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251 related items for PubMed ID: 19939931
1. Herpes simplex virus VP16, but not ICP0, is required to reduce histone occupancy and enhance histone acetylation on viral genomes in U2OS osteosarcoma cells. Hancock MH, Cliffe AR, Knipe DM, Smiley JR. J Virol; 2010 Feb; 84(3):1366-75. PubMed ID: 19939931 [Abstract] [Full Text] [Related]
2. ICP0, ICP4, or VP16 expressed from adenovirus vectors induces reactivation of latent herpes simplex virus type 1 in primary cultures of latently infected trigeminal ganglion cells. Halford WP, Kemp CD, Isler JA, Davido DJ, Schaffer PA. J Virol; 2001 Jul; 75(13):6143-53. PubMed ID: 11390616 [Abstract] [Full Text] [Related]
4. An activity specified by the osteosarcoma line U2OS can substitute functionally for ICP0, a major regulatory protein of herpes simplex virus type 1. Yao F, Schaffer PA. J Virol; 1995 Oct; 69(10):6249-58. PubMed ID: 7666525 [Abstract] [Full Text] [Related]
7. ICP0 is not required for efficient stress-induced reactivation of herpes simplex virus type 1 from cultured quiescently infected neuronal cells. Miller CS, Danaher RJ, Jacob RJ. J Virol; 2006 Apr; 80(7):3360-8. PubMed ID: 16537603 [Abstract] [Full Text] [Related]
8. Herpes simplex virus regulatory proteins VP16 and ICP0 counteract an innate intranuclear barrier to viral gene expression. Hancock MH, Corcoran JA, Smiley JR. Virology; 2006 Aug 15; 352(1):237-52. PubMed ID: 16750236 [Abstract] [Full Text] [Related]
17. Reactivation of expression from quiescent herpes simplex virus type 1 genomes in the absence of immediate-early protein ICP0. Preston CM. J Virol; 2007 Nov 15; 81(21):11781-9. PubMed ID: 17715242 [Abstract] [Full Text] [Related]