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467 related items for PubMed ID: 19607913
1. Is multiple sclerosis a mitochondrial disease? Mao P, Reddy PH. Biochim Biophys Acta; 2010 Jan; 1802(1):66-79. PubMed ID: 19607913 [Abstract] [Full Text] [Related]
2. Cyclophilin D inactivation protects axons in experimental autoimmune encephalomyelitis, an animal model of multiple sclerosis. Forte M, Gold BG, Marracci G, Chaudhary P, Basso E, Johnsen D, Yu X, Fowlkes J, Rahder M, Stem K, Bernardi P, Bourdette D. Proc Natl Acad Sci U S A; 2007 May 01; 104(18):7558-63. PubMed ID: 17463082 [Abstract] [Full Text] [Related]
3. Mitochondrial dysfunction plays a key role in progressive axonal loss in Multiple Sclerosis. Andrews HE, Nichols PP, Bates D, Turnbull DM. Med Hypotheses; 2005 May 01; 64(4):669-77. PubMed ID: 15694681 [Abstract] [Full Text] [Related]
13. Axonal degeneration in multiple sclerosis: the mitochondrial hypothesis. Su KG, Banker G, Bourdette D, Forte M. Curr Neurol Neurosci Rep; 2009 Sep 01; 9(5):411-7. PubMed ID: 19664372 [Abstract] [Full Text] [Related]
14. Axon loss in the spinal cord determines permanent neurological disability in an animal model of multiple sclerosis. Wujek JR, Bjartmar C, Richer E, Ransohoff RM, Yu M, Tuohy VK, Trapp BD. J Neuropathol Exp Neurol; 2002 Jan 01; 61(1):23-32. PubMed ID: 11829341 [Abstract] [Full Text] [Related]
16. [Neuronal injury in multiple sclerosis]. Correale J, Meli F, Ysrraelit C. Medicina (B Aires); 2006 Jan 01; 66(5):472-85. PubMed ID: 17137182 [Abstract] [Full Text] [Related]
17. Insights in pathogenesis of multiple sclerosis: nitric oxide may induce mitochondrial dysfunction of oligodendrocytes. Lan M, Tang X, Zhang J, Yao Z. Rev Neurosci; 2018 Jan 26; 29(1):39-53. PubMed ID: 28822986 [Abstract] [Full Text] [Related]