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243 related items for PubMed ID: 19799913
1. Human actin mutations associated with hypertrophic and dilated cardiomyopathies demonstrate distinct thin filament regulatory properties in vitro. Debold EP, Saber W, Cheema Y, Bookwalter CS, Trybus KM, Warshaw DM, Vanburen P. J Mol Cell Cardiol; 2010 Feb; 48(2):286-92. PubMed ID: 19799913 [Abstract] [Full Text] [Related]
2. The immediate effect of HCM causing actin mutants E99K and A230V on actin-Tm-myosin interaction in thin-filament reconstituted myocardium. Bai F, Caster HM, Dawson JF, Kawai M. J Mol Cell Cardiol; 2015 Feb; 79():123-32. PubMed ID: 25451174 [Abstract] [Full Text] [Related]
3. Functional Characterization of Cardiac Actin Mutants Causing Hypertrophic (p.A295S) and Dilated Cardiomyopathy (p.R312H and p.E361G). Hassoun R, Erdmann C, Schmitt S, Fujita-Becker S, Mügge A, Schröder RR, Geyer M, Borbor M, Jaquet K, Hamdani N, Mannherz HG. Int J Mol Sci; 2022 Apr 18; 23(8):. PubMed ID: 35457283 [Abstract] [Full Text] [Related]
9. Cardiomyopathy-associated mutations in tropomyosin differently affect actin-myosin interaction at single-molecule and ensemble levels. Kopylova GV, Shchepkin DV, Nabiev SR, Matyushenko AM, Koubassova NA, Levitsky DI, Bershitsky SY. J Muscle Res Cell Motil; 2019 Dec 07; 40(3-4):299-308. PubMed ID: 31643006 [Abstract] [Full Text] [Related]
10. Investigation of a truncated cardiac troponin T that causes familial hypertrophic cardiomyopathy: Ca(2+) regulatory properties of reconstituted thin filaments depend on the ratio of mutant to wild-type protein. Redwood C, Lohmann K, Bing W, Esposito GM, Elliott K, Abdulrazzak H, Knott A, Purcell I, Marston S, Watkins H. Circ Res; 2000 Jun 09; 86(11):1146-52. PubMed ID: 10850966 [Abstract] [Full Text] [Related]
14. Regulation of contraction in striated muscle. Gordon AM, Homsher E, Regnier M. Physiol Rev; 2000 Apr 09; 80(2):853-924. PubMed ID: 10747208 [Abstract] [Full Text] [Related]
15. Alterations in thin filament regulation induced by a human cardiac troponin T mutant that causes dilated cardiomyopathy are distinct from those induced by troponin T mutants that cause hypertrophic cardiomyopathy. Robinson P, Mirza M, Knott A, Abdulrazzak H, Willott R, Marston S, Watkins H, Redwood C. J Biol Chem; 2002 Oct 25; 277(43):40710-6. PubMed ID: 12186860 [Abstract] [Full Text] [Related]
17. R403Q and L908V mutant beta-cardiac myosin from patients with familial hypertrophic cardiomyopathy exhibit enhanced mechanical performance at the single molecule level. Palmiter KA, Tyska MJ, Haeberle JR, Alpert NR, Fananapazir L, Warshaw DM. J Muscle Res Cell Motil; 2000 Oct 25; 21(7):609-20. PubMed ID: 11227787 [Abstract] [Full Text] [Related]
18. Mechanism of regulation of phosphate dissociation from actomyosin-ADP-Pi by thin filament proteins. Heeley DH, Belknap B, White HD. Proc Natl Acad Sci U S A; 2002 Dec 24; 99(26):16731-6. PubMed ID: 12486217 [Abstract] [Full Text] [Related]
19. Effects of troponin T cardiomyopathy mutations on the calcium sensitivity of the regulated thin filament and the actomyosin cross-bridge kinetics of human β-cardiac myosin. Sommese RF, Nag S, Sutton S, Miller SM, Spudich JA, Ruppel KM. PLoS One; 2013 Dec 24; 8(12):e83403. PubMed ID: 24367593 [Abstract] [Full Text] [Related]
20. Effects of the cardiomyopathy-causing E244D mutation of troponin T on the structures of cardiac thin filaments studied by small-angle X-ray scattering. Matsuo T, Kono F, Fujiwara S. J Struct Biol; 2019 Feb 01; 205(2):196-205. PubMed ID: 30599212 [Abstract] [Full Text] [Related] Page: [Next] [New Search]