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302 related items for PubMed ID: 19451205
1. New roles of calsequestrin and triadin in cardiac muscle. Knollmann BC. J Physiol; 2009 Jul 01; 587(Pt 13):3081-7. PubMed ID: 19451205 [Abstract] [Full Text] [Related]
2. Triadin overexpression stimulates excitation-contraction coupling and increases predisposition to cellular arrhythmia in cardiac myocytes. Terentyev D, Cala SE, Houle TD, Viatchenko-Karpinski S, Gyorke I, Terentyeva R, Williams SC, Gyorke S. Circ Res; 2005 Apr 01; 96(6):651-8. PubMed ID: 15731460 [Abstract] [Full Text] [Related]
3. Ablation of triadin causes loss of cardiac Ca2+ release units, impaired excitation-contraction coupling, and cardiac arrhythmias. Chopra N, Yang T, Asghari P, Moore ED, Huke S, Akin B, Cattolica RA, Perez CF, Hlaing T, Knollmann-Ritschel BE, Jones LR, Pessah IN, Allen PD, Franzini-Armstrong C, Knollmann BC. Proc Natl Acad Sci U S A; 2009 May 05; 106(18):7636-41. PubMed ID: 19383796 [Abstract] [Full Text] [Related]
4. Modulation of ryanodine receptor by luminal calcium and accessory proteins in health and cardiac disease. Györke S, Terentyev D. Cardiovasc Res; 2008 Jan 15; 77(2):245-55. PubMed ID: 18006456 [Abstract] [Full Text] [Related]
5. Triadins modulate intracellular Ca(2+) homeostasis but are not essential for excitation-contraction coupling in skeletal muscle. Shen X, Franzini-Armstrong C, Lopez JR, Jones LR, Kobayashi YM, Wang Y, Kerrick WG, Caswell AH, Potter JD, Miller T, Allen PD, Perez CF. J Biol Chem; 2007 Dec 28; 282(52):37864-74. PubMed ID: 17981799 [Abstract] [Full Text] [Related]
6. Dual role of junctin in the regulation of ryanodine receptors and calcium release in cardiac ventricular myocytes. Altschafl BA, Arvanitis DA, Fuentes O, Yuan Q, Kranias EG, Valdivia HH. J Physiol; 2011 Dec 15; 589(Pt 24):6063-80. PubMed ID: 22025663 [Abstract] [Full Text] [Related]
7. Altered function in atrium of transgenic mice overexpressing triadin 1. Kirchhefer U, Baba HA, Kobayashi YM, Jones LR, Schmitz W, Neumann J. Am J Physiol Heart Circ Physiol; 2002 Oct 15; 283(4):H1334-43. PubMed ID: 12234783 [Abstract] [Full Text] [Related]
8. Junctin and the histidine-rich Ca2+ binding protein: potential roles in heart failure and arrhythmogenesis. Pritchard TJ, Kranias EG. J Physiol; 2009 Jul 01; 587(Pt 13):3125-33. PubMed ID: 19403607 [Abstract] [Full Text] [Related]
9. Regulatory roles of junctin in sarcoplasmic reticulum calcium cycling and myocardial function. Fan GC, Yuan Q, Kranias EG. Trends Cardiovasc Med; 2008 Jan 01; 18(1):1-5. PubMed ID: 18206802 [Abstract] [Full Text] [Related]
10. Sarcoplasmic reticulum Ca2+ release in neonatal rat cardiac myocytes. Gergs U, Kirchhefer U, Buskase J, Kiele-Dunsche K, Buchwalow IB, Jones LR, Schmitz W, Traub O, Neumann J. J Mol Cell Cardiol; 2011 Nov 01; 51(5):682-8. PubMed ID: 21871897 [Abstract] [Full Text] [Related]
11. Modulation of SR Ca2+ release by the triadin-to-calsequestrin ratio in ventricular myocytes. Kučerová D, Baba HA, Bokník P, Fabritz L, Heinick A, Mát'uš M, Müller FU, Neumann J, Schmitz W, Kirchhefer U. Am J Physiol Heart Circ Physiol; 2012 May 15; 302(10):H2008-17. PubMed ID: 22427521 [Abstract] [Full Text] [Related]
12. Protein protein interactions between triadin and calsequestrin are involved in modulation of sarcoplasmic reticulum calcium release in cardiac myocytes. Terentyev D, Viatchenko-Karpinski S, Vedamoorthyrao S, Oduru S, Györke I, Williams SC, Györke S. J Physiol; 2007 Aug 15; 583(Pt 1):71-80. PubMed ID: 17569730 [Abstract] [Full Text] [Related]
13. Sarcoplasmic reticulum calcium overloading in junctin deficiency enhances cardiac contractility but increases ventricular automaticity. Yuan Q, Fan GC, Dong M, Altschafl B, Diwan A, Ren X, Hahn HH, Zhao W, Waggoner JR, Jones LR, Jones WK, Bers DM, Dorn GW, Wang HS, Valdivia HH, Chu G, Kranias EG. Circulation; 2007 Jan 23; 115(3):300-9. PubMed ID: 17224479 [Abstract] [Full Text] [Related]
14. Complex formation between junctin, triadin, calsequestrin, and the ryanodine receptor. Proteins of the cardiac junctional sarcoplasmic reticulum membrane. Zhang L, Kelley J, Schmeisser G, Kobayashi YM, Jones LR. J Biol Chem; 1997 Sep 12; 272(37):23389-97. PubMed ID: 9287354 [Abstract] [Full Text] [Related]
15. Cardiac hypertrophy and impaired relaxation in transgenic mice overexpressing triadin 1. Kirchhefer U, Neumann J, Baba HA, Begrow F, Kobayashi YM, Reinke U, Schmitz W, Jones LR. J Biol Chem; 2001 Feb 09; 276(6):4142-9. PubMed ID: 11069905 [Abstract] [Full Text] [Related]
16. Triadin/Junctin double null mouse reveals a differential role for Triadin and Junctin in anchoring CASQ to the jSR and regulating Ca(2+) homeostasis. Boncompagni S, Thomas M, Lopez JR, Allen PD, Yuan Q, Kranias EG, Franzini-Armstrong C, Perez CF. PLoS One; 2012 Feb 09; 7(7):e39962. PubMed ID: 22768324 [Abstract] [Full Text] [Related]
17. Modest reductions of cardiac calsequestrin increase sarcoplasmic reticulum Ca2+ leak independent of luminal Ca2+ and trigger ventricular arrhythmias in mice. Chopra N, Kannankeril PJ, Yang T, Hlaing T, Holinstat I, Ettensohn K, Pfeifer K, Akin B, Jones LR, Franzini-Armstrong C, Knollmann BC. Circ Res; 2007 Sep 14; 101(6):617-26. PubMed ID: 17656677 [Abstract] [Full Text] [Related]
18. Overexpression of junctin causes adaptive changes in cardiac myocyte Ca(2+) signaling. Kirchhefer U, Hanske G, Jones LR, Justus I, Kaestner L, Lipp P, Schmitz W, Neumann J. Cell Calcium; 2006 Feb 14; 39(2):131-42. PubMed ID: 16289269 [Abstract] [Full Text] [Related]
19. The role of calsequestrin, triadin, and junctin in conferring cardiac ryanodine receptor responsiveness to luminal calcium. Györke I, Hester N, Jones LR, Györke S. Biophys J; 2004 Apr 14; 86(4):2121-8. PubMed ID: 15041652 [Abstract] [Full Text] [Related]
20. Triadin binding to the C-terminal luminal loop of the ryanodine receptor is important for skeletal muscle excitation contraction coupling. Goonasekera SA, Beard NA, Groom L, Kimura T, Lyfenko AD, Rosenfeld A, Marty I, Dulhunty AF, Dirksen RT. J Gen Physiol; 2007 Oct 14; 130(4):365-78. PubMed ID: 17846166 [Abstract] [Full Text] [Related] Page: [Next] [New Search]