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178 related items for PubMed ID: 19491250
1. KCNQ1 is the luminal K+ recycling channel during stimulation of gastric acid secretion. Song P, Groos S, Riederer B, Feng Z, Krabbenhöft A, Smolka A, Seidler U. J Physiol; 2009 Aug 01; 587(Pt 15):3955-65. PubMed ID: 19491250 [Abstract] [Full Text] [Related]
2. Unravelling a role for KCNQ1 in K+ recycling and gastric acid secretion. Rieg T, Dominguez JA. J Physiol; 2009 Sep 01; 587(Pt 17):4149-50. PubMed ID: 19720854 [No Abstract] [Full Text] [Related]
5. Independent trafficking of the KCNQ1 K+ channel and H+-K+-ATPase in gastric parietal cells from mice. Nguyen N, Kozer-Gorevich N, Gliddon BL, Smolka AJ, Clayton AH, Gleeson PA, van Driel IR. Am J Physiol Gastrointest Liver Physiol; 2013 Jan 15; 304(2):G157-66. PubMed ID: 23154976 [Abstract] [Full Text] [Related]
7. Ca2+ activated K+ channel Kca3.1 as a determinant of gastric acid secretion. Rotte A, Pasham V, Mack AF, Bhandaru M, Qadri SM, Eichenmüller M, Ruth P, Lang F. Cell Physiol Biochem; 2011 Jan 15; 27(5):597-604. PubMed ID: 21691077 [Abstract] [Full Text] [Related]
18. Deletion of the chloride transporter Slc26a9 causes loss of tubulovesicles in parietal cells and impairs acid secretion in the stomach. Xu J, Song P, Miller ML, Borgese F, Barone S, Riederer B, Wang Z, Alper SL, Forte JG, Shull GE, Ehrenfeld J, Seidler U, Soleimani M. Proc Natl Acad Sci U S A; 2008 Nov 18; 105(46):17955-60. PubMed ID: 19004773 [Abstract] [Full Text] [Related]