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251 related items for PubMed ID: 19660865
1. Role of substance P signaling in enhanced nociceptive sensitization and local cytokine production after incision. Sahbaie P, Shi X, Guo TZ, Qiao Y, Yeomans DC, Kingery WS, Clark DJ. Pain; 2009 Oct; 145(3):341-349. PubMed ID: 19660865 [Abstract] [Full Text] [Related]
2. Preprotachykinin-A gene disruption attenuates nociceptive sensitivity after opioid administration and incision by peripheral and spinal mechanisms in mice. Sahbaie P, Shi X, Li X, Liang D, Guo TZ, Qiao Y, Yeomans DC, Kingery WS, David Clark J. J Pain; 2012 Oct; 13(10):997-1007. PubMed ID: 23031399 [Abstract] [Full Text] [Related]
8. Chronic morphine administration enhances nociceptive sensitivity and local cytokine production after incision. Liang D, Shi X, Qiao Y, Angst MS, Yeomans DC, Clark JD. Mol Pain; 2008 Feb 22; 4():7. PubMed ID: 18294378 [Abstract] [Full Text] [Related]
10. Acute versus chronic phase mechanisms in a rat model of CRPS. Wei T, Guo TZ, Li WW, Kingery WS, Clark JD. J Neuroinflammation; 2016 Jan 19; 13():14. PubMed ID: 26785976 [Abstract] [Full Text] [Related]
18. Keratinocyte expression of inflammatory mediators plays a crucial role in substance P-induced acute and chronic pain. Wei T, Guo TZ, Li WW, Hou S, Kingery WS, Clark JD. J Neuroinflammation; 2012 Jul 23; 9():181. PubMed ID: 22824437 [Abstract] [Full Text] [Related]
19. Neuropeptides contribute to peripheral nociceptive sensitization by regulating interleukin-1β production in keratinocytes. Shi X, Wang L, Li X, Sahbaie P, Kingery WS, Clark JD. Anesth Analg; 2011 Jul 23; 113(1):175-83. PubMed ID: 21596883 [Abstract] [Full Text] [Related]
20. Hemokinin-1 is an important mediator of pain in mouse models of neuropathic and inflammatory mechanisms. Hunyady Á, Hajna Z, Gubányi T, Scheich B, Kemény Á, Gaszner B, Borbély É, Helyes Z. Brain Res Bull; 2019 Apr 23; 147():165-173. PubMed ID: 30664920 [Abstract] [Full Text] [Related] Page: [Next] [New Search]