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136 related items for PubMed ID: 1953684
1. p52(PAI-1) and actin expression in butyrate-induced flat revertants of v-ras-transformed rat kidney cells. Higgins PJ, Ryan MP. Biochem J; 1991 Nov 01; 279 ( Pt 3)(Pt 3):883-90. PubMed ID: 1953684 [Abstract] [Full Text] [Related]
2. P52PAI-1 gene expression in butyrate-induced flat revertants of v-ras-transformed rat kidney cells: mechanism of induction and involvement in the morphological response. Higgins PJ, Ryan MP, Jelley DM. Biochem J; 1997 Jan 15; 321 ( Pt 2)(Pt 2):431-7. PubMed ID: 9020877 [Abstract] [Full Text] [Related]
3. Identification of the 52 kDa cytoskeletal-like protein of cytochalasin D-stimulated normal rat kidney (NRK/CD) cells as substrate-associated glycoprotein p52 [plasminogen-activator inhibitor type-1 (PAI-1)]. Expression of p52 (PAI-1) in NRK/CD cells is regulated at the level of mRNA abundance. Higgins PJ, Ryan MP. Biochem J; 1992 Jun 01; 284 ( Pt 2)(Pt 2):433-9. PubMed ID: 1599429 [Abstract] [Full Text] [Related]
4. Cell-shape-dependent modulation of p52(PAI-1) gene expression involves a secondary response pathway. Higgins PJ, Staiano-Coico L, Ryan MP. Biochem J; 1995 Mar 01; 306 ( Pt 2)(Pt 2):497-504. PubMed ID: 7887903 [Abstract] [Full Text] [Related]
9. Cytoarchitecture of ras oncogene-expressing tumor cells: butyrate modulation of substrate adhesion, cytoskeletal actin content and subcellular microfilament distribution. Higgins PJ, Ryan MP. Int J Biochem; 1989 Mar 01; 21(10):1143-51. PubMed ID: 2684700 [Abstract] [Full Text] [Related]
10. p52 induction by cytochalasin D in rat kidney fibroblasts: homologies between p52 and plasminogen activator inhibitor type-1. Higgins PJ, Ryan MP, Zeheb R, Gelehrter TD, Chaudhari P. J Cell Physiol; 1990 May 01; 143(2):321-9. PubMed ID: 2332454 [Abstract] [Full Text] [Related]
11. Induced expression of p52(PAI-1) in normal rat kidney cells by the microfilament-disrupting agent cytochalasin D. Higgins PJ, Ryan MP, Providence KM. J Cell Physiol; 1994 Apr 01; 159(1):187-95. PubMed ID: 8138587 [Abstract] [Full Text] [Related]
12. A specific protein, p92, detected in flat revertants derived from NIH/3T3 transformed by human activated c-Ha-ras oncogene. Fujita H, Suzuki H, Kuzumaki N, Müllauer L, Ogiso Y, Oda A, Ebisawa K, Sakurai T, Nonomura Y, Kijimoto-Ochiai S. Exp Cell Res; 1990 Jan 01; 186(1):115-21. PubMed ID: 2153549 [Abstract] [Full Text] [Related]
15. [A study on alterations of gene expression in a flat revertant R1 from ras-oncogene transformed NIH/3T3 cells]. Müllauer L. Hokkaido Igaku Zasshi; 1993 Sep 01; 68(5):705-16. PubMed ID: 7693563 [Abstract] [Full Text] [Related]
19. [Identification of a specific protein in flat revertant cell lines derived from ras oncogene-transformed cells]. Fujita H. Hokkaido Igaku Zasshi; 1990 Mar 01; 65(2):210-20. PubMed ID: 2194920 [Abstract] [Full Text] [Related]
20. Control of p52(PAI-1) gene expression in normal and transformed rat kidney cells: relationship between p52(PAI-1) induction and actin cytoarchitecture. Ryan MP, Higgins PJ. Adv Exp Med Biol; 1994 Mar 01; 358():215-30. PubMed ID: 7801807 [No Abstract] [Full Text] [Related] Page: [Next] [New Search]