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Journal Abstract Search

135 related items for PubMed ID: 19733235

  • 1. NADPH oxidases are responsible for the failure of nitric oxide to inhibit migration of smooth muscle cells exposed to high glucose.
    Tong X, Schröder K.
    Free Radic Biol Med; 2009 Dec 01; 47(11):1578-83. PubMed ID: 19733235
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  • 2. Protein disulfide isomerase is required for platelet-derived growth factor-induced vascular smooth muscle cell migration, Nox1 NADPH oxidase expression, and RhoGTPase activation.
    Pescatore LA, Bonatto D, Forti FL, Sadok A, Kovacic H, Laurindo FR.
    J Biol Chem; 2012 Aug 24; 287(35):29290-300. PubMed ID: 22773830
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  • 7. Nox2 mediates high fat high sucrose diet-induced nitric oxide dysfunction and inflammation in aortic smooth muscle cells.
    Qin Z, Hou X, Weisbrod RM, Seta F, Cohen RA, Tong X.
    J Mol Cell Cardiol; 2014 Jul 24; 72():56-63. PubMed ID: 24631774
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  • 10. Effects of angiotensin II infusion on the expression and function of NAD(P)H oxidase and components of nitric oxide/cGMP signaling.
    Mollnau H, Wendt M, Szöcs K, Lassègue B, Schulz E, Oelze M, Li H, Bodenschatz M, August M, Kleschyov AL, Tsilimingas N, Walter U, Förstermann U, Meinertz T, Griendling K, Münzel T.
    Circ Res; 2002 Mar 08; 90(4):E58-65. PubMed ID: 11884382
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  • 17. Endothelin (ET)-1 inhibits nicotinamide adenine dinucleotide phosphate oxidase activity in human abdominal aortic endothelial cells: a novel function of ETB1 receptors.
    Dammanahalli JK, Sun Z.
    Endocrinology; 2008 Oct 08; 149(10):4979-87. PubMed ID: 18535108
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  • 18. Insulin-like growth factor-I induces reactive oxygen species production and cell migration through Nox4 and Rac1 in vascular smooth muscle cells.
    Meng D, Lv DD, Fang J.
    Cardiovasc Res; 2008 Nov 01; 80(2):299-308. PubMed ID: 18567639
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  • 19. Nox4-derived reactive oxygen species mediate cardiomyocyte injury in early type 1 diabetes.
    Maalouf RM, Eid AA, Gorin YC, Block K, Escobar GP, Bailey S, Abboud HE.
    Am J Physiol Cell Physiol; 2012 Feb 01; 302(3):C597-604. PubMed ID: 22031600
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