao Y, Dulce RA, Hare JM.
    Am J Physiol Heart Circ Physiol; 2014 Sep 01; 307(5):H710-21. PubMed ID: 25015966
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  • 12. Mitochondrial redox state and Ca2+ sparks in permeabilized mammalian skeletal muscle.
    Isaeva EV, Shkryl VM, Shirokova N.
    J Physiol; 2005 Jun 15; 565(Pt 3):855-72. PubMed ID: 15845582
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  • 17. Increased resting intracellular calcium modulates NF-κB-dependent inducible nitric-oxide synthase gene expression in dystrophic mdx skeletal myotubes.
    Altamirano F, López JR, Henríquez C, Molinski T, Allen PD, Jaimovich E.
    J Biol Chem; 2012 Jun 15; 287(25):20876-87. PubMed ID: 22549782
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  • 18. Skeletal muscle NADPH oxidase is increased and triggers stretch-induced damage in the mdx mouse.
    Whitehead NP, Yeung EW, Froehner SC, Allen DG.
    PLoS One; 2010 Dec 20; 5(12):e15354. PubMed ID: 21187957
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  • 20. IP3 receptor blockade restores autophagy and mitochondrial function in skeletal muscle fibers of dystrophic mice.
    Valladares D, Utreras-Mendoza Y, Campos C, Morales C, Diaz-Vegas A, Contreras-Ferrat A, Westermeier F, Jaimovich E, Marchi S, Pinton P, Lavandero S.
    Biochim Biophys Acta Mol Basis Dis; 2018 Nov 20; 1864(11):3685-3695. PubMed ID: 30251688
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