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Journal Abstract Search

228 related items for PubMed ID: 19635471

  • 1. Disruption of glutamate receptors at Shank-postsynaptic platform in Alzheimer's disease.
    Gong Y, Lippa CF, Zhu J, Lin Q, Rosso AL.
    Brain Res; 2009 Oct 06; 1292():191-8. PubMed ID: 19635471
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  • 3. Soluble beta-amyloid1-40 induces NMDA-dependent degradation of postsynaptic density-95 at glutamatergic synapses.
    Roselli F, Tirard M, Lu J, Hutzler P, Lamberti P, Livrea P, Morabito M, Almeida OF.
    J Neurosci; 2005 Nov 30; 25(48):11061-70. PubMed ID: 16319306
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  • 4. Pathological Role of Peptidyl-Prolyl Isomerase Pin1 in the Disruption of Synaptic Plasticity in Alzheimer's Disease.
    Xu L, Ren Z, Chow FE, Tsai R, Liu T, Rizzolio F, Boffo S, Xu Y, Huang S, Lippa CF, Gong Y.
    Neural Plast; 2017 Nov 30; 2017():3270725. PubMed ID: 28458925
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  • 5. Coupling of mGluR/Homer and PSD-95 complexes by the Shank family of postsynaptic density proteins.
    Tu JC, Xiao B, Naisbitt S, Yuan JP, Petralia RS, Brakeman P, Doan A, Aakalu VK, Lanahan AA, Sheng M, Worley PF.
    Neuron; 1999 Jul 30; 23(3):583-92. PubMed ID: 10433269
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  • 6. Pathological reorganization of NMDA receptors subunits and postsynaptic protein PSD-95 distribution in Alzheimer's disease.
    Leuba G, Vernay A, Kraftsik R, Tardif E, Riederer BM, Savioz A.
    Curr Alzheimer Res; 2014 Jan 30; 11(1):86-96. PubMed ID: 24156266
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  • 7. Importance of Shank3 protein in regulating metabotropic glutamate receptor 5 (mGluR5) expression and signaling at synapses.
    Verpelli C, Dvoretskova E, Vicidomini C, Rossi F, Chiappalone M, Schoen M, Di Stefano B, Mantegazza R, Broccoli V, Böckers TM, Dityatev A, Sala C.
    J Biol Chem; 2011 Oct 07; 286(40):34839-50. PubMed ID: 21795692
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  • 8. GluN2B-Containing NMDA Receptors Regulate AMPA Receptor Traffic through Anchoring of the Synaptic Proteasome.
    Ferreira JS, Schmidt J, Rio P, Águas R, Rooyakkers A, Li KW, Smit AB, Craig AM, Carvalho AL.
    J Neurosci; 2015 Jun 03; 35(22):8462-79. PubMed ID: 26041915
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  • 9. Disassembly of shank and homer synaptic clusters is driven by soluble beta-amyloid(1-40) through divergent NMDAR-dependent signalling pathways.
    Roselli F, Hutzler P, Wegerich Y, Livrea P, Almeida OF.
    PLoS One; 2009 Jun 23; 4(6):e6011. PubMed ID: 19547699
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  • 11. Kainate-induced seizures alter protein composition and N-methyl-D-aspartate receptor function of rat forebrain postsynaptic densities.
    Wyneken U, Smalla KH, Marengo JJ, Soto D, de la Cerda A, Tischmeyer W, Grimm R, Boeckers TM, Wolf G, Orrego F, Gundelfinger ED.
    Neuroscience; 2001 Jun 23; 102(1):65-74. PubMed ID: 11226670
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  • 12. Lysosomal dysfunction produces distinct alterations in synaptic alpha-amino-3-hydroxy-5-methylisoxazolepropionic acid and N-methyl-D-aspartate receptor currents in hippocampus.
    Kanju PM, Parameshwaran K, Vaithianathan T, Sims CM, Huggins K, Bendiske J, Ryzhikov S, Bahr BA, Suppiramaniam V.
    J Neuropathol Exp Neurol; 2007 Sep 23; 66(9):779-88. PubMed ID: 17805008
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  • 19. The postsynaptic scaffold proteins ProSAP1/Shank2 and Homer1 are associated with glutamate receptor complexes at rat retinal synapses.
    Brandstätter JH, Dick O, Boeckers TM.
    J Comp Neurol; 2004 Aug 02; 475(4):551-63. PubMed ID: 15236236
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  • 20. Activity level controls postsynaptic composition and signaling via the ubiquitin-proteasome system.
    Ehlers MD.
    Nat Neurosci; 2003 Mar 02; 6(3):231-42. PubMed ID: 12577062
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