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157 related items for PubMed ID: 19909779

  • 1. Polyglutamine-expanded ataxin-7 causes cerebellar dysfunction by inducing transcriptional dysregulation.
    Chou AH, Chen CY, Chen SY, Chen WJ, Chen YL, Weng YS, Wang HL.
    Neurochem Int; 2010 Jan; 56(2):329-39. PubMed ID: 19909779
    [Abstract] [Full Text] [Related]

  • 2. Polyglutamine-expanded ataxin-7 upregulates Bax expression by activating p53 in cerebellar and inferior olivary neurons.
    Wang HL, Chou AH, Lin AC, Chen SY, Weng YH, Yeh TH.
    Exp Neurol; 2010 Aug; 224(2):486-94. PubMed ID: 20546728
    [Abstract] [Full Text] [Related]

  • 3. Polyglutamine-expanded ataxin-3 causes cerebellar dysfunction of SCA3 transgenic mice by inducing transcriptional dysregulation.
    Chou AH, Yeh TH, Ouyang P, Chen YL, Chen SY, Wang HL.
    Neurobiol Dis; 2008 Jul; 31(1):89-101. PubMed ID: 18502140
    [Abstract] [Full Text] [Related]

  • 4. Polyglutamine-expanded ataxin-7 activates mitochondrial apoptotic pathway of cerebellar neurons by upregulating Bax and downregulating Bcl-x(L).
    Wang HL, Yeh TH, Chou AH, Kuo YL, Luo LJ, He CY, Huang PC, Li AH.
    Cell Signal; 2006 Apr; 18(4):541-52. PubMed ID: 15964171
    [Abstract] [Full Text] [Related]

  • 5. HDAC inhibitor sodium butyrate reverses transcriptional downregulation and ameliorates ataxic symptoms in a transgenic mouse model of SCA3.
    Chou AH, Chen SY, Yeh TH, Weng YH, Wang HL.
    Neurobiol Dis; 2011 Feb; 41(2):481-8. PubMed ID: 21047555
    [Abstract] [Full Text] [Related]

  • 6. Interferon β induces clearance of mutant ataxin 7 and improves locomotion in SCA7 knock-in mice.
    Chort A, Alves S, Marinello M, Dufresnois B, Dornbierer JG, Tesson C, Latouche M, Baker DP, Barkats M, El Hachimi KH, Ruberg M, Janer A, Stevanin G, Brice A, Sittler A.
    Brain; 2013 Jun; 136(Pt 6):1732-45. PubMed ID: 23518714
    [Abstract] [Full Text] [Related]

  • 7. Decreased expression of Hsp27 and Hsp70 in transformed lymphoblastoid cells from patients with spinocerebellar ataxia type 7.
    Tsai HF, Lin SJ, Li C, Hsieh M.
    Biochem Biophys Res Commun; 2005 Sep 09; 334(4):1279-86. PubMed ID: 16039988
    [Abstract] [Full Text] [Related]

  • 8. RNAi suppresses polyglutamine-induced neurodegeneration in a model of spinocerebellar ataxia.
    Xia H, Mao Q, Eliason SL, Harper SQ, Martins IH, Orr HT, Paulson HL, Yang L, Kotin RM, Davidson BL.
    Nat Med; 2004 Aug 09; 10(8):816-20. PubMed ID: 15235598
    [Abstract] [Full Text] [Related]

  • 9. Transcriptional alterations and chromatin remodeling in polyglutamine diseases.
    Helmlinger D, Tora L, Devys D.
    Trends Genet; 2006 Oct 09; 22(10):562-70. PubMed ID: 16911843
    [Abstract] [Full Text] [Related]

  • 10. p53 activation mediates polyglutamine-expanded ataxin-3 upregulation of Bax expression in cerebellar and pontine nuclei neurons.
    Chou AH, Lin AC, Hong KY, Hu SH, Chen YL, Chen JY, Wang HL.
    Neurochem Int; 2011 Feb 09; 58(2):145-52. PubMed ID: 21092747
    [Abstract] [Full Text] [Related]

  • 11. Polyglutamine-expanded ataxin-3 activates mitochondrial apoptotic pathway by upregulating Bax and downregulating Bcl-xL.
    Chou AH, Yeh TH, Kuo YL, Kao YC, Jou MJ, Hsu CY, Tsai SR, Kakizuka A, Wang HL.
    Neurobiol Dis; 2006 Feb 09; 21(2):333-45. PubMed ID: 16112867
    [Abstract] [Full Text] [Related]

  • 12. Polyglutamine-expanded ataxin-7 decreases nuclear translocation of NF-kappaB p65 and impairs NF-kappaB activity by inhibiting proteasome activity of cerebellar neurons.
    Wang HL, He CY, Chou AH, Yeh TH, Chen YL, Li AH.
    Cell Signal; 2007 Mar 09; 19(3):573-81. PubMed ID: 17005371
    [Abstract] [Full Text] [Related]

  • 13. Spinocerebellar ataxia type 7 cerebellar disease requires the coordinated action of mutant ataxin-7 in neurons and glia, and displays non-cell-autonomous bergmann glia degeneration.
    Furrer SA, Mohanachandran MS, Waldherr SM, Chang C, Damian VA, Sopher BL, Garden GA, La Spada AR.
    J Neurosci; 2011 Nov 09; 31(45):16269-78. PubMed ID: 22072678
    [Abstract] [Full Text] [Related]

  • 14. T1-11 and JMF1907 ameliorate polyglutamine-expanded ataxin-3-induced neurodegeneration, transcriptional dysregulation and ataxic symptom in the SCA3 transgenic mouse.
    Chou AH, Chen YL, Chiu CC, Yuan SJ, Weng YH, Yeh TH, Lin YL, Fang JM, Wang HL.
    Neuropharmacology; 2015 Dec 09; 99():308-17. PubMed ID: 26254860
    [Abstract] [Full Text] [Related]

  • 15. H1152 promotes the degradation of polyglutamine-expanded ataxin-3 or ataxin-7 independently of its ROCK-inhibiting effect and ameliorates mutant ataxin-3-induced neurodegeneration in the SCA3 transgenic mouse.
    Wang HL, Hu SH, Chou AH, Wang SS, Weng YH, Yeh TH.
    Neuropharmacology; 2013 Jul 09; 70():1-11. PubMed ID: 23347954
    [Abstract] [Full Text] [Related]

  • 16. Reduction of mutant ataxin-7 expression restores motor function and prevents cerebellar synaptic reorganization in a conditional mouse model of SCA7.
    Furrer SA, Waldherr SM, Mohanachandran MS, Baughn TD, Nguyen KT, Sopher BL, Damian VA, Garden GA, La Spada AR.
    Hum Mol Genet; 2013 Mar 01; 22(5):890-903. PubMed ID: 23197655
    [Abstract] [Full Text] [Related]

  • 17. Nuclear localization or inclusion body formation of ataxin-2 are not necessary for SCA2 pathogenesis in mouse or human.
    Huynh DP, Figueroa K, Hoang N, Pulst SM.
    Nat Genet; 2000 Sep 01; 26(1):44-50. PubMed ID: 10973246
    [Abstract] [Full Text] [Related]

  • 18. Spinocerebellar ataxia type 7 associated with pigmentary retinal dystrophy.
    Michalik A, Martin JJ, Van Broeckhoven C.
    Eur J Hum Genet; 2004 Jan 01; 12(1):2-15. PubMed ID: 14571264
    [Abstract] [Full Text] [Related]

  • 19. Neuronal dysfunction in a polyglutamine disease model occurs in the absence of ubiquitin-proteasome system impairment and inversely correlates with the degree of nuclear inclusion formation.
    Bowman AB, Yoo SY, Dantuma NP, Zoghbi HY.
    Hum Mol Genet; 2005 Mar 01; 14(5):679-91. PubMed ID: 15661755
    [Abstract] [Full Text] [Related]

  • 20. Disease progression despite early loss of polyglutamine protein expression in SCA7 mouse model.
    Helmlinger D, Abou-Sleymane G, Yvert G, Rousseau S, Weber C, Trottier Y, Mandel JL, Devys D.
    J Neurosci; 2004 Feb 25; 24(8):1881-7. PubMed ID: 14985428
    [Abstract] [Full Text] [Related]

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