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Journal Abstract Search


214 related items for PubMed ID: 19744465

  • 1. Energetic performance is improved by specific activation of K+ fluxes through K(Ca) channels in heart mitochondria.
    Aon MA, Cortassa S, Wei AC, Grunnet M, O'Rourke B.
    Biochim Biophys Acta; 2010 Jan; 1797(1):71-80. PubMed ID: 19744465
    [Abstract] [Full Text] [Related]

  • 2. Pharmacological and physiological stimuli do not promote Ca(2+)-sensitive K+ channel activity in isolated heart mitochondria.
    Cancherini DV, Queliconi BB, Kowaltowski AJ.
    Cardiovasc Res; 2007 Mar 01; 73(4):720-8. PubMed ID: 17208207
    [Abstract] [Full Text] [Related]

  • 3. Mitochondrial Ca2+-activated K+ channels in cardiac myocytes: a mechanism of the cardioprotective effect and modulation by protein kinase A.
    Sato T, Saito T, Saegusa N, Nakaya H.
    Circulation; 2005 Jan 18; 111(2):198-203. PubMed ID: 15623543
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  • 4. Time and charge/pH-dependent activation of K+ channel-mediated K+ influx and K+/H+ exchange in guinea pig heart isolated mitochondria; role in bioenergetic stability.
    Malas KM, Lambert DS, Heisner JS, Camara AKS, Stowe DF.
    Biochim Biophys Acta Bioenerg; 2022 Nov 01; 1863(8):148908. PubMed ID: 35961396
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  • 5. Determination of the rate of K(+) movement through potassium channels in isolated rat heart and liver mitochondria.
    Bednarczyk P, Barker GD, Halestrap AP.
    Biochim Biophys Acta; 2008 Jun 01; 1777(6):540-8. PubMed ID: 18471430
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  • 6. Mitochondrial Ca2+-induced K+ influx increases respiration and enhances ROS production while maintaining membrane potential.
    Heinen A, Camara AK, Aldakkak M, Rhodes SS, Riess ML, Stowe DF.
    Am J Physiol Cell Physiol; 2007 Jan 01; 292(1):C148-56. PubMed ID: 16870831
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  • 8. Cd2+-induced swelling-contraction dynamics in isolated kidney cortex mitochondria: role of Ca2+ uniporter, K+ cycling, and protonmotive force.
    Lee WK, Spielmann M, Bork U, Thévenod F.
    Am J Physiol Cell Physiol; 2005 Sep 01; 289(3):C656-64. PubMed ID: 15843441
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  • 10. Identification of the Large-Conductance Ca2+-Regulated Potassium Channel in Mitochondria of Human Bronchial Epithelial Cells.
    Sek A, Kampa RP, Kulawiak B, Szewczyk A, Bednarczyk P.
    Molecules; 2021 May 27; 26(11):. PubMed ID: 34072205
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  • 11. Essential role of mitochondrial Ca2+-activated and ATP-sensitive K+ channels in sildenafil-induced late cardioprotection.
    Wang X, Fisher PW, Xi L, Kukreja RC.
    J Mol Cell Cardiol; 2008 Jan 27; 44(1):105-13. PubMed ID: 18021798
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  • 13. Large-conductance Ca²⁺-activated potassium channel in mitochondria of endothelial EA.hy926 cells.
    Bednarczyk P, Koziel A, Jarmuszkiewicz W, Szewczyk A.
    Am J Physiol Heart Circ Physiol; 2013 Jun 01; 304(11):H1415-27. PubMed ID: 23542921
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  • 14. Opening of mitochondrial K(ATP) channels attenuates the ouabain-induced calcium overload in mitochondria.
    Ishida H, Hirota Y, Genka C, Nakazawa H, Nakaya H, Sato T.
    Circ Res; 2001 Nov 09; 89(10):856-8. PubMed ID: 11701611
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  • 15. Bioenergetic consequences of opening the ATP-sensitive K(+) channel of heart mitochondria.
    Kowaltowski AJ, Seetharaman S, Paucek P, Garlid KD.
    Am J Physiol Heart Circ Physiol; 2001 Feb 09; 280(2):H649-57. PubMed ID: 11158963
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  • 16. Opening of potassium channels protects mitochondrial function from calcium overload.
    Crestanello JA, Doliba NM, Babsky AM, Doliba NM, Niibori K, Osbakken MD, Whitman GJ.
    J Surg Res; 2000 Dec 09; 94(2):116-23. PubMed ID: 11104651
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  • 18. The direct physiological effects of mitoK(ATP) opening on heart mitochondria.
    Costa AD, Quinlan CL, Andrukhiv A, West IC, Jabůrek M, Garlid KD.
    Am J Physiol Heart Circ Physiol; 2006 Jan 09; 290(1):H406-15. PubMed ID: 16143645
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  • 19. Mitochondrial K(ATP) channel activation reduces anoxic injury by restoring mitochondrial membrane potential.
    Xu M, Wang Y, Ayub A, Ashraf M.
    Am J Physiol Heart Circ Physiol; 2001 Sep 09; 281(3):H1295-303. PubMed ID: 11514300
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  • 20. The permeability of uncoupled heart mitochondria to potassium ion.
    Jung DW, Brierley GP.
    J Biol Chem; 1984 Jun 10; 259(11):6904-11. PubMed ID: 6202687
    [Abstract] [Full Text] [Related]


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